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hypertensive相关的网络例句

查询词典 hypertensive

与 hypertensive 相关的网络例句 [注:此内容来源于网络,仅供参考]

Mast cells could develop a key role in the expression of these three phenotypes because their mediators have the ability to produce all the aforementioned alterations, both at the splanchnic level (portal hypertensive enteropathy, mesenteric adenitis, liver steatosis) and the systemic level (portal hypertensive encephalopathy).

肥大细胞可能在这一过程中其关键作用,因为它的介质可以引起前面提到的变化,包括内脏(门静脉高压性肠病,肠系膜淋巴结炎,肝脂肪变性)和全身。

Sum up as follows now. This group uses 1 clinical data ancon vein is pressed determine heart function of the patient 44, male 24, female 20; age 46 years old of ~79 year old, average 56 years old of; classification by function of new York heart, heart failure Ⅲ spends 6,Ⅱ spends 23,Ⅰ spends 15; right heart failure 31, complete heart failure heart disease of sex of source of 13; lung 14, hypertensive heart disease 8, coronary heart disease 5 (among them acute cardiac muscle straightens dead 2), outspread sex cardiac muscle is ill 7, rheumatism sex heart disease 4, hypertensive heart disease is amalgamative coronary heart disease 3, be short of disease of cardiac muscle of courage and uprightness 2, congenital heart disease 1. 2 detect 2.1 equipment treat the method dish 1, l measure press glass to be in charge of 1 (long 30cm, internal diameter 4mm), also can use measure a head to press a canal to replace, 10mL injector 1, 12500U heparin fluid 1. 2.2 operations patient is taken smooth lie, make be exhibited outside the upper arm show ° of 45 °~60 with trunk, and difficulty breathes like the patient in same horizontal; with right atrium, desirable lie partly, arm height is equivalent to the 2nd costal region.

现总结如下。1临床资料本组采用肘静脉压测定病人的心功能44例,男24例,女20例;年龄46岁~79岁,平均56岁;按纽约心功能分级,心力衰竭Ⅲ度6例,Ⅱ度23例,Ⅰ度15例;右心力衰竭31例,全心力衰竭13例;肺源性心脏病14例,高血压心脏病8例,冠心病5例(其中急性心肌梗死2例),扩张性心肌病7例,风湿性心脏病4例,高血压心脏病合并冠心病3例,缺血性心肌病2例,先天性心脏病1例。2检测方法2.1设备治疗盘1个,L型测压玻璃管1个(长30cm,内径4mm),也可以用测脑压管代替,10mL注射器1具,12500U肝素液1支。2.2操作病人取平卧位,使上臂外展和躯干呈45°~60°,且和右心房在同一水平面;如病人呼吸困难,可取半卧位,手臂高度相当于第2肋间。

Portal hypertensive enteropathy,whose fundamental pathologic change is a vasculopathy characterized histopathologically by mucosal and submucosal vessel dilatation,oedema and congestion,mainly results from a combination of increased intrahepatic vascular resistance and increased blood flow through the portal venous system.

肝硬化门静脉高压性肠病(portal hypertensive enteropathy,PHE)的2大主要发病机制是肝内门脉血流阻力增加和流经门脉血流增多。PHE的基本特征是组织病理学上的特征性血管病变。临床表现无特异性,但却是下消化道出血的主要原因。

In hypertensive patients with heart failure,the ratio of ADM/ADT decreased with the deterioration of heart failure Conclusion:The oversecretion of ADT and slow compensation of ADM is a characteristic deregulation of vasoactive peptide in the process of hypertensive heart failure.

ADM代偿缓慢和ADT分泌过度是高血压心力衰竭过程中血管活性肽调节紊乱的一个特点。

Methods Wistar rats were divided into four groups:normal group(n=8),model group with cirrhosis and portal hypertensive gastropathy(n=12),disconnective group(n=12)and post-disconnection plus CGRP group (n=12).Gastric mucosal cellular proliferative parameters from the above groups were measured by the radioscintigraphy.

Wistar大鼠分正常对照组8只,肝硬变合并门静脉高压性胃病(portal hypertensive gastropathy,PHG)模型对照组12只,断流手术组12只,断流术加CGRP保护组12只,用放射自显影技术测定各组胃粘膜细胞增殖参数。

The molecular mechanism in hypertensive kidney lesion was demonstrated by monitoring the levels of serum TGF-β, nitric oxide and plasma AngH in essential hypertensive patients and investigating the effect of information convection in endothelial cell on fibrillar collagen metabolism and its relationship with hypertensive kidney lesion.

该课题通过测定原发性高血压病人及伴肾脏损害病人的TGF-β_1、NO和Ang Ⅱ的水平,探讨内皮细胞产生的信息传递对纤维胶原代谢的影响及与高血压性肾损害的关系,以揭示高血压性肾损害发生发展的细胞分子机制。

The fundus showed hypertensive retinopathy (arteriolar constriction, hemorrhage, cotton wool spots, and focal intraretinal periarteriolar transudates), hypertensive choroidopathy (exudative retinal detachment, choroidal infarction, and RPE degeneration), and hypertensive optic neuropathy.

眼底可以看到高血压视网膜病变(动脉挛缩,网膜出血,棉花状斑,以及局部网膜内动脉旁漏出物FIPTs),高血压脉络膜病变(渗出性视网膜剥离,脉络膜梗塞,以及色素网膜变性),及高血压视神经病变。

This investigation sets out to examine the effect of rilmenidine administered systemically on basal and reflexly activated renal sympathetic nerve activity in spontaneous hypertensive rats.

观察利美尼定对高血压鼠(spontaneous hypertensive rat-SHR)基础和反射激活肾交感神经活动(renal sympathetic nerve activity, RSNA)的影响。高血压鼠分为对照组和实验组,每组各10只。

Methods 96 cases of hypertensive patients underwent an acute saline loading test to determine salt sensitives. A 6-day diet survey were undergone to calculate the amount of carbohydrate intake. Results Plasma insulin levels and blood pressures were higher in high carbohydrate intake group in SS hypertensive patients than those in NSS hypertensive patients.

方法对96例高血压患者采用急性盐水负荷试验,确定63例为盐敏感性高血压,33例为盐不敏感性高血压患者,分别测其血压、血糖、血胰岛素水平和体重指数后,1月间先后2次每次3天的饮食回顾性调查,计算其糖的摄入量。

Background: New studies showed that changes in the balance between haemostatic and antithrombotic processes, including a prethrombotic state might contribute to the development of atherosclerotic lesions which occured in hypertensive vascular complications. This may be one of the causes that satisfactory blood pressure reduction with antihypertensive drugs does not lead to an equal reduction in heart attacks and strokes. With the development of traditional presentation of blood coagulation, the role of tissue factor pathway has been emphasized.

中文题名原发性高血压患者组织因子途径变化与血栓前状态关系的临床与实验研究副题名外文题名 Clinical and experimental study of relation between changes of tissue factor pathway and prethrombotic state in hypertensive patients 论文作者张卫茹导师孙明教授学科专业内科学研究领域\研究方向学位级别博士学位授予单位中南大学学位授予日期2002 论文页码总数70页关键词血栓组织因子高血压馆藏号BSLW /2003 /R544 /3 背景:新近的研究表明,凝血与抗凝系统失衡所导致的血栓前状态与高血压并发症的发生关系密切,这可能是高血压病人虽经有效的降压治疗但发生心、脑、肾、血管并发症的危险仍很高的主要原因之一。

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