查询词典 fat cell

The animal model of this method system have, the characteristics of the high fat, high leptin, high Ins, and match with the characteristic of clinical and the pathologic of simple obesity. This studies showed: The acupuncture can obviously reduce the vaule of intaking food and water of the fat rats, and lower the body weight and fat; The acupuncture can urge the diameter, area, physical volume of white fat cell contract, Brown fat tissue of fat rat group surroundings vascular containing large quantity of white fat cell, the acupuncture group was less. this showed: The acupuncture enhanced the white fat cell metabolism, and promotesed the white fat cell to convert to brown fat cell; The frequency of spontaneous electric of nerve cell of pvn of the fat rat is high, the acupuncture counld depress the spontaneous electric of nerve cell of pvn, This showed: the acupuncture can depress the appetite passing to adjust function of pvn nerve; The monoaminergic neurotransmitter of inside of the fat rat brain is disfunction, the acupuncture can rectify this disfunction, and adjust level of 5-HT, and affect appetite, and improve metabolism; The level of leptin and Ins of fat rat increased high, The level of it of acupuncture group decreased, compared with the fat group p.001, This showed: the fat rats have the defeat of leptin and Ins in the body, The acupuncture can lowering this defeat, The level of leptin and Ins of the fat rat in the brain is less than the normal group, The level of leptin of the acupuncture group is high than the fat group, This showed: the acupuncture can increase the transportation of leptin from blood to brain.

本方法所制肥胖大鼠模型具有高体重、高脂、高leptin和高Ins血症的特点，符合人类单纯性肥胖病的临床和病理特征；实验研究显示：针灸能明显减少肥胖大鼠的摄水、摄食量及降低其体重、体脂；针灸能促使白色脂肪细胞直径、面积、体积缩小，减少肥胖大鼠棕色脂肪组织血管周围白色脂肪细胞数量，显示：针灸增强了白色脂肪细胞的代谢，促进了白色脂肪细胞向棕色脂肪细胞的转化：肥胖大鼠PVN神经细胞自发放电频率增加，脑内单胺递质紊乱，针灸通过抑制PVN亢奋的自发放电，调整5-HT水平，达到影响食欲，改善代谢的作用；肥胖大鼠血中leptin及Ins水平增高，与正常组大鼠比有明显差异，提示大鼠体内存在leptin及Ins抵抗，针灸后肥胖大鼠随着体重体脂的下降，血中leptin、Ins水平降低，与肥胖组比均有显著性差异，而针灸后大鼠脑内leptin增加，与肥胖组比P.05，提示：针灸具有改善leptin、Ins抵抗及增加leptin脑内转运的作用。

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Due to the complexity of the cell jitter, the NonSynchronous Tining Recovery methods are currently not mature With the emphasis being given to the Class A CBR traffic, this paper analyzes the performance of the queueing delay and cell jitter at the source node and intermediate nodes, and discusses the Source Timing Recovery at the destination node in ATM networks Firstly, this paper presents a description of the cell jitter of CBR traffic, and gives the definitions of two kinds of cell jitter regarding the Source Timing Recovery for CBR traffic Then, by using exact mathematical models and analysis methods, this paper analyzes the impact of the factors, such as the capacity of the queueing buffer, the randomness, the deterministic nature and the correlation in cell arrivals of the background traffic sources, on the queueing delay and cell jitter performance of the CBR traffic through Statistical Multiplexitng To obtain an insight into the power spectral distribution and look for better schemes for the depression and filtering of the cell jitter, within the analyses we succeed deriving the power spectrum of the cell jitter for CBR traffic Hence, not only the power spectral distribution of the cell jitter can in the frequency domain be qualitatively understood, but also can the rms (root-meansquare) value of the cell jitter be quantitatively obtained so as to more accurately measure the amplitude of the jitter In the end-to-end performance analysis of the queueing delay and cell jitter, we propose a kind of quasi-periodic cell stream model to characterize the jittered CBR traffic, and present an initial queueing analysis of the CBR traffic following such a model at a generic intermediate node Additionally, we briefly discuss the buildout/playout and Source Timing Recovery functions of the destination node Finally, regarding the Source Timing Recovery of CBR traffic, this paper systematically discusses several important principles of the cell jitter filtering and depression reported in the literature, introduces several implementation schemes of the Source Timing Recovery e.

由于信元抖动的复杂性，非同步定时恢复方法目前还很不成熟。本文针对A类CBR业务流在ATM网络源节点和中间节点的排队时延和信元抖动性能，以及在目的节点的源定时恢复问题作了较为全面的研究。首先，文中描述了CBR业务流的信元抖动，并具体地给出了两种与CBR业务源定时恢复有关的信元抖动的定义。然后，采用了精确的数学模型和分析方法，有针对性地分析了业务背景中信元到达的纯随机性、确定性和相关性以及排队缓存器容量等因素对CBR业务流经过统计复用后的排队时延和信元抖动性能的影响。为了了解信元抖动的功率频谱分布和寻求更好的抑制和滤除抖动的方法，在性能分析中，我们成功地完成了CBR业务流信元抖动的功率谱分析，使得不但可以从频域定性地认识信元抖动的能量分布特性，而且还可以定量地求出信元抖动的均方根值(rms：root-mean-square)，以更为准确地衡量抖动的大小。在CBR业务流的多节点端-端排队时延和信元抖动性能分析中，我们提出了一种准周期性(quasi-periodic)信元流模型来描述感染了信元抖动的CBR业务流，并基于这一模型进行了CBR业务流中间节点的初步排队分析。

Chairman Mr. Cai Zilai Units from one sewing machine, 8000 yuan start business after several years of steady, and now has developed into a more than 100 high-speed machines, complete range of professional machinery, staff of nearly 200 people, on tens of millions of enterprise assets .2005 Companies also targeting fat people without professional clothing manufacturer blank market, people invest in the establishment of Wuhan fat Zhuang Fu apparel company, the company's technology is strong in the development of powerful, in the spirit of professional services groups of fat people, fat people to shape the purpose of beauty, developing a series of fat people fit the particular body style, the Han are the only faction apparel specializing in the production of fat people apparel manufacturers, we strive to create excellent Chinese designed and manufactured for the fat people fashion clothing business, for the cause of fat people to create the blue sky.

董事长蔡子来先生由一台缝纫机，8000元钱开始创业，经过几年稳扎稳打，现在已经发展成为拥有100多台高速机，各种专业机械齐全，员工近200人，资产上千万的企业。2005年公司又瞄准胖人服装无专业生产厂家的空白市场，投资成立武汉胖人福庄服饰公司，公司技术力量雄厚，开发力量强大，本着专业服务胖人群体，塑造美丽胖人的目的，开发出一系列适合胖人特殊体型的款式，是汉派服装中唯一专业生产胖人服饰的厂家，我们力争创中国优秀的为胖人设计生产时尚流行的服装企业，为胖人事业共创蓝天。

1、Cur inhibits K562 cells growth and induces cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as p-Erk1/2、c-myc which are relevant with cell growth and apoptosis; 2、Cur synergizes STI571 to inhibit K562 cell growth and induce cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as Hsp90、PKC which are relevant with cell growth and apoptosis; 3、Cur reverses the resistance of K562/G01 cells to STI571, and synergizes STI571 to inhibit K562/G01 cell growth and induce cell apoptosis; 4、Cur inhibits human originated CML CD34~+ cell growth、induces cell apoptosis, and enhances STI571 to down-regulate the expression of p210~, finally inhibit cell growth and induce cell apoptosis.

从以上实验结果我们得出如下结论： 1、Cur抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游p-Erk1/2、c-myc等信号分子有关； 2、Cur协同STI571抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制Hsp90和下游PKC等信号分子有关； 3、Cur可逆转K562/G01细胞对STI571的耐药性，并与STI571协同抑制K562/G01细胞增殖和诱导凋亡，其抑制K562/G01细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游Procaspase-3和NF-κB等信号分子有关； 4、Cur可抑制来源于CML患者骨髓的CD34~+细胞的增殖并诱导其凋亡，还可协同STI571下调CML CD34~+细胞p210~表达，进而协同抑制细胞增殖、诱导细胞凋亡。

Higher Ca distributed in bulliformcell than in mesophyll cell and bundle sheath cell of dune reed, higher Mg distributedin mesophyll cell and higher K, Na and Cl distributed in sheath cell HigherNa and Mg distributed in mesophyll than in bulliform cell and bundle sheathcell of light salt meadow reed, and higher K, Ca and Cl distributed in itsbundle sheath cell. Higher Na and Mg distributed in bulliform cell than in mesophyllcell and bundle sheath cell of heavy salt meadow reed, higher K, Ca and Cl distributedin its mesophyll cell. This paper discussed the distribution conditions of theabove five ions in leaf cell of the four reed ecotypes and the meaning ofphysiological adaptation to habitat in detail.

沙丘芦苇的泡状细胞内Ca分布较叶肉细胞和鞘细胞高，叶细胞内Mg分布较高，在鞘细胞内K，Na和Cl布较高；轻度盐化草甸芦苇叶肉细胞内分布了较多的Na和Mg，在鞘细胞内K，Ca和C1分布较叶肉细胞和泡状细胞高；而重度盐化草甸芦苇泡状细胞内分布了较多的Na和Mg，叶肉细胞分布了较多的K，Ca和Cl；详细讨论了以上五种离子在不同生态型芦苇叶片内不同细胞类型的分布状况与环境适应的意义。

In the gravid uterus, The CKs immunolabelling were detected in glandular cell, luminal epithelial cell, traphoblast cell, endoblastic cell and allantoic cell; Vimentin immunolabelling were detected in stromal cell and endoblastic cell; CK7 immunolabelling were not detected in any tissue of the yak utenus but in endoblastic cell and some luminal epithelial cell.

对分离得到的子宫内膜基质细胞和子宫内膜腺上皮细胞进行免疫组织化学标记的结果显示在体外子宫内膜基质细胞表达泛角蛋白，子宫内膜腺上皮细胞表达波形蛋白，并且这一特性不因为传代而发生丢失。

2B8a was weakly reactive to neutrophils (23.72%) and negative for T cells, NK, DC, RBC and Plt. The antibody reacted to all 3 marrow CD34+ cells with an average positive rate of 39.33% while it was negative for G-CSF-mobilized CD34+ peripheral blood stem/progenitor cells (PBSC, 1.25%). Cell line analysis showed that the antibody notably reacted to three out of 4 cell lines (Raji, SMS-SB, Nalm-6 and Nall-1) with the positive rates of 98.78%, 98.61%, 94.93% respectively and weakly to one of them with 5.68% in B lineage cell lines and monoblastic cell line (U937, 67.78%) while it was only weakly positive or negative for other myeloid leukemia cell lines including Meg01 (33.40%), HL-60 (29.70%),K562 (28.19%), KG1a (16.23 %) and HEL92.1.7 (8.02%). Among 4 T lineage leukemia,5 neuroblastoma and 1 colon cancer cell lines tested, only Molt-3 was found weakly positive (31.40%) for 2B8a, while the remaining 3 T cell lines (Molt4, JM and CCRF-CEM), 5 neuroblastoma cell lines (LA-N1, KCNR, BE, SK-N-SH, SK-N-AS) and the colon cancer cell line (HR8348) tested were negative.

结果表明： 2B8a抗原在外周血B细胞上表达（3/3例，平均阳性细胞数为26.29 ％），而在T淋巴细胞和NK细胞上不表达（0/3例）；在粒细胞和单核细胞上阳性表达均为2/3例，平均阳性细胞数分别是23.72 ％和59.84 ％；在DC细胞、红细胞和血小板上均不表达（0/3例）。2B8a抗原在骨髓CD34＋细胞上的阳性表达是3/3例，平均阳性细胞数39.33 ％，而在G-CSF动员的外周血CD34+细胞上的阳性表达仅1/3例，平均阳性细胞数为1.25 ％。2B8a抗原在B系细胞系Raji、SMS-SB、Nalm-6和Nall-1上的平均阳性细胞数分别为98.78 ％、98.61 ％、94.93 ％和5.68 ％；在T系细胞系Molt-3上的平均阳性细胞数为31.40 ％，而在Molt-4、JM和CCRF-CEM 细胞上不表达；在髓系细胞系U937、Meg-01、HL-60、K562、KG1a和HEL92.1.7上的平均阳性细胞数分别为67.78 ％、33.40 ％、29.70 ％、28.19 ％、16.23 ％和8.02 ％；在神经母细胞瘤细胞系SK-N-SH、KCNR、BE、LAN-1和SK-N-AS细胞以及结肠癌细胞系HR8348细胞上均不表达，而在羊膜细胞系FL细胞上呈一定的阳性表达，平均阳性细胞数为45.03％。

Cell line analysis showed that the antibody notably reacted to three out of 4 cell lines (Raji, SMS-SB, Nalm-6 and Nall-1) with the positive rates of 98.78%, 98.61%, 94.93% respectively and weakly to one of them with 5.68% in B lineage cell lines and monoblastic cell line (U937, 67.78%) while it was only weakly positive or negative for other myeloid leukemia cell lines including Meg01 (33.40%), HL-60 (29.70%),K562 (28.19%), KG1a (16.23 %) and HEL92.1.7 (8.02%). Among 4 T lineage leukemia,5 neuroblastoma and 1 colon cancer cell lines tested, only Molt-3 was found weakly positive (31.40%) for 2B8a, while the remaining 3 T cell lines (Molt4, JM and CCRF-CEM), 5 neuroblastoma cell lines (LA-N1, KCNR, BE, SK-N-SH, SK-N-AS) and the colon cancer cell line (HR8348) tested were negative.

结果表明： 2B8a抗原在外周血B细胞上表达（3/3例，平均阳性细胞数为26.29 ％），而在T淋巴细胞和NK细胞上不表达（0/3例）；在粒细胞和单核细胞上阳性表达均为2/3例，平均阳性细胞数分别是23.72 ％和59.84 ％；在DC细胞、红细胞和血小板上均不表达（0/3例）。2B8a抗原在骨髓CD34＋细胞上的阳性表达是3/3例，平均阳性细胞数39.33 ％，而在G-CSF动员的外周血CD34 细胞上的阳性表达仅1/3例，平均阳性细胞数为1.25 ％。2B8a抗原在B系细胞系Raji、SMS-SB、Nalm-6和Nall-1上的平均阳性细胞数分别为98.78 ％、98.61 ％、94.93 ％和5.68 ％；在T系细胞系Molt-3上的平均阳性细胞数为31.40 ％，而在Molt-4、JM和CCRF-CEM 细胞上不表达；在髓系细胞系U937、Meg-01、HL-60、K562、KG1a和HEL92.1.7上的平均阳性细胞数分别为67.78 ％、33.40 ％、29.70 ％、28.19 ％、16.23 ％和8.02 ％；在神经母细胞瘤细胞系SK-N-SH、KCNR、BE、LAN-1和SK-N-AS细胞以及结肠癌细胞系HR8348细胞上均不表达，而在羊膜细胞系FL细胞上呈一定的阳性表达，平均阳性细胞数为45.03％。

HD-G Multifunctional Slimming Machine combine main and collateral channels and acupuncture therapy,with modern computer technology,Control electric pulse by the procedure,simulate acupunture massage acupunture point,can make human's body organ relating physiological reaction,adjust bio computer and metabolism,increase human body's lactate dehydrogrnase,adjust human body's incretion,improve human body's metabolism, slow down the speed of glucose transformed into fat,especially,to paly the important part to fat metabolism,can hydrolyze most 3 sour and sweet axunge,and make its leave fat cell,through a series of special decomposition approach, 3 sour and sweet axunge will be transfromed into energy Co2 and water being consumed,that fat cell volume will be small,that effect in weight loss and tightening skin

HD-G美体减肥仪器结合了中医的经络学和针灸理疗学，应用现代电脑技术，通过程序控制电子脉冲，模拟针灸按摩穴位，让人体相关的器脏产生相应的生理反应，有效调解生物电脑和新陈代谢，增加人体内乳酸脱氢酶活力，彻底调整人体内分泌，改善人体新陈代谢，减缓葡萄糖转化成脂肪的速度，特别是对脂肪代谢的分解发挥作用，可使原有脂肪中绝大部分的三酸甘油脂水解成脂肪酸和甘油，并使之离开脂肪细胞，通过一系列特殊分解途径，最好转化为能量二氧化碳和水被消耗。从而使脂肪细胞体积变小，达到减肥收紧皮肤的效果。

I didn't say that you can see this news in DA forum.

他没说你可以在论坛上看到这新闻。

The high performance liquid chromatograph shows that the final hydrolysis products include glucose and acetic acid.

等离子体与淀粉溶液反应产生的酸对淀粉水解的贡献很小。