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fatty degeneration相关的网络例句

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与 fatty degeneration 相关的网络例句 [注:此内容来源于网络,仅供参考]

The rates of fatty degeneration, cytoplasmic rarefaction and balooning degeneration in lithogenous group were 77.8%, 44.6%and 33.3%, but in antilithic group they were only 15.4%,23.9%and 7.7%.

致石组动物肝脏脂肪变性、胞质疏松、气球样变的发生率分别为77.8%,44.6%,33.3%,而防石组仅为15.4%,23.9%,7.7%。

Results The liver tissues of the rats in model group showed obvious hydropic and fatty degeneration.And the necrosis foci of hepatocytes were seen in liver tissue.Hepatocytes in curcumine treatment group showed no necrotic changes,and the degree of hydropic and fatty degeneration was markedly reduced,especially in the high-dose curcumine treatment group.

结果 模型组大鼠的肝组织呈现明显的水样变性和脂肪变性,并可见肝细胞坏死灶,姜黄素治疗组的肝细胞未见坏死改变,水样变性和脂肪变性程度减轻,以高剂量姜黄素组的治疗效果最为明显。

RESULTS The proliferation of fibroblasts was inhibited by K102 in vitro. A dose-dependent antagonistic effect was found between K102 and bFGF. The serum levels of ALT, AST and the liver hydroxyproline level in the treated groups were lower than that in the CCl4 model group. Pathological examination showed a slight fatty degeneration and hydropic degeneration was observed in the group treated with the high dose K102, the low dose K102, and the control group.

结果体外生物活性检测:K102明显抑制成纤维细胞的生长,bFGF对K102有一定拮抗作用,且具有剂量依赖性;体内生物活性检测:治疗给药组血清ALT,AST和肝组织羟脯氨酸含量较CCl4模型组明显改善。

Participants and methods 1、All participants were diagnosed with NAFLD byabdominal ultrasonography in lst affiliated hospital of JI NAN University frommay 2006 to march 2007. 2、All participants had no history of viral hepatitis orexcessive drinking(alcohol intake less than 140g per week for male and 70g perweek for female). 3、Detail history, anthropometrical parameters (height,weight,waistline and hipline). A B-mode ultrasonic examination was performed todetermine the liver fat content and participants were divided into the following threegroups based on their liver fat content: mild fatty degeneration group, moderate fattydegeneration group,severe fatty degeneration group.

对象与方法1、本研究以2006年9月至2007年3月在暨南大学附属第一医院内分泌科门诊、消化科门诊和体检中心初诊为NAFLD者为研究对象,总共有NAFLD组病人405例,男性232例,女性173例,年龄在20-80岁之间,平均年龄56.6±6.89岁,按B超的检查结果NAFLD组病人分为轻度NAFLD组、中度NAFLD组、重度NAFLD组,各组性别构成和年龄差异无统计学意义。2、选择年龄在20-80岁之间的健康体检者并行B超检查排除NAFLD者为对照组,共119例。

It was suggested that the pharmacodynamics and therapeutic function of Qizhi complex prescription to fatty liver were: 1. Adjust lipometabolism: reducing TC and TG; 2. Decreasing fatty degeneration of liver cell; 3. Promoting the convalescence of hepatocellular function; 4. Anti-lipid peroxide: increasing the activation of peroxidase, decreasing the content of products of lipid peroxide. 5. Restraining Kupffer cell function of fatty liver rat; 6. Improving fluidity of hepatocellular microsome and mitochondria.

芪枳合剂治疗脂肪肝的效应及药效学机制为: 1、调整脂质代谢:降低血清甘油三脂和胆固醇; 2、减轻肝细胞脂肪变性; 3、促进肝细胞功能恢复,有效治疗脂肪性肝炎; 4、抗脂质过氧化:提高抗氧化酶活性、降低脂质过氧化产物含量; 5、抑制脂肪肝大鼠Kupffer细胞功能; 6、改善肝细胞微粒体、线粒体膜流动性。

The pharmacodynamics and therapeutic function of Ganzhixiao decocting prescription to fatty liver as the following:1. Adjust lipometabolism:reducing TC and TG;Decreasing fatty degeneration of liver cell;2. promoting the convalescence of hepatocellular function;3. Anti-lipid peroxide:increasing the the activation of peroxidase, decreasing the content of productsof lipid peroxide.

肝脂消颗粒剂治疗脂肪肝的效应及药效学机制为: 1、调整脂质代谢:使肝酶活性增强,降低血清甘油三脂和胆固醇; 2、减轻肝细胞脂肪变牲; 3、促进肝细胞功能恢复,有效治疗脂肪性肝炎; 4、抗脂质过氧化:提高抗氧化酶活性、降低脂质过氧化产物含量。

Results The incidence of fatty degeneration among all the 3000 cases liver biopsied specimens was 12.7%, and the incidence of nonalcoholic fatty liver disease was about 6.1%.

结果在所有3000例行肝活检的病例中,出现肝细胞脂肪变的病例约占12.7%,而NAFLD的发生率约6.1%。

Sinusoid from the fifth week of expansion, swelling and degeneration of liver cells has varying degrees of fatty degeneration, infiltration of inflammatory cells at the same time.

从第5周起肝窦扩张,肝细胞变性肿胀并有不同程度的脂肪变性,同时出现炎症细胞浸润。

ResultsThe incidence of fatty degeneration among all the 3000 cases liver biopsied specimens was 12.7%, and the incidence of nonalcoholic fatty liver disease was about 6.1%.

结果在所有3 000例行肝活检的病例中,出现肝细胞脂肪变的病例约占12.7%,而NAFLD的发生率约6.1%。

Results: The lipid particles can significantly reduce the non-alcoholic fatty liver rat model of liver fat level, to improve the fatty degeneration and reduce the liver inflammation.

结果:降脂颗粒能明显降低非酒精性脂肪肝大鼠模型肝脂的水平,改善脂肪变性,降低肝脏炎症反应。

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