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We found that the subglottal pressure, mean air flow rate and the glottal efficiency were increased when the intensity was increased.

此外我们发现声门下压力、平均气流速度、及声门效率均会随著音量增大而增大。

When 2.0 g/L of malate was added to the fermentation medium, the metabolic flux of byproducts L-Ala and lactic acid decreased by 22.1% and 16.5%. At the same time, the metabolic channeled to EMP and glyoxyl circle decreased by 2.26% and 9.09%. However, the metabolic flux channeled to HMP increased by 2.26%.The metabolic flux channeled to the L-glutamic acid synthesis pathway increased from 73.59% to 79.92%.

在L-谷氨酸发酵中、后期添加2.0 g/L苹果酸后，合成副产物L-丙氨酸和乳酸的代谢流量明显减少，分别降低了22.1%和16.5%，EMP途径和乙醛酸循环的代谢流分别减少了2.26%和9.09%，HMP途径的代谢流增加了2.26%，而L-谷氨酸生物合成的代谢流从73.59%增长至79.92%，较未添加前提高了6.33%。

According the research, by inducing TiC into conventional carbon/ graphite materials, their flexural strength increased 13.4%, compressive strength increased 38.1%, void ratio decreased 16.9% in respective. The reason of the performance improvement is as follows: TiC decomposed during the process and hence Ti accelerate the graphitize process, which can increase the mechanical performance of the carbon/ceramic composite materials.

从研究结果可以看出，TiC掺杂可使碳陶瓷复合材料的抗折强度提高13.4%，抗压强度提高38.1%，气孔率降低16.9%；其机理在於TiC掺杂在碳陶瓷复合材料制备过程中能促进碳陶瓷石墨化，使晶体更加完整、细化，有利於力学性能的提高。

In Anxi County, food production service values in prohibited grazing region in lowland halophytic meadows and desert grassland increased 16.25 yuan/hm^2 and 7.75 yuan/hm^2, and total values increased by 176.5 yuan/hm^2 and 170.5 yuan/hm^2, respectively.

安西县低地盐生草甸草地禁牧区较非工程区食物生产价值增加了16.25元/hm^2，总价值增加了176.5元/hm^2；温性荒漠类草地禁牧区较非工程区食物生产价值增加了7.75元/hm^2，总价值增加了170.5元/hm^2。

The results showed : thymus, Bursa, Harderian gland and spleen weight increase with age, gradually increasing; Thymus index, Bursa index and Harderian gland, the index increased with age gradually and the spleen index increase with age in the first increased and then decreased.

结果表明：胸腺、法氏囊、哈德氏腺和脾脏的重量随日龄的增长，逐渐增大；胸腺指数、法氏囊指数和哈德氏腺指数随日龄的增长逐渐变小，脾脏指数随日龄的增长，先增加后减小。

Then the tissue and blood samples were obtained and measured.Result: In the muscovite group,restored mucosa thickness increased,cystically dilated glands decreased,microvessels in connective tissue increased,the secretion of mucus,hexosamine, PGE2,EGF,bFGF were enhanced,and the express of EGFR was stronger.

结果：经云母治疗后，大鼠再生黏膜厚度增加、囊状扩张腺体数目减少，肉芽组织炎症细胞浸润数量减少、微血管数量增加、胶原面积增加，黏膜中性黏液和氨基己糖分泌增多，血浆PGE2，EGF分泌增多，免疫组化染色中黏膜局部EGFR，bFGF阳性细胞表达增强。

Results Compared with low-salt diet control group, the renal changes in CsA-L group were not obvious at the 2nd weeks, the urine volume and the level of BUN were remarkably increased in CsA-H group. At the 4th weeks, the urine volume was decreased; the levels of Crea and BUN were significantly increased and showed a significant positive correlation with the blood CsA concentration. Morphologic changes consisted of vacuolization of renal tubular cells, hyalinization of small arterioles and the light renal interstitial fibrosis.

结果 与低盐对照组相比，药后第2周CsA-L组各指标无明显变化，CsA-H组尿量增多，尿蛋白降低，尿素氮升高；药后第4周时，尿量呈减少趋势，肌酐和BUN明显升高，且与CsA血药浓度呈显著正相关，病理表现为肾小管细胞空泡变性及小动脉和肾间质病变，CsA-H组各种变化较CsA-L组更为明显。

Results Microscopically, different degrees of hepatocytic degenerative changes and hyperplasic fibrous tissue and typical false lobule formation could be seen. Timm's staining result showed uneven distributed black granular deposits in the hepatocytes. No specific PAS staining was observed. Utrastructurally, the mitochondria were increased in volume and dramatically different in shape. The number of lysomes were increased.

结果 光镜下肝细胞表现为不同程度的退行性变，胶原纤维增生以及典型假小叶形成；Timm's染色阳性，发现不均匀分布黑色颗粒或团块样物质沉积；PAS染色则普遍缺乏特异性染色；超微结构显示线粒体形态多样，体积增大，溶酶体增多，粗面内质网管腔扩张，附着核糖体的脱颗粒，以及胞质水肿，质膜溶解。

Results are as followed:1 Exposure of HELF cells to BP caused c-Jun activation,and increased the activity of MAPK,PI-3K,p53 and cyclin D1 pathway.2 BP-induced c-Jun activation was inhibited by dominant negative mutants of extracellular signal-regulated protein kinase or c-Jun NH_2-terminal kinase,but not by p38,impling that JNK and ERK pathways medicate c-Jun activation induced by BP.3 Overexpression of dominant-negative mutants PI-3K and Akt potently blocked phosphorylations of c-Jun and ERK,but not JNK in response to BP,suggesting that PI-3K/Akt pathway positively regulates BP-induced c-Jun activation through ERK.4 Inhibition of p53 by its chemical or molecular inhibitor markedly increased the phosphorylation levels of c-Jun,Akt and ERK upon BP stimulation,indicating that p53 negatively medicates BP-induced c-Jun activation through PI-3K/Akt/ERK pathway.5 The cell lines expressed TAM67 exhibits no significant affecting normal cell growth properties.6 TAM67 was able to significantly block G_1-S transition and subsequent cell proliferation,suggesting that c-Jun is essential for cell cycle alternations elicited by BP.7 Overexpression of TAM67 impaired BP-induced cyclin D1 activation,decreasing expression of E2F1 and pRb,indicating that c-Jun participates in the modulation of BP-induced activation of cyclin D1/pRb/E2F1 pathway.8 Stably expression of TAM67 led to the increases in the expression levels of p53 and p21,elevating phosphorylation level of p53,clearly indicating that c-Jun regulates p53/p21 pathway activation induced by BRCollectively,PI3K/Akt/ERK pathway mediated BP-induced c-Jun activation through p53-dependent mechanism.

结果显示：1BP刺激细胞可促进c-Jun活化，并伴随着MAPK、PI-3K、p53和cyclinD1通路各组成成分的活性增强。2利用MAPK通路的显性失活突变体分别阻断细胞外信号调节激酶和c-Jun氨基末端激酶活性，均可明显抑制BP诱导的c-Jun活化，但阻断p38活性对BP引起的c-Jun活化无明显影响，提示JNK和ERK通路参与调控BP诱导的c-Jun活化。3过表达PI-3K和Akt的显性失活突变体也可显著抑制BP诱导的c-Jun活化，并降低磷酸化ERK的表达水平，但对磷酸化JNK的表达水平无明显影响，说明PI-3K/Akt通路通过ERK正性调控了BP诱导的c-Jun活化。4p53的化学/分子抑制剂能使BP作用的细胞内c-Jun活性明显增加，并同时诱导Akt和ERK的磷酸化水平的升高，表明p53可通过PI-3K/Akt/ERK通路对BP诱导的c-Jun活化进行负性调控。5随后观察转染细胞的生长情况，发现TAM67对细胞正常生长和形态无明显影响。6稳定表达TAM67可有效抑制BP诱导的S期细胞数的增加，提示c-Jun在BP致细胞周期改变的过程中发挥了重要作用。7TAM67过表达能够抑制BP诱导的cyclin D1活化，降低磷酸化Rb以及E2F1蛋白表达水平，表明c-Jun参与调控BP诱导的cyclin D1/Rb/E2F1通路的活化。8过表达TAM67可使BP刺激的细胞中p53、p21总蛋白以及p53磷酸化的表达水平明显升高，可见c-Jun也参与调控BP诱导的p53/p21通路活化。

In lungs, histological observation showed that perivascular immunocytes recruitment increased slightly. Local interalveolar septums were incrassated and congestive, and cell number increased.

组织学观察显示肺部小血管周围有少量免疫细胞募集，局部肺泡隔增厚充血，细胞增多。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。