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glia相关的网络例句
与 glia 相关的网络例句 [注:此内容来源于网络,仅供参考]

dopamine and acetylcholine inhibited the expression of iNOS in primary glia challenged by LPS in different ways.

结论多巴胺和乙酰胆碱通过不同的方式抑制LPS刺激的原代胶质细胞iNOS的表达。

Expression of Sox2 in the chick retina was detected in progenitor cells, in cells at a discrete location in the layers of amacrine and ganglion cells, and in Muller glia.

在多能干细胞中检测到了鸡视网膜中Sox2的表达,其位于无长突细胞,节细胞和Muller胶质细胞层的离散位置的细胞中。

Those proteins are the cell special proteins of neuron, glia, photoreceptor, bipolar cell, amacrine cell, horizontal cell and the ganglion cell of retina respectively.

并且可表达视网膜的终末分化细胞的标志,包括:感光细胞标志Rhodopsin、双极细胞标志PKC、无长突细胞标志Syntaxin、水平细胞和无长突细胞标志Calbindin和视网膜节细胞标志Thy-1。

The expression level of metabolitic enzyme (CYP1A1, CYP1B1) and its correlation with cell differentiation /apoptosis were studied as well. The influence of resveratrol in cell cycle was determined with FCM. Results 1 Resveratrol not only suppresses the growth of medulloblastoma cells in a dose and time related fashion but also induces cell differentiation and apoptosis. Resveratrol promotes differention of UW228-1,-2 cells to forward glia, UW228-3 and Med-3 cells to neuron. A treatment of resveratrol in the concentration of 100μM for 48hrs comit most of cells die of apoptosis. 2 Among the four cell lines Fas and Caspase-3 were constantly expessed, whereas FasL was absent irrespective to the drug treatment. 3 ICC, RT-PCR, Western-blot hybridization and EROD enzymatic activity assay demonstrated that expression of CYP1A1 is different after treatment with resveratrol in four cell lines, up-regulated in three UW228 cell lines in a dose dependent manner but down-regulated in Med-3 cells. Suppressive effect of resveratrol on CYP1B1 expression is the same among four medulloblatoma cell lines. 4 Cell cycle distribution of UW228-3 was greatly changed after treatment.

结果:1、白藜芦醇以时间和剂量依赖性方式抑制髓母细胞瘤细胞生长,同时促进细胞的分化和凋亡:白藜芦醇促使UW228-1、-2靶细胞向神经胶质细胞方向成熟分化,UW228-3、Med-3细胞向神经元细胞方向成熟分化,以100μM作用48小时效果显著;2、四个细胞系均有Fas和Caspase-3表达,但无Fas-L基因表达和蛋白活性产生,因此难以形成Fas相关性死亡通路;3、白藜芦醇处理前后CYP1A1基因的表达在各细胞系略有不同:UW228-1,-2,-3三个细胞系白藜芦醇以剂量相关性方式上调CYP1A1的表达,而Med-3细胞系,白藜芦醇却抑制CYP1A1的表达;四个细胞系在白藜芦醇处理后CYP1B1的表达均受到抑制;4、UW228-3细胞系经白藜芦醇作用后细胞周期有较大改变,表现在G0/G1期在整个细胞周期中所占的比例增加,而其它时期则相应减少。

Objective: To investigate the dedifferentiation of glia cells and the expression of related molecules after optic nerve injury.

目的:研究视神经损伤后神经胶质细胞去分化的程度及相关分子的表达。

RESULTSdiabetic rats showed typical symptoms involving elevated serum glucose and weight loss (2)The rats in DM+VaD group reacted slower and made more error numbers in Y-maze than the rats in VaD group did, with a prolonged total reacting time in a whole day.(3)More neurons with serious damages, such as edema, ischemia or pyknosis could be found in DM+VaD group than in VaD group. More disarrangement of cone neurons, obvious glia proliferation, and more neuron apoptosis were found in CA area of hippocampus. CONCLUSIONS (1)2-VO in STZ-induced diabetic rats succeeded in establishing VaD animal models.(2) The cognitive dysfunction induced by VaD rats had been deteriorated by diabetes mellitus.(3)Morphology evidences proved that diabetes mellitus aggravated brain damages induced by VaD.PART II Effect of diabetes mellitus on the cholinergic nervous systemin CA1 area of hippocampus of VaDOBJECTIVE To estimate the role of cholinergic nervous system in hippocampus during the process of cognitive dysfunction aggravated by diabetes mellitus in DM+VaD group. METHODS Observe the changes of ChAT protein by immunohistochemistry stain. Chose three rats randomly from each group at each pointat two week, four week, and eight week, then sepa

经腹腔注射链脲佐菌素诱导慢性实验性糖尿病,1周后永久性结扎双侧颈总动脉(2-VO)制作VaD模型记录术后2周、4周和8周各组大鼠的体重及血糖;应用Y-迷宫检测空间定向学第二军医大学博士论文中…丈摘…要1ia;一色恤~﹁染澎糕行HE染色观察组织病理学孪叱;胶质细胞原纤维酸性蛋白protellZ,Cf冰尸标记观察星形胶质细舱的激活情况、橄声软公点娜育票票众糯黑橄居端粗默孺篇筑价井袱第二部分糖尿病对血管性痴呆大鼠海马c心区胆碱能神经系统的影响探讨海马CAI区胆碱能神经系统在糖尿病加重确D一大鼠认知障碍中所实验动物及分组同第一部分,应用免疫组织化学染色方法现察海马CAI{介狱牛第晕军l袭拔常博粗{论义

Pial basement membrane breaches and glia limitans disruptions are the underlying causes of cerebellar granule neuron ectopia in POMGnT1 knockout mice and that migration into the IGL is not required for their differentiation.

1。在POMGnT-1基因敲除小鼠中,导致小脑颗粒细胞异位的原因是软脑膜基底膜层和胶质细胞的界膜的破裂,而颗粒细胞迁移到IGL并非是分化所必需。

The activation of glia in the spinal cord was one important reason of the change in substance P excreted from cornu dorsale medullae spinalis. Inhibitor of glia activation will be a new way to treat CP/CPPS.

胶质细胞活化是CP/CPPS大鼠脊髓背角P物质变化的重要原因,胶质细胞活化抑制剂的应用将是治疗CP/CPPS的新亮点。

In order to explore the function of glia in the progress the experiment was performed both in presence of glia and in the absence of glia The mouse mesencephalic cells were exposed with MPP+ with different concentration. Iloprost was used to investigate whether it can prevent dopamine neuron from the neurotoxic of MPP~..

为了观察胶质细胞在其中的作用,将细胞分为未抑制胶质细胞和胶质细胞抑制组;将不同浓度的MPP~+加入体外培养的中脑多巴胺能神经元培养液中,并加入Iloprost来研究其对MPP~+所导致的神经毒作用是否有保护作用。

In H/R injury of mixedly cultured cerebral cortex neuron and glia, the increase of NO production which was mainly generated by glia, participated in oxidative insult.

在混合培养的大鼠大脑皮质神经细胞和神经胶质细胞

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然而,正如其名字所指出的那样,CD盘不能写,也不能用任何方式改变其内容。

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