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epithelial cells相关的网络例句

查询词典 epithelial cells

与 epithelial cells 相关的网络例句 [注:此内容来源于网络,仅供参考]

When SA-bFGF-Math1 transferred to the epithelial cells(UEC-4) of utricle,these cells showed the potential to differentiate hair-cell-like cells.

通过脂质体将bFGF-Math1转染大鼠椭圆囊斑的上皮细胞(UEC-4),可使其分化为新毛细胞。

During the telogen or resting phase of the hair cycle, the epithelial strand subsequently shortens to the level of the arrector pili muscle and leaves in its wake a small aggregate of epithelial cells exposed to surrounding dermis.

杵状毛一直留在缩小的毛囊内,直到有新的生长期毛囊发育。

Moreover, expression of a constitutively active form of Notch4 in mammary epithelial cells inhibits epithelial differentiation and leads to tumor formation in this organ.

再者,乳腺上皮细胞中一种持续活化形式Notch4的表达也能抑制上皮细胞的分化,从而导致肿瘤形成。

At 24 hours after the removal of 5-FU, most of the epithelial cells were increased in number, showing cuboidal and merged into pieces. At 48 hours, only few positive cells could be seen with some of the pseudostratified mucociliary epithelium, with ciliated cells.

氟尿嘧啶去除6h后,气管基底膜上可见少量扁平样细胞。24h后基底膜上细胞数目增加,并逐渐分化呈立方状,连接成片。48h后气管上皮中出现假复层柱状上皮,并可见纤毛细胞。

It is well known that retinal pigment epithelial cells are predominant proliferative cells in PVR which is an excessively wound healing response occurring after RPE cell wounding. The activated RPE cells migrate from their normal, sessile state to vitreous and onto both surfaces of the retina, in which they begin to dedifferentiate, migrate, proliferate, change phenotype and secrete ECM.

既往研究证明视网膜色素上皮(retinal pigmentepithelial,RPE)细胞是参与PVR的主要细胞,PVR是视网膜脱离后RPE细胞损伤的一种自我修复过程,是一种过度的眼内创伤修复反应;RPE细胞在正常位置上处于静止状态,而在某些病理条件下,如视网膜裂孔形成和视网膜脱离后,RPE细胞脱离原位,开始去分化、移行、增生、发生表型转化并分泌胶原等ECM,最终在视网膜前后表面和玻璃体内形成具有收缩能力的增生膜,造成牵拉性视网膜脱离。

Meanwhile the lung tissue slide was prepared for electron microscopic observation at 180 minutes after reperfusion.Result: HO-1 expression was upregulated in two groups in the pulmonary endothelial cells,part of pulmonary vascular smooth muscle cells,extima of vessels and epithelial cells of airway,the absorbance was 0.148±0.013,0.158±0.012,respectively.The Shenmai injection group showed higher absorbance than those of the IRI group( P .01),lower W/D and IAR values than those of the IRI group( P .01) significantly and lighter abnormal changes of the lung tissue in morphologically than those of the PIRI group.

结果:HO-1在I-R组、SM组肺血管内皮、部分血管平滑肌、外膜层及部分气道上皮均有阳性表达,原位杂交的平均吸光度分别为0.148±0.013,0.158±0.012,SM组HO-1的表达水平明显高于I-R组( P 。01),W/D,IAR值显著低于I-R组( P 。01),肺组织形态学异常改变轻于I-R组。

Theurinary IL-6 level positively correlated with density of glomerular matrix membrane, global sclerosis, fiber or fibrocellular crescents and interstitial fibrosis (p. 05). According to the degree of density of glomerular matrix membrane and interstitial fibrosis, urinary Col-IV level had better correlation than urinary TGF-betal and IL-6 levels.In IgAN, Col-IV showed increased expression in diseased renal tissue whereas the site of expression of TGF-betal was mainly localized within the cytoplasm of tubular epithelial cells. Interstial expressionwas also present but glomerular TGF-betal expression was found only in patients with heavy mesangial proliferation. There was a significant correlation between glomerular positivity for Col-IV and severity of histological damage. There was also a significant correlation between positivity for TGF-betal and Col-IV in the tubular epithelial and interstitial lesions. In contrast, there was no ralationship between glomerular positivity for TGF-betal and severity of histological damage.The urinary TGF-betal level paralleled tubular TGF-betal expression.

结果 ①IgAN患者尿TGF-β1、IL-6、Col-Ⅳ水平较健康人明显增高(P<0.01),该变化与血中的浓度无关(P>0.05);②尿TGF-β1水平与小管间质TGF-β1阳性表达呈正相关(P=0.000),而与小球TGF-β1阳性表达无关(P>0.05),尿Col-Ⅳ水平与小球和小管间质Col-Ⅳ阳性表达均呈良好的相关性(P<0.01),还与小管间质TGF-β1阳性表达呈正相关(P<0.05):③小球Col-Ⅳ阳性表达与肾组织慢性病变密切相关(P<0.05),小管间质Col-Ⅳ和TGF-β1阳性表达均与肾小管间质病变呈良好的相关性(P<0.01),而小球TGF-β1阳性表达与肾组织损伤无关(P>0.05);④尿TGF-β1、Col-Ⅳ水平与肾小球基质基底膜面密度、小管间质病变呈正相关(P<0.01),与小球内细胞数呈负相关(P<0.05),该结果与其在组织中的表达一致;尿IL-6水平浙江大学硕士学位论文尿TGF一B一、IL一6和Col一IV在IgA肾病中的应用价值与基质基底膜面密度、球性硬化、纤维或细胞纤维新月体所占肾小球百分数及小管间质病变均有显著的相关性(F.05);在轻度肾病理损伤时,尿'l'G卜pl、I卜6、Col一IV水平即升高,而尿Col一W在反映细胞外基质积聚和间质纤维化程度上比尿TGF一pl和IL一6有更好的相关性。

M^2, p=0.46. The study showed no difference between the juvenile macromastic breast and non-tumor part of cancer breast in mammary epithelial cells, suggesting that the cause of juvenile macromastia may be from factors other than abnormal mammary epithelial cell size.

69.5±10.0μm^2, p=0.46。本研究发现青春期巨乳症的乳房组织与乳癌病患未受癌症感染之乳房组织,在上皮细胞之尺寸方面并没有差异,青春期巨乳症的成因可能是与乳房上皮细胞之生长较为相关。

Natural monoclonal antikeratin antibody IgM 3B4 can inhibit the adhesion of Candida albicans to the malpighian cells, human buccal epithelial cells and endothelial cells of fetal umbilical vein, and decrease the invasion of Candida albicans due to its inhibitory action on the formation of Candida albicans germ tube,which indicates that natural antibody IgM plays a role in the natural immunity against yeast infection.

单克隆天然抗角蛋白抗体IgM 3B4在体外能够抑制白念珠菌芽管形成,能够抑制白念珠菌与人角质形成细胞、人口腔黏膜上皮细胞、胎儿脐静脉内皮细胞的粘附,从而降低白念珠菌的侵袭力,提示天然抗体IgM在机体抗真菌感染的天然免疫中可能具有积极的作用。

RESULTS: It was found that the different concentrations of natural monoclonal anti-keratin antibody IgM 3B4 could inhibit Candida albicans germ tube formation and adhesion of Candida albicans to malpighian cells, human buccal epithelial cells and endothelial cells of fetal umbilical vein.

单克隆天然抗角蛋白抗体IgM3B4在体外能够抑制白念珠菌芽管形成,能够抑制白念珠菌与人角质形成细胞、人口腔黏膜上皮细胞、胎儿脐静脉内皮细胞的粘附,从而降低白念珠菌的侵袭力,提示天然抗体IgM在机体抗真菌感染的天然免疫中可能具有积极的作用。

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On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面,更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值,分为几个相等的区间作为横轴,并将各区间内所测定值依所出现的次数累积而成的面积,用柱子排起来的图形,也叫做柱状图。

You rap, you know we are not so good at rapping, huh?

你唱吧,你也知道我们并不那么擅长说唱,对吧?