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The summary results are below:1. GUS expression under the driving of the BjCHI1 promoter (-1060/+17) was essentially undetectable in the young seedlings under normal growth conditions. GUS activity was first detected in the stigma of young flowers, peaked in the young siliques, and decreased when the siliques became older. No GUS expression was found in the mature siliques, seeds or root.2. The BjCHI1 promoter (-1060/+17) was inducible by NaCl, PEG, wounding and MeJA treatments. High levels of GUS expression were detected in the transgenic tobacco and Arabidopsis plants after wounding, NaCl, PEG, and MeJA treatment, indicating that the BjCHI1 promoter responses to both biotic and abiotic stresses.3. RT-PCR analysis confirmed that the expression of the BjCHI1 gene in B. juncea was inducible by PEG and NaCl.4. The transcription start site was determined by 5′-RACE, and was located at the 17th nucleotide upstream of the translation initiation codon of the BjCHI1 gene.5. A -805/+17 promoter fragment was enough to response to wounding and MeJA induction, which was proved in transgenic tobacco and Arabidopsis plants. The 397 bp region between -805 and -409 of the BjCHI1 promoter contains a cis-acting element that is essential for the wounding and MeJA inducibility.6. The -695/-620 region was necessary but not sufficient to confer MeJA-responsive expression. A T/G-box locates in -353 play an important role in the expression of the BjCHI1 gene in response to MeJA treatment. The 76 bp region is coupled with the T/G-box to confer full MeJA-inducible transcription of the BjCHI1 gene.

主要结果如下：1、利用转基因拟南芥植株分析表明，正常生长条件下，BjCHI1启动子(-1060/+17)驱动GUS基因主要在花柱中表达，幼嫩的荚也有表达，并随着果荚的成熟而减弱，成熟的果荚、种子和根没有显示GUS活性。2、BjCHI1启动子(-1060/+17)能驱动GUS基因在转基因烟草和拟南芥中响应伤害的诱导，转基因拟南芥的分析还证明BjCHI1启动子也受MeJA、NaCl和PEG的诱导，证明BjCHI1启动子是一个伤害、MeJA、NaCl和PEG等生物和非生物因素诱导启动子。3、RT-PCR进一步证明芥菜中BjCHI1基因也受NaCl和PEG的诱导表达。4、5′-RACE法鉴定了BjCHI1启动子的转录起始位点，位于翻译起始位点ATG上游第17个碱基A.5、转基因烟草和拟南芥分析证明，-805/+17的启动子片段足以响应伤害和MeJA的诱导，-805和-409之间397 bp的启动子片段含有对伤害和MeJA诱导必要的元件。6、本明烟叶片瞬时表达系统分析证明，一段76 bp的序列(-695/-620)对BjCHI1启动子响应MeJA的诱导是必要的，但不足以响应MeJA的诱导，位于-353的T/G-box也参与MeJA的诱导。76 bp的序列(-695/-620)与T/G-box协同起作用，赋予BjCHI1启动子MeJA诱导性。

When aborted rats in group E were treated with the dose of 50 IU/g IFN-γ, expression of TNF-αin this group was compared with the other four groups, the results were as follows: Compared with group A, the nucleus pre-opticus medialis, nucleus pre-opticus mango celluaris, nucleus periventricularis hypothalami, nucleus ventromedialis hypothalami, nucleus dorsomedialis hypothalami, nucleus arcuatus hypothalami and pars intermedia had a highly expression of TNF-α, in the other nuclei of hypothalamus, neurohypophsis and adenohypophysis, the change was not significantly, the expression of TNF-αin follicle and corpus luteum were decreased distinctly, the variation in uterus was slight and no difference was exsited; Compared with group B, the expression of TNF-αin nucleus nucleus lateralis hypothalami, nucleus ventromedialis hypothalami, nucleus arcuatus hypothalami, adenohypophysis and uterus presented a remarkable enhancement, conversely, the TNF-αexpression in the nucleus pre-opticus medialis, nucleus pre-opticus suprachiasmaticus, nucleus supra-opticus, nucleus pre-opticus mango celluaris, nucleus periventricularis hypothalami, nucleus paraventricularis hypothalami, neurohypophsis, follicle and corpus luteum had been significantly decreased; Compared with group C, the nucleus pre-opticus medialis, nucleus ventromedialis hypothalami, nucleus dorsomedialis hypothalami, nucleus arcuatus hypothalami, adenohypophysis, pars intermedia and uterus had upregulated the expression of TNF-α, however, it was decreased in the nucleus periventricularis hypothalami and corpus luteum; When compared with group D, the expression of TNF-αin nucleus lateralis hypothalami, nucleus dorsomedialis hypothalami, nucleus ventromedialis hypothalami, pituitary, follicle and ovary stroma were obviously enhanced, while in the nucleus supra-opticus and nucleus arcuatus hypothalami, the situation was definitely opposite, in the other parts, the change was not significant.

外源腹腔注射50 IU/g IFN-γ后，同A组相比：E组TNF-α免疫阳性产物在下丘脑视前内侧核、视前大细胞核、室周核、腹内侧核、背内侧核、弓状核、垂体中间部显著增高，在下丘脑其它核团、神经垂体、垂体前叶较A组差异不显著，在卵泡、黄体中阳性产物表达均较A组显著降低，在子宫中表达较A组变化不明显；同B组相比：E组TNF-α免疫阳性产物在下丘脑外侧核、腹内侧核、弓状核、垂体前叶、子宫均显著增高，在视前内侧核、视交叉上核、视上核、视前大细胞核、室周核、室旁核、神经垂体、卵泡、黄体中阳性物质表达均显著降低；同C组相比：E组下丘脑视前内侧核、腹内侧核、背内侧核、弓状核、垂体前叶与中间部、子宫均显著增高，在室周核、黄体中均显著降低；同D组相比：E组下丘脑外侧核、背内侧核、腹内侧核、垂体各部、卵泡、卵巢基质均显著增高，在视上核、弓状核显著降低，在其它部位表达变化差异不显著。

In the model of tumor cells metastases with cell line A673, 10〓 tumor cells can be detected after only one ampliation with outer primers, which means we can find 1 tumor cells in 10〓 normal PBMC, and enlarged again with inner primers, 10〓 tumor cells can be detected, which means we can find 1 tumor cell in 10〓 normal PBMC. In the simulative model of tumor cells metastases with RNA, 10〓 tumor cell can be detected that is to say we can find 1 tumor cell in about 10〓 normal PBMC. In all 15 samples of 11 patients, 2 patients'EWS/FLI1 expressions were negative, no evidence of metastases was found followed up by 8 months and 30 months respectively. Semi-quantitative study was used to detect the expression level of EWS/FLI1 mRNA in the other 9 positive patients, and metastases occurred in 6 high-level expression patients in 2-24 months after the operation, no evidence of metastases in the other 3 low-level expression patients up to now.

应用A673细胞系建立的肿瘤转移模型，单独应用外引物进行一次扩增，可以检测到10〓级肿瘤细胞，即可以从10〓个正常细胞中检测到1个肿瘤细胞；而在此基础上再次应用内引物扩增，则可以检测到10〓级肿瘤细胞，即可以从10〓个正常细胞中检测到1个肿瘤细胞；在用RNA模拟的肿瘤细胞转移模型中，两次扩增可以检测到10〓级肿瘤细胞，即可以从10〓个正常细胞中检测到1个肿瘤细胞。11例患者的15份标本中，2例没有检测到EWS/FLI1 mRNA的表达，分别随访8个月和30个月，没有发现临床转移的证据；对其余9例阳性表达的病例进行半定量的研究，6例EWS/FLI1 mRNA高表达的病例，分别在术后2-24个月发生转移，而3例EWS/FLI1 mRNA低表达的病例，则至今没有发现转移的证据。

Then Find Me A Maker Bye Girrll Shes Doing Resting On The Hours That Pass Her By Id Pay Attention If I Thought It Was Worth The Time I Tell Her Easy But Her Hands They Find A Way Confusing Passion For The Love We Never Gave Fall Back On Reasons That We Know Won't Stand A Chance Watching Her Shoulders Like A Memory From The Past I Tell Her Easy But Her Hands They Find A Way Confusing Passion For The Love We Never Gave I Couldn't Get Rid Of Shawty If Wanted Cause She Was Down But I Was Tired Of Playin The Role Like I Ain't Know That She Get Around Something About This Girl Facinates Me Shes The Type That Likes To Hang With The Stars The Way She Makes Me Feel And Sedates Me Who Knew We Would Take It This Far Everywhere That I Try To Go She Gotta Wanna Post Up With My Face On It Soon As I Walk Out The Door There It Is Wanna Postin Up With My Face On It Can't Blame My Self And Its Not My Fault Cause Nothing Was Promised At Least I Could Say I Was Honest But Bye Girll Shes Doing Resting On The Hours That Pass Her By Id Pay Attention If I Thought It Was Worth The Time I Tell Her Easy But Her Hands They Find A Way Confusing Passion For The Love We Never Gave Fall Back On Reasons That We Know Won't Stand A Chance Watching Her Shoulders Like A Memory From The Past I Tell Her Easy But Her Hands They Find A Way Confusing Passion For The Love We Never Gave Shes Doing Resting On The Hours That Pass Her By Id Pay Attention If I Thought It Was Worth The Time I Tell Her Easy But Her Hands They Find A Way Confusing Passion For The Love We Never Gave Fall Back On Reasons That We Know Won't Stand A Chance Watching Her Shoulders Like A Memory From The Past I Tell Her Easy But Her Hands They Find A Way Confusing Passion For The Love We Never Gave

法官或试图更改，因为你，你是谁我喜欢的是她沉溺于毒品，我不能离开我了她总是把计划……然后给我找一个日期，再见Girrll制造商她做上休息的时间，而过如果我想Id注意这是经得起时间的考验我告诉她她的手很简单，但他们找到一种方式热爱我们的爱情混乱从来没有放弃依靠的原因，我们知道没机会了看她的肩膀上像一个记忆从过去我告诉她她的手很简单，但他们找到一种方式热爱我们的爱情混乱从来没有放弃我无法摆脱Shawty如果希望因为她很失落，但我感到厌倦的角色球员像我不知道她四处走动一些关于这个女孩Facinates我喜欢的那种类型的，她总是和天上的星星她使我感到，我就知道我们会摇摆拿这么远的地方我试着去到处张贴，她必须要用我的脸一旦我走出房门那儿是想Postin与我的脸不能怪我自己和它的不是我的过错造成没有答应我至少可以说我很诚实，但再见Girll吗她做上休息的时间，而过如果我想Id注意这是经得起时间的考验我告诉她她的手很简单，但他们找到一种方式热爱我们的爱情混乱从来没有放弃依靠的原因，我们知道没机会了看她的肩膀上像一个记忆从过去我告诉她她的手很简单，但他们找到一种方式热爱我们的爱情混乱从来没有放弃她做上休息的时间，而过如果我想Id注意这是经得起时间的考验我告诉她她的手很简单，但他们找到一种方式热爱我们的爱情混乱从来没有放弃依靠的原因，我们知道没机会了看她的肩膀上像一个记忆从过去我告诉她她的手很简单，但他们找到一种方式热爱我们的爱情混乱从来没有放弃

Objective It was reported that rALR (rat augmenter of liver regeneration) distribute in most of tissue and organ of rat, no-tissue specility; mRNA expression of rALR was identified variedly in different tissue, strongest in testis. But nowadays there was not some study on ALR in rat testis. Here to confirm the expression and localization of ALR in infancy,adult and old rat testis,comparing the change of ALR expression degree before per 70 PH and after per 70 PH. In order to evaluate expression principle of ALR in rat testis and know if ALR has tight connection with spermatogenesis and development of testis, to explore the relation between ALR and reproduction, to discover the possible expression character and functional methods of ALR.

目的：文献报道rALR分布于各组织器官，无组织特异性，但其mRNA在各组织的表达强度有差异，在睾丸组织表达最强，目前对大鼠睾丸ALR表达尚无深入的研究；本实验研究不同年龄段大鼠睾丸组织中肝再生增强因子的表达水平、表达部位、以及表达的方式，进而探讨睾丸ALR与睾丸发育、精子生成以及生殖活动的关系；同时探究睾丸组织ALR表达是否参与了肝脏的再生过程中，进一步了解睾丸ALR表达的特性及作用方式。

From the angle of the dialectical relation of difference and identity, connecting further peoples real patterns of expression (the pattern of expression of the category, the pattern of expression of groups, the pattern of expression of the individuals), we may make the essence of human being to be particularized to three aspects of essence of the category, the groups and the individuals, these three aspects not only prescribe intrinsically the corresponding patterns of expression ofjustice (the justice of the category, the justice of the groups, the justice ofthe individuals), but also prescribe intrinsically the corresponding contents of development and ways of realization of justice.

从差异与同一的辩证关系角度，再结合人的现实的表现样态(类的表现样态、群体的表现样态、个体的表现样态)，我们可以把人的本质具体化为类本质、群体本质、个体本质三个方面，这三个方面不仅内在地规定着相应的正义的表现形态(类的正义、群体的正义、个体的正义)，而且内在地规定着相应正义的发展内容和实现方式。

Results 2 ClAdo and 2 CldAdo had different cytotoxicities on the cells, while 2' dAdo had no cytotoxicity on the cells. 2 CldAdo blocked the cell growth at the phase of S and G2/M, while 2 ClAdo at the phase of G0/G1. 2 CldAdo mainly induced cell necrosis, while 2 ClAdo induced both cell necrosis and apoptosis. Down regulated Bcl 2 expression and up regulated p53 and Bax expression were associated with 2 ClAdo, while 2 CldAdo down regulated Bcl 2 expression and up regulated mildly p53 expression without any effect on Bax expression.

结果 2 ClAdo、2 CldAdo对所研究的细胞均有程度不等的毒性；2' dAdo对各细胞生长、增殖均无明显抑制（c=100 μmol/L即30 μg/mL内）；2 CldAdo明显阻断细胞于S、G2/M期，2 ClAdo阻断细胞于G0/G1期；2 CldAdo 致细胞坏死为主，2 ClAdo 既致细胞坏死又诱导凋亡；2 ClAdo 下调Bcl 2，上调P53、Bax蛋白表达；2 CldAdo小幅上调P53、小幅下调Bcl 2，不影响Bax蛋白表达。

The positive expression both of p14ARF and mtp53 protein in NSCLC tissues were significantly different from that in nonmal lung tissues and tissues adjacent to the cancer (P.01).The expression of p14ARF protein was correlated with pathological degree（P＜0.01）,but it was uncorrelated with pathological staging,lymphaden metabasis and vessel invasion(P＞0.05).The expression of mtp53 protein was correlated with clinical pathology staging,pathological degree and vessel invasion(P＜0.05 or P＜0.01),but it was uncorrelated with lymphaden metabasis(P＞0.05).(2) The coexpression of p14ARF and mtp53 was 36.7％,their consistency was 38.8％,but the expression of p14ARF protein were negatively correlated with the expression of mtp53 protein(r=-0.3553,P＜0.001).Conclusion The results suggest that the decrease of p14ARF and the increase of mtp53 protein may play an important role in the genesis and development of NSCLC.

结果 (1)在正常肺组织、癌旁组织和NSCLC中p14ARF蛋白的阳性表达率分别为93.3%、83.3%、55.1%，mtp53蛋白的阳性表达率分别为6.7%、44.4%、79.6%；p14ARF蛋白和mtp53蛋白在NSCLC中的表达与正常肺组织和癌旁组织比较均有显著性差异(P＜0.01)；p14ARF蛋白表达与临床病理分期、有无淋巴结转移、有无脉管浸润无关(P＞0.05)，而与病理分化程度有关（P＜0.01）；mtp53蛋白表达与临床病理分期、病理分化程度(P＜0.01)及有无脉管浸润(P＜0.05)有关，而与有无淋巴结转移无关(P＞0.05)；(2)在NSCLC中，pl4ARF与mtp53蛋白共表达为36.7％，一致性为38.8％，二者表达呈负相关(r=-0.3553，P＜0.001)。

From the femur bone density analysis of protein expression, we can see that light chain lactoferrin, annexin decreased and the expression of enolase, ATP synthase, acetyl-coenzyme A reductase, the expression of troponin, may be related to ovariectomized femoral bone density in osteoporosis-related happened; strong bone of the femoral osteoporosis Po intervention effect may be related to reduced enolase, ATP synthase, troponin, CK-MB, acetyl phosphoglycerate enzyme, the expression of myosin, and the increase in lactoferrin light chain, pyruvate kinase isoenzyme, protein expression crown.

从股骨密质骨蛋白质组表达分析可知，乳铁蛋白轻链、膜联蛋白A3的表达下降及烯醇化酶、ATP合酶、乙酰辅酶A还原酶、肌钙蛋白的表达增强，可能与去卵巢后股骨密质骨骨质疏松的发生相关；强骨宝对股骨骨质疏松的干预效应可能与下调烯醇化酶、ATP合酶、肌钙蛋白、肌酸激酶同工酶、磷酸甘油酸变味酶、肌球蛋白的表达，和上调乳铁蛋白轻链、丙酮酸激酶同工酶、冠蛋白的表达有关。

Rosiglitazone can decrease the expression of RBP4mRNA in 3T3-L1adipocyte that used dexamethasone induced into insulin resistance model, and can improve the expression of GLUT4mRNA. 3. Rosiglitazone can increase the glucose intake in 3T3-L1 adipocyte. Chapter 2 Superficial Observed the Expression of Rebinding Protein 4 in OLETF rats OBJECTIVE Compare the level of the expression RBP4 between OLETF rats and LETO rats in adipose tissue; observe the function that rosiglitazone interfere in the expression of RBP4; observe the relationship between RBP4 and GLUT4, then approach the possible mechanism that RBP4 induce to insulin resistance.

第二章罗格列酮对OLETF鼠RBP4的影响，以及RBP4在OLETF鼠内脏脂肪表达的初步观察比较自发性2型糖尿病OLETF鼠与同家系LETO鼠脂肪组织视黄醇结合蛋白(RBP4)水平；观察罗格列酮对RBP4的干预作用；观察脂肪组织RBP4与GLUT4水平的相关性，从而探讨RBP4导致胰岛素抵抗的可能机制。

As of Tuesday, Google's results were still censored in China.

截至周二，谷歌的搜索结果仍受中国审查。

In order to make the positive action increase and negative one decrease, the sub-forces of the social factors must be adjusted to form a centripetal force.

在这一过程中，人的主体性发挥是社会有机体健康发展的灵魂。