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hypoxia相关的网络例句

查询词典 hypoxia

与 hypoxia 相关的网络例句 [注:此内容来源于网络,仅供参考]

Results In hypoxia control group, the time for OPS to disappear was (186±16) seconds; HIP occurred in all slices within about 10 minutes of hypoxia and lasted for (113±16) seconds. When normal ACSF superfusion was restored, the rates of OPS recovery and OPS amplitude were 25% and (30±23)% respectively.

结果 缺氧组OPS消失时间为(186±26)s,在缺氧10min左右,均出现HIP,持续时间为(113±16)s;恢复供氧后,OPS恢复率和恢复幅度分别为25%和(30±23)%。

By superfusion of ET-1,the hypoxia-induced decrease in RPF was remarkably potentiated and the occurrence of pacemaker arrest was shifted to an earlier time. The h3,poxia-induced effects could be effectively attenuated after pretreatment with BQ-123,implying the role of endogenous ET-1 release in hypoxia-induced effects.

ET-1可显著加重缺氧所致的RPF降低,使起搏细胞停跳时间前移;而以ET*受体阻断剂BQ-123预处理窦房结标本后,则能有效地缓解缺氧对起搏细胞的效应,提示内源性ET-1的释放在缺氧效应中的作用。

The experiments showed a significant increase in Bax mRNA levels (P.01) in neurons cultured at severe hypoxia condition compared with those cultured at normoxia. In hypoxia groups, tolbutamide increased Bax mRNA expression (P.05), while diazoxide reduced it (n=5 for each).

缺氧+二氮嗪组中基因Bax的mRNA表达水平与单纯缺氧组相比下降(P.01),缺氧+甲苯磺丁脲组中基因Bax的mRNA表达水平相对于单纯缺氧组也有提高(P.05)。

Objective To investigate the influence of hypoxia on the expressions of hypoxia inducible factor 1α and vascular endothelial growth factor,the angiogenic response and its mechanism.

目的:探讨缺氧对人胶质瘤细胞株U251缺氧诱导因子-1α和血管内皮生长因子表达及血管生成反应的影响及机制。

The result indicates that Bassia rapa L. 65%, 95% alcohol extract and water extract can prolong the survival time of mice in hypoxia under ordinary pressures significantly. It can also prolong the survival time of mice with acute cerebral ischemia and sodium nitrite significantly, and increase the number of erythrocytes and the content of hemoglobin. It demonstrates that Bassia rapa L. extract has anti-hypoxia activity.

结果表明,芫根水提取物、65%乙醇提取物及95%乙醇提取物均能明显延长模型小鼠在常压缺氧、急性脑缺血性缺氧及亚硝酸钠中毒时的存活时间,并可增加外周血象中红细胞数与血红蛋白含量,显示芫根提取物具有抗缺氧活性。

But the charges Vick with campana, said the four near-death experiences of the subjects had never experienced the state of brain hypoxia, it should not be a near-death experiences the brain hypoxia causes.

我立即打电话给她的妹妹,她的回答是更可怕的:我的脑袋里的东西,我一闭上眼睛,她将可以使用我的手写字,在课堂上给你的信我写出…她说,想成为我的朋友,不会伤害谁,他们不害怕…。

2The distribution of HIF-1 a and COX-2 was unequal;.(3)These findings indicate that HIF-1a and COX-2 may be two important factors in pulpal lesions and there is a significantly hypoxia condiction in inflamed pulp .(4)The increase of HIF-1 a is a possible mechanism for hypoxia preconditioning .(5)Our results suggested that pulpal fibroblasts as well as macrophages might be involved in the pathogenesis of irreversible pulpitis.

和COX一2的表达均有不同程度的变化,在牙髓炎病程中可能发挥着重要的生理和病理作用,③在牙髓炎进程中和牙髓自身修复过程中存在着缺氧环境和缺氧耐受机制,④牙髓成纤维细胞有可能与单核巨噬细胞一同参与牙髓炎的病理进程,且具有修复和免疫功能。

The mechanism might be elevated the HIF-la in histocyte through which accelerate the expression of EPO level, enhance the body's ability of carrying and transporting oxygen and increase the activity of key enzymes in glycolysis in hypoxia mice, and elevated the ATP level to remission the energy metabolism in hypoxia mice.

马齿苋总黄酮能够提高小鼠抗缺氧能力,机制可能通过提高组织HIF-1α含量,促进EPO表达,增加了血红蛋白的合成和红细胞的数量,增强机体携运氧能力,同时促进了无氧酵解进行,缓解了缺氧小鼠的能量代谢障碍。

The mechanism might be elevated the HIF-la in histocyte through which accelerate the expression of EPO level, enhance the bodys ability of carrying and transporting oxygen and increase the activity of key enzymes in glycolysis in hypoxia mice, and elevated the ATP level to remission the energy metabolism in hypoxia mice.

马齿苋总黄酮能够提高小鼠抗缺氧能力,机制可能通过提高组织HIF-1α含量,促进EPO表达,增加了血红蛋白的合成和红细胞的数量,增强机体携运氧能力,同时促进了无氧酵解进行,缓解了缺氧小鼠的能量代谢障碍。

The cell suppression ratio and surviving ratio were detected respectively under conditions of simple hypoxia, or 5mmol/L glucose complicated with hypoxia. The mRNA levels of key enzymes that are involved in regulating glyco-metabolism were detected, including the mRNA levels of pyruvate-kinase, hexokinase, succinic dehydrogenase and isocitric dehydrogenase, and the activity of lactate dehydrognase.

以SMMC-7721人肝癌细胞和L02正常肝细胞作为研究对象,分别在单纯缺氧及加葡萄糖缺氧条件下,观察细胞生长,并检测糖代谢关键酶:丙酮酸激酶(pyruvate-kinase, PK)、己糖激酶(hexokinase, HK)、琥珀酸脱氢酶(succinic dehydrogenase, SDH)、异柠檬酸脱氢酶(isocitric dehydrogenase, IDH) mRNA表达水平和乳酸脱氢酶(lactate dehydrogenase, LDH)活性。

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