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endothelial cells相关的网络例句

查询词典 endothelial cells

与 endothelial cells 相关的网络例句 [注:此内容来源于网络,仅供参考]

Results (1) Cells in frontal recess: The existence of terminal cells was 45.5% in frontal sinusitis group and 23.7% in, control; anterior ethmoid cell 31.8% and 15.3%; agger nasi cells 28.8% and 13.5%.(2) Cells in frontal sinus: for perifrontal cells there were 42.4% and 22%, respectively; superaorbital cells 33.3% and 25.4%; intersinus septal cells 27.3% and 20%.

①额隐窝内出现的相关气房(无额窦炎组出现率/额窦炎组出现率):终末气房(23.7%/45.5%),前筛气房(15.3%/31.8%),鼻丘气房(13.5%/28.8%);②额窦内出现的相关气房:额气房(22%/42.4%),眶上气房(25.4%/33.3%),额窦中隔气房(20%/27.3%)。

Results were shown as followings:(1) Sodium selenite at 0~2.5 μmol/L significantly increased the antioxidative capacity of L-02 cells without having remarkable impact on SMMC-7721 cells;(2) Sodium selenite at concentrations above significantly increased telomerase activity, hTERT gene expression and telomere length of L-02 cells without significant impact on SMMC-7721 cells;(3) Sodium selenite at higher concentrations (larger than 5 μmol/L) resulted in peroxidation of L-02 cells, while scutellarin significantly counteracted its effect;(4) Selenium-rich amino acids from silkworm pupas in the range of 0.5~2.5 μmol/L Se significantly inhibited SMMC-7721 cell growth, induced apoptosis and cell cycle change, and the generation of reactive oxygen species. In contrast, sodium selenite and selenomethionine only had weak impact on them at the same concentrations;(5) A new selenium-containing protein was found from selenium-rich silkworm pupas, which is worthy to be study further;(6) An expression vector containing ansense RNA of hTERT gene were constructed and used to transfect SMMC-7721 cells. They were observed to inhibit hepatoma cells.

结果如下:(1)0~2.5μmol/L亚硒酸钠显著性增强L-02细胞的抗氧化能力;而对SMMC-7721细胞的作用不显著;(2)该浓度硒显著性提高L-02细胞端粒酶活性、增强hTERT基因表达和延长细胞端粒长度;而对SMMC-7721细胞的作用均不显著;(3)高浓度硒(5μmol/L以上)显著性抑制L-02细胞生长、致细胞过氧化,灯盏花素能拮抗硒所致过氧化、降低硒毒性;(4)0.5~2.5μmol/L富硒蚕蛹氨基酸显著性抑制肝癌细胞SMMC-7721生长、导致细胞凋亡和周期改变、诱导细胞产生活性氧,同浓度亚硒酸钠和硒代蛋氨酸对其抑制不显著;(5)富硒蚕蛹蛋白经分离纯化和鉴定后发现存在一新含硒蛋白,其结构和功能有待研究;(6)通过已有的含hTERT基因质粒,成功构建hTERT反义RNA表达质粒,转染SMMC-7721细胞后对其生长具有抑制作用。

Methods: Parallel-plate-flow-chamber was used to assay the influence of shear stress on endothelial cells morphology and cytoskeleton and apoptosis and NO secretion. endothelial cells morphology was observed under microscopy, cytoskeleton was showed by Coomassie brilliant blue staining, fluorescence stain, and transmission electron microscopy, apoptosis was detected by FCM, NO of circulating culture medium was assayed by NO kit.

细胞形态显微镜下直接观察;细胞骨架通过考马斯亮蓝染色、荧光标记和透射电镜进行观察;细胞凋亡通过流式细胞仪检测进行分析,一氧化氮通过流动循环培养基的收集后应用检测试剂盒进行检测分析。

The mechanism probably is that Pishang can inhibit the growth, movement and adheresion to extracellular matrix and endothelial cell of liver tumor cells, up-regulate the expression of nm23, down-regulate the expression of CD44, MMP-2, and inhibit the tumor neovascular by supressing the vascular endothelial cell, down-regulating the expression of VEGF, damaging the primitive mesenchymal cells and inhibiting neovascular gemmation.

砒霜具有明显的抗肝癌细胞作用,并有一定的抗侵袭与转移作用,其机制可能与其能抑制肝癌细胞增殖、迁移、阻止肝癌细胞与细胞外基质和内皮细胞粘附、上调肿瘤转移抑制基因nm23表达、下调肿瘤侵袭分子CD44、MMP-2表达以及通过抑制内皮细胞增殖、下调VEGF表达、损伤间充质细胞、阻止新生血管芽生等途径从而抑制肿瘤新生血管形成有关。

VEGF protein was induced by hypoxia in rat cerebral microvascular endothelial cells, and NYA could upregulate the expression of VEGF protein, which may be one of the protection mechanisms for cerebral microvascular endothelial cells.

缺氧可诱导脑微血管内皮细胞VEGF蛋白的表达;脑溢安可以上调VECF蛋白的表达,这可能是脑溢安对缺氧的脑微血管内皮细胞的保护作用机制之一。

Purpose: To investigate the influence of retinal photocoagulation with an indirect ophthalmoscopy contact lens on corneal endothelial cells.Methods: Seventeen eyes of 17 patients were evaluated, and corneal endothelial cells were measured twice: just before retinal photocoagulation using an indirect ophthalmoscopy contact lens and 33.2 ± 10.2 days after surgery.

为研究采用间接检眼镜接触镜进行视网膜激光光凝对角膜内皮细胞的影响,对17例患者的17眼进行检测并对角膜内皮细胞检测两次:视网膜激光光凝术前和术后33.2±10.2天检测。

HA increased level of NO by activating NOS after combining with H〓 and H〓 receptors; and on the one hand 5-HT caused brain microvessel endothelial cells constracting strongly, made the tight joint opening, increased the permeability of BBB, and on the other hand 5-HT increased the quantity of pinocytosis vesicle in endothelial cells and enhance the permeability of BBB after comining with 5-HT〓 receptor.

HA与H〓和H〓受体结合,激活NO合成酶,使NO含量增多而诱发BBB通透性增加;5-HT与5-HT〓受体结合,一方面引起脑微血管内皮细胞强烈收缩,使内皮细胞间的紧密连接开放,BBB通透性增高,另一方面可引起内皮细胞转运小泡增加,BBB通透性增高。

For example, if the microvascular endothelial cell is the principal target of radiotherapy and damage to the epithelial stem cell is a secondary event, this relationship may also hold for endothelial cells and the tumor cells they support.

举例来说,如果微血管内皮细胞是放射治疗的主要目标,而上皮干细胞的损伤是继发性事件的话,内皮细胞与其所供养的肿瘤细胞的关系也是如此。

For example, if the microvascular endothelial cell is the principal target of radiotherapy and damage to the epithelial stem cell is a secondary event, this relatio hip may also hold for endothelial cells and the tumor cells they su ort.

举例来说,如果微血管内皮细胞是放射治疗的主要目标,而上皮干细胞的损伤是继发性事件的话,内皮细胞与其所供养的肿瘤细胞的关系也是如此。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞,肾小管上皮细胞和血管内皮细胞表达均阴性。

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