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endothelial cells相关的网络例句

查询词典 endothelial cells

与 endothelial cells 相关的网络例句 [注:此内容来源于网络,仅供参考]

These indicated that endothelial cells of the mural coronary artery are free from atherosclerosis due to higher shear stress caused by myocardial bridge. The intima of the section proximal to mural coronary artery was likely to be sclerosed due to the lower and oscillatory shear force.

提示 壁冠状动脉内皮细胞受到心肌桥压迫导致血流切变力增高产生适应性反应,对内皮细胞有保护作用,而其近段内皮细胞由于切变力较低容易被损伤,成为动脉粥样硬化发生的基础。

The HUVECs retained spindled shaped or cobblestone appearance and positively stained by anti-CD31. DAPI staining showed that 90%of scaffolds surface was reendothelialized and endothelial cells were distributed evently on the surface forming a confluent monolayer.

脱细胞支架细胞成分完全脱去,胞外基质保存完好;分离的细胞培养一周后,细胞呈梭形或多角形,核仁清晰,大小均匀,呈典型的铺路石样;免疫荧光检测可见细胞胞浆内有颗粒状红色阳性信号,说明Ⅷ因子相关抗原染色阳性;膜表面有蓝色阳性信号,说明CD31抗原染色阳性。

HAase may degrade antiangiogenic HA of extracellular matrix, which may stim ulate proliferation of endothelial cells and enhance the curative effect of grow th factors to myocardial ischemia.

HAase可降解细胞外基质中具有抗血管新生作用的HA,促进内皮细胞的增殖,有可能提高促血管生长因子治疗心肌缺血的局部疗效。

Objective To perform an initial study on possible molecular regulatory mechanisms of TNF-α to the permeability of strial capillary endothelial cells in guinea pig cochlea.

目的 探讨肿瘤坏死因子α对豚鼠耳蜗血管纹微血管内皮细胞通透性的影响及可能的分子调控机理。

TNF-α can increase permeability of strial capillary endothelial cells of suinca pig cochlea.

TNF-α能使内皮细胞中的F-actin含量明显减少(P<0.01),且TNF-α的浓度越高F-acitn的含量降低越明显;L-Arg能进一步降低F-actin的含量(P<0.05);而L-NMMA则能抑制这种趋势(P<0.05)。

Methods Strial capillary endothelial cells in guinea pig cochlea was dissociated and cuhureed to establish a model for its permeability in vitro.,The animals were divided into TNF-α,TNF-α+L-arginine,TNF-α+NG-minomethyl-L-arginine and control groups.

体外分离培养豚鼠耳蜗血管纹微血管内皮细胞并建立内皮细胞通透性体外模型,观察TNF-α、TNF-α连同L-精氨酸(L-arginine,L-Arg)或L-单甲基精氨酸(NG-monomethyl-L-arginine,L-NMMA)对内皮细胞通透性的影响。

There were significant differences between all the experimental groups and the control group (F=193.25, P<0.01). Conclusions IL-1β and TNF-α induce a strong expression of MCP-1 mRNA and protein in HUVECs. Both cytokines may be involved in the atherogenesis by inducing the liberation of MCP-1 by endothelial cells and increasing the recruitment of monocytes into the subendothelial space.

IL-1β和TNFα均能显著增强内皮细胞MCP-1 mRNA和蛋白的表达,提示它们通过诱导内皮细胞产生MCP-1,吸引单核细胞迁入动脉内膜而参与动脉粥样硬化的发生和发展。

Among the earliest events in atherogenesis are the binding of monocytes to endothelium and their migration via the junctional spaces between endothelial cells till to the subendothelial spaces.

在高脂血等情况下,血液中的单核细胞不断粘附、进入动脉内膜并在内膜下聚集成为泡沫细胞是动脉粥样硬化发生发展过程中的重要环节。

In the experimental group (transplanted with embryonic arcuate nucleus), the endothelial cells remained intact, and connected tightly with subendothelial tissue, occasionally twisted nucleus could be seen.

实验证明,在被毁损弓状核区重新植入胚鼠弓状核细胞后对动脉粥样硬化有明显恢复作用,从而证实下丘脑弓状核对动脉粥样硬化的形成具有重要调控作用。

The present results suggested that CRP may be the direct activator to WISP2; thereas regulate development of inflammation in endothelial cells.

本研究同时发现p53信号显著增强,达到对照组30.50倍,而bcl-x和bcl-2相关基因则分别下调10.4倍和2.01倍。

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