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hypersecretion相关的网络例句

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与 hypersecretion 相关的网络例句 [注:此内容来源于网络,仅供参考]

AIM: To explore the mechanism of signal cascades in cigarette smoke-induced mucous hypersecretion.

目的:探讨香烟对气道黏液分泌的影响及其信号转导机制。

Aim Establish a novel mouse model of airway goblet cell hyperplasia and mucus hypersecretion.

目的 建立一种新的小鼠气道上皮杯状细胞增生和粘液高分泌模型。

This is associated with chronic airways inflammation and structural remodeling, including loss of alveolar walls, and goblet cell metaplasia with mucus hypersecretion.

COPD患者的肺功能进行性下降,只有部分可逆,这与慢性气道炎症和气道结构重塑有关,包括肺泡壁的丧失,杯状细胞增生和粘液腺高分泌状态。

Objective To explore the effect of simvastatin on airway mucus hypersecretion in rats.

目的 了解辛伐他汀对丙烯醛引起的气道黏液高分泌的作用及其可能的机制。

Objective To investigate the effects of extracts of Pinellia on a rat model of airway mucus hypersecretion induced by LPS.

目的 探讨半夏提取物对脂多糖诱导的大鼠气道黏液高分泌模型的干预作用。

Lung tissues were examined for cell infiltration and mucus hypersecretion, and the expression of inflammatory biomarkers.

取肺组织检测细胞浸润、粘液的过度分泌以及炎症性生物标记物的表达。

Objective: Mucus hypersecretion is a distinguishing pathophysiological feature of many airway diseases.

目的 气道粘液高分泌是呼吸道多种疾病的重要病理生理特征。

This model helps to screen the new drugs for mucus hypersecretion and research on the pathological mechanism of airway mucus hypersecretion.

这一模型有助于筛选抗黏膜高分泌的新药和研究气道黏膜高分泌的病理机制。

All people completed diagnostic questionnaires for GERD and chronic mucus hypersecretion and lung function test.

所有研究对象完成反流性疾病诊断问卷、慢性黏液高分泌症问卷和肺功能检查。

Cholesterol gallstones formed in C57L mice and fatty livers developed in AKR mice. Conclusions Biliary cholesterol hypersecretion is the key pathophysiological defect of gallstone formation, lith genes have effects on biliary cholesterol hypersecretion and susceptibility to cholesterol gallstone formation in C57L mice. Lithogenic bile is formed at the canalicular membrane and precedes the development of cholesterol gallstones. It is most likely that cholesterol and bile acid hyposecretion make the AKR strain susceptible to the development of fatty livers and resistant to gallstone formation.

结论胆道胆固醇的高分泌是胆囊胆固醇结石形成的主要病理生理基础,结石基因决定了C57L鼠肝内胆汁中胆固醇的高分泌和胆囊胆固醇结石的易患性,成石胆汁形成在肝内胆管,先于胆囊结石的形成;肝内胆汁中胆固醇和胆酸的低分泌可能与AKR鼠脂肪肝的发生和胆囊结石的免患性有关。

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