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hyperphagia相关的网络例句

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与 hyperphagia 相关的网络例句 [注:此内容来源于网络,仅供参考]

If they can tease out the physiological, genetic and chemical causes of hyperphagia among people with disorders, that knowledge may prove a potent tool in combating obesity in the general population.

如果他们能发现患有贪食症的人群的生理学、遗传学及化学病因,这就能为广大人群对抗肥胖提供潜在的可能。如JIM KANE所说的:我们为大众的健康而作个体的研究。

Mutations of the melanocortin-4 receptor (MC4R) are associated with hyperphagia, obesity, and accelerated longitudinal growth in pig, mice and human. However, little is known about the functions of this gene in chicken.

黑素皮质素受体(melanocortin-4 receptor,MC4R)基因的突变与猪、鼠和人等的食欲、肥胖和生长有关联性,然而对鸡的MC4R基因的功能却知之甚少。

Levin BE, Triscari J, Sullivan AC. Metabolic features of dietinduced obesity without hyperphagia in young rats .

实验结果证明后天环境因素如饮食结构异常在胰岛素抵抗综合征的发生中起了重要的作用。

There hae been no new cases of postoperatie hyperphagia, morbid obesity, or behaioral dysfunction in this prospectie cohort.

在这组前瞻队列组中无一例患者发生术后饮食过量、病态肥胖症及行为障碍。

Among the many questions to be explored through working with PWS as a model for obesity research: What role do sensory perceptions -- taste and smell -- play in hyperphagia?

在用PW综合征作为肥胖研究模型的过程中暴露出很多问题:感受器——味觉和嗅觉——在暴食症中起什么作用?

Kane's father, Jim Kane of Towson, this month helped organize a conference for researchers who work with genetic disorders that are characterized by hyperphagia.

Kane的父亲来自Towson的Jim Kane本月协办了一个由研究贪食症等遗传疾患专家参与的学术会议。

Clinical symptoms are difficult to diagnose in infants and only become clearer at later ages as the patients develop hyperphagia and morbid obesity.

普拉德-威利综合征(Prader-Willi Syndrome,PWS)是一种基因组印记相关的疾病,是引起肥胖最常见的遗传综合征。

In another embodiment, the invention discloses pharmaceutical compositions comprising such NPY Y1 receptor antagonists as well as methods of using them to treat obesity, metabolic disorders, eating disorders such as hyperphagia, and diabetes.

在另一个实施方案中,本发明公开了包含这种NPY Y1受体拮抗剂的药物组合物以及使用所述组合物治疗肥胖、代谢紊乱、饮食紊乱和糖尿病的方法。

Here, we show that mice genetically deficient in Toll-like receptor 5 (TLR5), a component of the innate immune system that is expressed in the gut mucosa and that helps defend against infection, exhibit hyperphagia and develop hallmark features of metabolic syndrome, including hyperlipidemia, hypertension, insulin resistance, and increased adiposity.

但美国Emory University的Matam Vijay-Kumar和Gewirtz等人在前人研究的基础上,意外的发现TLR5(Toll样受体-5)缺陷的小鼠较正常小鼠重20%,而且血甘油三酯、血胆固醇、血压及血糖偏高。TLR5缺陷的小鼠比正常小鼠进食多10%,经限制饮食后前者体重下降,但同时对胰岛素的反应下降;在经高脂饮食后前者体重快速增加并表现出&糖尿病&和&代谢综合征&表现。

Levin BE, Triscari J, Sullivan AC. Metabolic features of diet????induced obesity without hyperphagia in young rats \.

实验结果证明后天环境因素如饮食结构异常在胰岛素抵抗综合征的发生中起了重要的作用。

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