查询词典 in disorder

Emmenagogue and antispasmodic is superior to single western drugs in improving the functional disorder of oxidative stress in patients with hypertension.

解痉通络活血药物对高血压病患者的氧化应激功能紊乱的改善作用，较单纯常规西药治疗效果好。

The morbidity of ocular fundus disorder is higher in high plateau area of China than that in backland.

高原地区眼底异常高于内地，AMD、不明原因的视神经萎缩及视网膜血管性疾病为最主要的致盲眼底病。

There are great variations in the pattern of mood swings in bipolar disorder.

有很大的变化格局情绪波动在双相情感障碍。

In the second half of the 18th century, the British capital London in chaos and disorder is a real crime capital.

18世纪下半叶，英国首都伦敦处于混乱和无序状态，是一个名副其实的罪恶之都。

The pair were initially diagnosed by Roberto Vetrugno of the University of Bologna and his colleagues as suffering from REM behavioural disorder, in which the paralysis, or cataplexy, that normally prevents sleeping people from acting out their dreams is lost. This can cause people in REM sleep to twitch and groan, sometimes flailing about and injuring their bedmates.

他们二人最初被 University of Bologna 的 Roberto Vetrugno 及其同事诊断为 REM 睡眠行为障碍，伴有瘫痪和猝倒症，这种行为障碍的表现通常是睡觉时按照梦境有所动作而无法控制，致使 REM 睡眠期间出现肢体抽动和呻吟，有时乱踢乱打而伤到同床者。

Consider to discover, OSAHS patient appears because of relapsing in nightly Morpheus disease of blood of low oxygen, tall carbonic acid, consensual as disorder as parasympathetic nerve function, already made coronary heart disease (the cardiovascular disease such as CHD) sparks importantly element, can cause all sorts of rhythm of the heart wrong, aggravating cardiac muscle is short of blood, produce sudden death of heart source sex even. 1 O SAH S and hypertensive disease consider to discover, OSAHS patient's nightly small arousal can bring about sympathetic stimulant increase, adrenalin can nerve medium if adrenalin, go adrenalin secretes armour grow in quantity, heighten of material of phenolic amine of catechu of nightly make water.

探究发现，OSAHS患者夜间睡眠中因反复出现低氧、高碳酸血症、交感和副交感神经功能紊乱，已成为冠心病等心血管疾病重要的触发因素，可诱发各种心律失常、加重心肌缺血，甚至发生心源性猝死。1 O SAH S和高血压病探究发现，OSAHS患者夜间的微觉醒可导致交感神经兴奋性增强，肾上腺素能神经介质如肾上腺素、去甲肾上腺素分泌增多，夜间尿儿茶酚胺类物质增高。

The blood pressure of.1 of pathology physiology mechanism that SAS causes CVD breathes time-out to be able to bring about airframe in elevatory Morpheus anoxic, sympathetic is excessive and excited, element of serous catechu phenolic amine, kidney and hemal endodermis element are elevatory, bring about hemal easy of hemal convulsion; to shrink the function is disorder, can make blood-vessel flowing flesh happening reframes and fleshy, systemic blood-vessel obstruction adds element of the kidney when; is chronic and anoxic, system of hemal and nervous element is activationed, bring about blood pressure to lift. Obstruction of way of energy of life of patient of OSAS of disease of blood of 1.2 low oxygen increases, cause air current to interrupt, breath pauses, at the same time airframe gets used to low oxygen environment gradually, breathing centre drops to low oxygen and sensitivity of disease of blood of tall carbonic acid, breath suspends a frequency increasing, farther aggravating airframe is anoxic. 1.3 heads are self-adjusting the function drops normal person changes quickly in systematic circulation blood pressure when, the head can be passed adjust independently functional generation protects effect, make change of cerebral blood flow not big.

SAS诱发CVD的病理生理机制。1血压升高睡眠中呼吸暂停可导致机体缺氧，交感神经过度兴奋，血浆儿茶酚胺、肾素以及血管内皮素升高，导致血管痉挛；血管舒缩功能紊乱，可使血管平滑肌发生重构和肥厚，全身血管阻力增加；慢性缺氧时肾素—血管紧张素系统被激活，导致血压升高。1.2低氧血症OSAS患者气道阻力增加，造成气流中断、呼吸暂停，同时机体逐渐适应低氧环境，呼吸中枢对低氧和高碳酸血症敏感性下降，呼吸暂停次数增加，进一步加重机体缺氧。1.3脑自动调节功能下降正常人在体循环血压快速变化时，脑可通过自主调节功能产生保护效应，使脑血流量变化不大。

They have also observed unusual concentrations of sodium and potassium in patients with celiac disease, a disorder in digesting fats.

他们还发现，脂性下痢患者的毛发中含钾、钠的量不正常。

In his entire writing career, Gaddis has been a social and cultural critic, showing his deep concern over the entropic collapse in the post-war American society and the tangible threat of disorder in the postmodern human world.

纵观其写作生涯，加迪斯以一个社会和文化批评家的身份，对战后美国社会的衰退和战后混乱给人类社会带来的切实威胁表现出极大的关注。

Results 1、 Generally, we can see the original blue and white, shiny, no cracks in the articular surface of the cartilage after the stress increases gradually yellow, surface roughness, cracks appear; when the pressure decreases, the yellowing, rough, the color of the fracture restore gradually and become shiny.2、the shiny smooth surface can be seen under a light microscope, formation, cell distribution, tidy, clear the level of cartilage at the articular surface stress increases, the surface roughness changes, defects, disordered cells, uneven dyeing ; when the articular surface of the pressure gradually decreased, the cartilage gradually repair and the surface of cells at the surface appear only disorder.3、immunohistochemical observation can be seen throughout the observation period, cartilage cells are type Ⅱ collagen expression and expression after 3 weeks gradually weakening, when the seventh week begin to strong gradually.4、 electron microscopy shows that when stress increases the articular surface, the cartilage cells became flat, the cytoplasm in the endoplasmic reticulum, Golgi apparatus decreased with collagen disorders; and when stress decreases the articular surface, cartilage cells gradually returned normal, cytoplasm in the endoplasmic reticulum, Golgi body gradually restore quantity; collagen fibers with a gradual rules.

结果：①大体观察可见到原本蓝白色、有光泽、无裂纹的软骨在关节面压力增大后，逐渐呈灰黄色，表面粗糙，出现裂隙；当压力逐渐减小后，变黄、粗糙、有裂隙的软骨颜色逐渐恢复，变得有光泽②光镜下可见表面光滑、平整，细胞分布均匀、整齐，层次清楚的软骨在关节面压力增大后，表面变粗糙、缺损，细胞排列紊乱、染色不均；当关节面压力逐渐减小后，软骨表面逐渐修复，细胞仅在表层排列紊乱③免疫组织化学观察可见整个观察期内软骨细胞胞浆内均有Ⅱ型胶原表达，术后3周内表达逐渐变弱，从第7周时开始逐渐变强。④电镜下可见当关节面压力增大后，软骨细胞逐渐变扁，胞质中内质网膜、高尔基体减少，胶原排列紊乱；当关节面压力减小，软骨细胞形态逐渐恢复正常，胞质中内质网膜、高尔基体数量逐渐恢复；胶原纤维排列逐渐有规则。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。