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glycogen相关的网络例句

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与 glycogen 相关的网络例句 [注:此内容来源于网络,仅供参考]

Hypoxia and/or exercise increased the content of muscle glycogen,and exercise increased more obviously.

3低氧和/或运动能够增加骨骼肌肌糖原的含量,但常氧运动更有利于肌糖原的合成。

Objective: To observe the effects of Hovenia dulcis Thunb on blood sugar and hepatic glycogen in diabetic mice.

目的:考察枳椇水提取液对四氧嘧啶糖尿病小鼠血糖和肝糖原含量的影响。

In contrast, glycogen obtained from muscle or liver disperse s readily in hot water to make a turbid solution.

相反,从肌肉和肝脏中获得的糖原很容易分散在热水中,产生混浊不清的溶液。

Large heavy-weight Protein muscles with lots of stored Glycogen energy are desirable because they are strong and work efficiently.

大量重量的蛋白质肌肉适合大量储存的肝糖能量,因为它们强壮且运转有效。

MAIN OUTCOME MEASURES: Cell morphology and viability, content of glycogen, as well as urea level in the supernatant.

主要观察指标:肝细胞形态、活性,糖原水平,培养上清中的尿素浓度。

This hormone, insulin, causes the liver to convert more glucose into glycogen (this process is called glycogenesis), and to force about 2/3 of body cells (primarily muscle and fat tissue cells) to take up glucose from the blood, thus decreasing blood sugar levels.

这种激素,胰岛素,导致肝脏葡萄糖转化成糖原更多(这一过程被称为glycogenesis ),并迫使约2 / 3的体细胞(主要是肌肉和脂肪组织细胞)采取了葡萄糖从血液,从而降低血糖水平。

Glucosidase deficiency disease is a rare genetic disorder in which the lysosomal enzyme α-glucosidase is markedly reduced and leads to accumulation of glycogen in various tissues. Its clinical features include massive enlargement of the internal organs, skeletal muscle weakness and hypotonia, and respiratory insufficiency.

阿法-糖苷酶缺乏之全身性糖原质病是一种少见的遗传性疾病,此病主要是因为缺乏阿法-糖苷酶而造成过多的糖原质堆积在各种组识中,导效患者在临床上会有内脏扩大、肌肉无力和呼吸不足等症状。

We also have observed some organs pathological changes of juvenile Jian Carp, such as pancreas acinous gland and zymogen granules of acinous gland cell disappearing, intercellular substance hyperplasia and inflammatory cells soakage, hepatatrophia, liver cell granular or vacuolar degeneration and necrosis, karyolysis or pyknosis, glycogen granules decreasing, metanephros atrophy, metanephric canaliculus epithelium granular or vacuolar degeneration and necrosis, mitochondrion swelling and mitochondrion cristae disappearing, karyolysis, distal convolutal tubule microvilli desquamating, spleen marrow cell degeneration and necrosis, intercellular substance of spleen hyperplasia, spleen atresia, blood corpuscle disappearing.

后肾土黄色、淡褐色或苍白色,肾小管上皮细胞肿胀、颗粒或水泡变性、坏死,细胞内有大量血细胞流出,线粒体肿胀,嵴结构消失,细胞核溶解,肾间质甲状腺滤泡和拟淋巴细胞增生,远曲小管微绒毛脱落、管道细胞界限不清。心脏肌纤维肿胀、颗粒或空泡变性,严重的肌纤维溶解、变细或断裂,肌纤维间水肿、炎性细胞浸润,部分心肌细胞核浓缩。脾脏暗褐色,脾髓质细胞变性、坏死,拟淋巴细胞明显减少,淋巴细胞岛少见、岛中细胞成份减少,黑素巨噬细胞中心减少、体积缩小,脾脏网状基质水肿,脾窦闭锁,血细胞减少。

Results:Six months after the childhood miniature pig transverse fracture of mandibular condyle,MRI medium-sized signal in both two sides in symmetry,no muscle necrosis under light microscope,and decrease of mitochondria,more glycogen granulation,clear myotome were found and no abnormal mitochondria and fatty degeneration under transmission electron microscope were shown.

光镜观察咀嚼肌和翼外肌横截面切片在不同时间均未显示任何破坏现象,透射电镜下髁突横断骨折后6个月组与正常对照组比较无明显变化,只显示线粒体数目减少、糖原颗粒较多、肌节各带清晰,无异常形态线粒体及脂肪变性。

Objective To establish a method for measuring glycogen phosphorylase isoenzyme BB.

目的 建立ELISA法测定糖原磷酸化酶同工酶BB的方法。

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