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epilepsy相关的网络例句

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RESULTS Based on the epilepsy typing method and Video-EEG data,54pa-tients(42.2%)were diagnosed for epilepsy syndrome in the128child patients,including25cases of benign epilepsy of children with centrotemporal spikes,3cases of childhood ab-sence epilepsy,1case of juvenile myoclonic epilepsy,5cases of frontal lobe epilepsy,13cases of West syndrome and7cases of Lennox-Gastaut syndrome.The types of epilepsy seizures in49(38.3%)patients were conformed,including10cases of tonic and/or clonic seizures,33cases of partial seizures,5cases of myoclonic seizures and1cases of atonic seizures.

结果 128例癫痫患儿中有54例(42.2%)进一步确定了癫痫综合征的诊断,其中儿童良性癫痫伴中央-颞区棘波25例,儿童失神癫痫3例,少年肌阵挛癫痫1例,额叶癫痫5例,West综合征13例,Lennox-Gastaut综合征7例;有49例(38.3%)可明确其发作类型,其中强直或阵挛发作10例,部分性发作33例,肌阵挛发作5例,失张力发作1例,另外有25例(19.5%)尚无法分类。

Moutains of evidences from laboratary and clinical data show that gene abnormality is the major etiological factor of over 40% patients affected with epilepsy.There were also 6 epilepsy genes been cloned successfully, and mutations of over 1000 genes maybe related to epilepsy. These abnormal genes influence many aspects from molecular level to the neuronal plasticity, such as the development of brain,neuronic degeneration, remodeling of neural circuits, energy metabolism, ion channel and so on.These factors would at last affect the formation of epilepsy focus, propagations of epileptic potentials, vulnerability of epilepsy cells.

大量的实验和临床资料提示基因异常是40%以上癫痫患者的病因,有6种常见全身癫痫的基因已被成功克隆,1000种以上的基因突变可能与癫痫的发作有关,这些表达异常的基因分布在脑发育、神经元变性及神经环路重组、能量代谢、离子通道等多个环节,从分子、细胞、神经元可塑性等多个方面影响着癫痫灶的形成、痫性放电的扩布及癫痫细胞的损伤,构成了癫痫的基因机制。

Hewever the caspase-3 expression was still very high in cortex. Conclusion: In KA model the hippocampus amygdala plays a key role in epilepsy. Therefore it provides a mimic optimal conditions for human temporal lobe epilepsy study. Its an effective method of studying the development, change and pathopoiesis of epilepsy. There is a close relationship between NPY and epilepsy. The regulation of NPY via VPA may be one of the antiepilepsy mechanism of mechanism of VPA. Moreover NGF can enhance the caspase-3 expression. Excito-amino acid can aggravate the apoptosis, which may be one of the reason for intractability of epilepsy.

KA模型因其海马杏仁核在癫痈中起核心作用,故具有模拟人类颗叶癫痈的理想条件,是研究癫痈发展变化、致痈机理的有效工具,NPY与癫病有着密切的联系,VPA调节NPY有可能是其抗癫痈机理之一,而NGF对caspase一3表达有促进作用,兴奋性氨基酸可造成细胞凋亡的增加,而凋亡细胞的增多有可能是癫痈久治不愈的原因之一。

Disorders using International League Against Epilepsy classifications, such as Lennox-Gastaut syndrome, benign childhood epilepsy with centrotemporal spikes, childhood absence epilepsy and juvenile myoclonic epilepsy are increasingly being recognised in Hong Kong.

根据国际抗癫痫联盟的分类命名,诸如 Lennox-Gastaut 综合徵、伴中央颞区棘波的儿童期良性癫痫、儿童失神癫痫及青少年肌阵挛性癫痫等病在香港正逐渐被认识。

Methods We retrospectively studied clinical and EEC features of 80 patients with ICE. Ten patients were diagnosed with juvenile myoclonic epilepsy, 35 has absence epilepsy, 28 had generalized tonic clonic seizure, and 7 had adult-onset idiopathic generalized epilepsy.

回顾性分析80例特发性癫痫患者的临床和常规EEG特征。10例患者诊断为青少年肌痉挛癫痫,35例患者有失神发作,28例患者为全面强直痉挛发作,7例患者为成年期起病的特发性全面性癫痫。

In recent years,mutations of gene encoding gamma-aminobutyric acid A-receptor γ2 subunit (GABRG2) were founded linked to many kinds of epilepsy syndromes such as Febrile Seizure, Generalized Epilepsy with Febrile Seizure plus, Childhood Absence Epilepy, Severe Myoclonic Epilepsy of Infancy.

近年来许多学者通过体外扩增基因等研究发现在许多种癫痫综合征患者种都存在编码γ-氨基丁酸A型受体γ亚单位的基因异常,进而提出了GABRG2基因异常与多种癫痫的发病有关。

Intergrading the experts on neurosurgery,neurology, pediatrics ,neuroradiology,neuropsychology,neuroimaging ,clinical pharmacy , the epilepsy center in SAH-DMU is the only epilepsy center which provides the multidisciplinary comprehensive treatment in Northeast China including clinical standard diagnosis and evaluation,systematic standard medication,surgery for refractory epilepsy,psychology intervention and so on.

整合了神经内科、神经外科、神经儿科、神经影像、神经心理、神经电生理及临床药学等多学科专家,同时开展癫痫的临床诊断、评估,药物的系统、规范化治疗,顽固性癫痫的手术治疗及心理干预治疗等综合治疗方案,是目前东北三省唯一能提供癫痫的多学科专业综合治疗的癫痫中心。

Methods: Adopting pig bile culture medium, the inhibiting or killing effects on pig bladder worm in vitro were observed;by using the methods of xylene-induced ear inflammation in mice and granuloma swelling induced by cotton ball in rats, the effects of anti-inflammation were observed;by using the methods of body twisting and hot-plate in mice, the effects of analgesia were observed. Through the mice auricle microcycle, the rats thrombus law in vitro and bleeding time, clotting time determination to the mice, the function of invigorating the blood were observed;adopting the penicillin abdominal cavity injection method, the mice epilepsy model were prepared, and epilepsy mice brain organization MDA content and SOD, ATPase vigor be measured by using chemical colorimetry.

利用猪胆汁培养基观察该方在体外对猪囊尾蚴的抑制或杀灭作用;采用二甲苯所致小鼠耳肿胀法、大鼠棉球肉芽肿法以观察该方的抗炎作用;采用热板法和化学刺激扭体法观察该方的镇痛效果;通过小鼠耳廓微循环法、大鼠体外血栓法和对小鼠出、凝血时的测定观察该方的活血作用;采用青霉素腹腔注射法制备小鼠癫痫模型,并利用化学比色法检测癫痫小鼠脑组织中MDA的含量及SOD、ATPase的活力。

The epilepsy model induced by kainic acid is a dynamical model condition. 5-7 days after acute seizure episodes induced by a single systemic injection of a convulsant dose (10mg/kg) of KA the rats developed a long-lasting increase in seizure susceptibility and the characteristic histopathological features in the hippocampus similar to human TLE. So the KA model has been a well-known model to study TLE or intractable epilepsy.

颞叶癫癎红藻氨酸(kainic acid,KA)模型具有明确的动态变化过程,一次全身性给予惊厥剂量(10mg/kg)KA诱发SD大鼠急性癫癎发作后5-7天,动物开始出现癫癎发作敏感性长期增强,脑内亦发生与人类颞叶癫癎相似的神经病理、神经生化及神经分子生物学方面的改变,因此被认为是研究颞叶癫癎和难治性癫癎的理想模型之一。

Methods By the comprehensive application of the imaging examination, EEG, electrocorticogram, PET-CT and image fusion techniques, the epilepsy focus was precisely located,and the psychologic status was evaluated before and during operation, 35 patients with refractory epilepsy were treated surgically.

综合运用影像学、脑电图、PET-CT、脑功能皮层定位检查和偶极子定位与磁共振融合等方法,对35例难治性癫痫患者进行癫痫灶定位和外科手术治疗,并进行手术前后心理状况评估。

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