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glial相关的网络例句

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与 glial 相关的网络例句 [注:此内容来源于网络,仅供参考]

The co-cultured allogenic BMSCs can be induced into neurocyte such as neuron and glial cell in vitro.

同种异体共培养的骨髓间充质干细胞可在体外定向诱导为神经元和胶质细胞等神经细胞。

At histopathologic analysis, neuronal tumors are usually classified as pure neuronal cell tumors (gangliocytoma, Lhermitte-Duclos disease , central neurocytoma) and mixed neuronal-glial tumors (ganglioglioma, desmoplastic infantile ganglioglioma, dysembryoplastic neuroepithelial tumor, ganglioneuroma).

根据组织病理学的分析,神经元肿瘤通常被分为:单纯的神经元细胞肿瘤(神经节细胞瘤,Lhermitte-Duclos 病〔发育不良性小脑神经节细胞〕瘤),中枢神经细胞瘤)和混合性的神经元-胶质肿瘤(神经节神经胶质瘤,成结缔组织性婴儿节细胞胶质瘤,胚胎发育不良性神经上皮瘤,神经节瘤)。

Focal cytopathic areas were observed in cerebral cortical area and so did neuron necrosis around by gliacytes formed stellitorsis, neuronophagia and glial nodule.

RVJR2 3 株在人脑神经细胞和Balb/c小鼠脑组织中均能形成感染,感染为非杀细胞性

Results B, C can cause the hypomnesia of the Wistar rats(P<0.05) and result in decreasing amounts of pyramidale in CA1 sector of hippocampus. The neurotropic and glial nodule were observed.

结果 氯胺酮和D-半乳糖分别可致大鼠记忆功能明显下降(P<0.05)和海马CA1区锥体细胞减少,并可见嗜神经细胞现象和胶质细胞小结。

Human neurotropic JC virus is able to infect human brain glial cells. The causative agent of the fatal human demyelinating disease progressive multifocal leukoencephalopathy, that cytolyses and destroys oligodendrocytes, the myelin-producing cells of the central nervous system. Also results from several experiments have indicated the JCV is able to induce neoplasms.

人类嗜神经性JC病毒可感染人类脑细胞是引起进行性多病灶脑白质病的因素,为一种致命性的脱髓鞘疾病,其导致神经系统产髓质细胞的溶解破坏,但JCV也在许多动物实验上显示其有促成肿瘤的潜在性。

Here, we examined the effects of rehabilitative therapies on proliferation and survival of distinct glial populations in the perilesional area of photochemically induced focal ischemic infarcts in the forelimb sensorimotor cortex in rats.

现在,作者设计一项实验来检测康复训练对光化学诱导的局灶性脑缺血损伤灶周围(梗死灶位于大鼠前肢感觉运动皮质功能区)的不同胶质细胞种群增殖和存活的影响。

A very small number of double-positive cells expressed with PCNA and glial fibrillary acidic protein were first detected in infract boundary in preoptic area on day 3 and onward.

免疫组化双标显示再灌流3 d视前区有很少量的增殖性细胞核抗原/胶质纤维酸性蛋白双阳性细胞,随后增多。

In this study, the genomic structure and regulatory elements of ApoD from 10 representive species including protozoan, invertebrate, protochordate and vertebrate were analyzed and compared. The genomic structure of ApoD is less conserved in organisms from protostome to deuterostome invertebrates, while it is highly conserved among chordates including amphioxus and verebrates. All four conserved cysteine residues are present in amino acid sequence of deuterostome ApoDs, while there are only two cysteine residues in amino acid sequence of protostomes ApoD. Structure divergence between protostome and deuterostome ApoD proteins suggests their function difference. The majority of regulatory elements are present in nearly all organism ApoD genes ranging from unicellular protozoan to mammals, suggesting that ApoD plays a very fundamental role, and possesses a conserved regulatory mechanism. However, there also exist some specific regulatory elements, which are present only in certain species and may perform some special roles.ApoD mRNA expression in murine NIH/3T3 fibroblasts exposed to various stresses such as as hydrogen peroxide and UV light shows dose-dependent increase. And a fly homolog of ApoD, Glial Lazarillo, whose overexpression results in increased resistance to hyperoxia as well as a extension of lifespan under normoxia and resistance to starvation without altering lipid or protein content.

本文首先从生物信息学角度对分属于原生动物、无脊椎动物、头索动物和脊椎动物类群的10种动物ApoD的基因结构及调控区的调控元件进行分析及比较,发现:(1)ApoD基因外显子-内含子结构从原生动物草履虫到原口动物再到后口动物海胆的进化过程中不保守,但在分析的几种脊椎动物中相当保守;(2)文昌鱼ApoD基因扮演从无脊椎动物到脊椎动物承上启下的角色,可能代表了脊椎动物ApoD基因原型;(3)四个半胱氨酸保守位点在后口动物中都存在,而在原口动物中只存在两个,原口、后口动物ApoD蛋白一级结构上的差异反映蛋白功能上可能也存在一定差别;(4)调控区大多数主要调控元件为不同动物共有,说明ApoD主要功能及其表达调控在进化中相当保守;(5)ApoD基因个别调控元件是随着物种进化而出现并开始发挥相关作用,如SF-1;还有一些调控元件在进化过程中还没有发现其规律,这说明ApoD某些功能和基因表达调控模式可能因物种不同而存在一定的差异。

Whereas in glial cells, SP-induced calcium transients had fast kinetics with a rapid rise phase (0.99±0.25/s) and an exponential decaying phase (t〓=12. 6 s). In addition, the spike-like SP response had a steady duration and a distinct delay. SP receptors desensitized completely within a few minutes, and no resensitization was observed within an hour.

而且,SP在胶质细胞上引起的这种峰样〓变化具有稳定的时程,与给药开始时间之间有一个显著的延迟,并极易脱敏,几分钟内就可完全脱敏,1小时内没有发现复敏。

Results In the normal control brains, 14-3-3 immunoreactivity was localized mainly in the neuronal somata and processes, and some glial cells showed only weak immunoreactivity.

结果 在正常脑组织标本中,13-3-3蛋白主要表达于神经元的胞体和突起,仅在少数的胶质细胞中可见14-3-3蛋白的弱表达。

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