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endarterium相关的网络例句

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与 endarterium 相关的网络例句 [注:此内容来源于网络,仅供参考]

The primary shortcoming of tissue engineering blood vessel before was the short-term opening of vessel cavity due to the thrombosis and over hyperplasia of endarterium.

以往的组织工程血管因易产生血栓和内膜过度增生而致开放时间短和闭塞。

The expression of ICAM1 proteinum at the endarterium of FTL and FTH group rats was obviously fewer than those of model group, and those of FTL group was fewer than those of FTH group.

免疫组化结果显示,模型组大鼠主动脉内皮细胞ICAM1表达强阳性,药物干预后FTL、FTH大鼠ICAM1表达明显减少(P<0.01),FTH低于FTL(P<0.05)但仍显著高于空白对照组(P<0.01)。

Results: in control group, there were no atherosclerotic changes on endarterium; in model group, there were obviously atherosclerotic changes on endarterium, in "National pits1573" 520、450、380 Alcohol group and 450 control Alcohol group, there were unconspicuously atherosclerotic changes on endarterium.

结果:空白对照组主动脉内膜未见粥样硬化改变;模型组主动脉内膜粥样硬化斑块形成,部分斑块病灶范围较大;45°对照酒组仅见少量散在脂质条纹;"国窖1573"52°、45°组,仅见轻度主动脉粥样硬化改变;38°组,主动脉壁见散在少量小范围脂质条纹。

Weeks later, the serum soluble intercellular adhesion molecule1 (sICAM1) level in the rats was measured by enzymeliked immunosorbent assay kit; the expression of ICAM1 proteinum in the endarterium of AS rats was detected by immunohistochemical technique; the pathological changes in the endarterium of AS rats was observed by transmission electron microscope.

用药6 w后采用酶联免疫吸附法测大鼠血清可溶性细胞间黏附分子1(sICAM1)水平,免疫组化观察主动脉ICAM1表达,透射电镜观察主动脉内膜病变情况。

METHODS: Endarterium was injuried and coronary thrombi were formed gradually through direct electrical stimulation on the coronary artery of Chinese experimental miniature swine.

直接电刺激中国实验小型猪冠状动脉造成动脉内膜损伤,逐渐形成冠脉内血栓。

Results: The tHey and blood fat level in blood plasma and expression of MCP-1mRNAof aorta on model group is obviously higher than that of control group (P<0.05);the result of immunohistochemistry shows that the expression of VCAM-1 and MMP-2enhanced, Bcl-2 is lower; aorta slice shows endarterium discontinuation, strippingpartly, smooth muscle cell arrange puffly and chaotic, infiltration of lipoid ofdifferent degree are observed in aorta wall, pathological changes suffusion, morexanthoma cell, plaque protrude lumens. The they and blood fat level in blood plasmaand expression of MCP-lmRNA, VCAM-1, MMP-2 of aorta on intervention group aremuch lower than that of model group (P<0.05); Bcl-2 of intervention group isstrengthened, and the pathomorphology and immunohistochemistry of aorta showpathological changes are slighter than model group.

结果:模型组,血浆中同型半胱氨酸浓度、血脂水平、主动脉MCP-1mRNA表达显著高于正常对照组(P<0.05),免疫组化示主动脉内皮细胞VCAM-1、MMP-2表达增强,Bcl-2表达降低;主动脉切片发现动脉内膜不连续,部分剥脱,平滑肌细胞排列疏松、紊乱,主动脉壁均见到程度不同的脂质浸润,其病变弥漫,泡沫细胞较多,可见斑块突出管腔;干预组,血浆中同型半胱氨酸含量、血脂水平显著低于对照组和模型组(P<0.05),主动脉MCP-1mRNA、VCAM-1、MMP-2表达显著弱于对照组和模型组(P<0.05),Bcl-2表达增强(P<0.05),主动脉病理形态学和免疫组织化学检测示病变轻于模型组。

Results: in control group, there were no atherosclerotic changes on endarterium; in model group, there were obviously atherosclerotic changes on endarterium, in "National pits1573" 520、450、380 Alcohol group and 450 control Alcohol group, there were unconspicuously atherosclerotic changes on endarterium.

结果:空白对照组主动脉内膜未见粥样硬化改变;模型组主动脉内膜粥样硬化斑块形成,部分斑块病灶范围较大;45°对照酒组仅见少量散在脂质条纹;&国窖1573&52°、45°组,仅见轻度主动脉粥样硬化改变;38°组,主动脉壁见散在少量小范围脂质条纹。

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