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elliptic modular group相关的网络例句

查询词典 elliptic modular group

与 elliptic modular group 相关的网络例句 [注:此内容来源于网络,仅供参考]

To the moduli with special forms (such as Mersenne numbers, pseudo-Mersenne numbers, generalized Mersenne numbers), we analysis and study modular arithmetic laws, and obtain the conclusions as follows: for Mersenne numbers and pseudo-Mersenne numbers, we get modular arithmetic formulas, and determine exact expressions of the number of modular addition to generalized Mersenne numbers generated by irreducible monic trinomials and pentanomials.

针对具有特殊形式的模数(如Mersenne数,伪Mersenne数,广义Mersenne数等),深入分析了其取模运算规律,得到如下结果:对Mersenne数和伪Mersenne数,给出了取模运算转换为模加或模减运算的公式;对模数为任意首一三项式和五项式产生的广义Mersenne数,推导了相应取模运算的复杂度计算解析表达式。

This task designs a regulated power supply of direct current with numerical control and numeric display, this power supply includes the modular circuit of direct current of stabilized voltage, protective circuit modular and voltage adjust modular etc.

中文摘要该课题设计一个数控数显直流稳压电源,该电源包括直流稳压电路模块,电压调整电路模块和保护电路模块等部分组成。

An effect of laser energy enhancement occurs in dual-modular, the output energy of dual-modular is 15% higher than twifold energy of single modular.

实验表明双模块器件存在很强的激光脉冲能量增强效应,双模块串联输出的激光能量比单模块激光能量的两倍还大15%。

The method this paper discussed was based following theoretic analysis: Square and multiplication of large integer are two basic operations in modular exponential m_ary algorithm. The main operations of m_ary algorithm of point multiplication in elliptic curve encryption were doublings and point addition. The steps of m_ary algorithm in point multiplication was similar to modular exponential.

其基于以下理论分析:模幂m_ary算法的基本运算为大数乘法,其中包括大数平方算法和一般大数乘法;椭圆曲线加密算法中点乘的m_ary算法步骤与模幂的m_ary算法相同,后者的基本运算为倍乘和加法。

(1) cerebral ischemical reperfusion injury rats'limbs motor function is variable. Acupuncture could promote lims'functional recovery.(2) PCNA masc cells is visible in cerebral ischemical semidarkness region. There is cell regeneration phenomenon. Acupuncture could strengthen injury region's PCNA expression, could profit injury recovery and functional reconstruction.(3) In ischemia semidarkness region for the model group and acupuncture group, PCNA masc cells percentage of 14days group is lower than 7days group. Along with the recovery of injury, cell multiplication is weaken.(4) In cerebral ischemia semidarkness region, there is VEGF masc cells and regeneration phenomenon. Acupuncture could strengthen injury region's VEGF expression, could profit protection after injury and blood vessel regenerate.(5) In ischemia semidarkness region for the model group and acupuncture group, VEGF masc cells percentage of 14days group is lower than 7days group. Along with the recovery of semidarkness region, ischemia and anoxemia state is getting improved, and VEGF is reduce.(6) As there are PCNA and VEGF masc cells in brain injured region, we could conclude that, after brain ischemical reperfusion injury, there are blood vessel regeneration phenomenon. Acupuncture could promote blood vessel regeneration, recovery blood supply sufficiently and quickly, and promote the recovery of brain injury region.(7)The VEGF masc cells percentage of inhibitor group is lower than acupuncture group. It state that the effect of acupuncture promote VEGF is partly depend on the existing of eNOS.

实验结论:(1)脑缺血再灌注损伤后大鼠的肢体运动功能发生改变,针刺可以促进肢体功能恢复;(2)脑缺损伤区可见PCNA阳性细胞,存在细胞再生现象,针刺可以增强损伤区PCNA的表达,有利于损伤的修复和功能重建;(3)针刺组和模型组14d时缺血损伤区PCNA阳性细胞百分比低于7d组,随着损伤逐渐得到修复,细胞增殖现象减弱;(4)脑缺血损伤区可见VEGF阳性细胞,存在内皮型细胞再生现象,针刺可以增强损伤区VEGF的表达,有利于脑损伤后保护和缺血区血管再生;(5)针刺组和模型组14d时缺血损伤区VEGF阳性细胞百分比低于7d组,随着缺血损伤的修复,缺血缺氧状态得到改善,产生的VEGF减少;(6)由于脑损伤区同时出现PCNA阳性细胞和VEGF阳性细胞,前者是增殖细胞的标志,后者是促进血管再生的重要因子,可以推断,脑缺血再灌注损伤后脑内存在血管再生现象,针刺可以促进损伤区的血管再生,更迅速而充分的恢复损伤区的血供,促进脑损伤区的修复;(7)抑制剂针刺组脑损伤区VEGF阳性细胞百分比与针刺组相比有不同程度的降低,说明针刺促进缺血损伤区VEGF表达部分依赖eNOS的存在。

Materials and methods:Male Sprague—Dawley rats were randomly divided into four groups as follow:Normoxia sedentary group,Hypoxic exposure group,Normoxia exercise group,living high training low group.the normoxia sedentary group lived in normoxic environment,hypoxic exposure group made hypoxic exposure for 12h in normobaric hypoxic tent(oxygen concentration 13.6%),Normoxia exercise group took 1h endurance exercise in normoxic environment at daytime,Hiho group was exposured in hypoxia at night after trained at daytime,the mode and duration of the both load were identical to hypoxic exposure group and normoxia exercise group.

研究方法:以雄性SD大鼠为研究对象,分成4组:常氧安静组、低氧暴露组、常氧运动组、高住低训组。常氧安静组在常氧环境下安静生活,低氧暴露组每晚在常压低氧(氧浓度13.6%)帐篷中低氧暴露12h,常氧运动组白天在常氧环境中进行耐力运动1h,高住低训组运动和低氧暴露的方式和时间分别同常氧运动组和低氧暴露组。

12H treatment group compared with 12h model group: the density of light of HSP70 of the Gardenoside group is not different and which of it of TGFβ〓 increased with the model group; the density of light of the HSP70 of the Baicalin group in not different and which of the TGF-β〓 is no different with the model group, the density of HSP70 of the Concha Margatitifera Usta is not different and which of TGFβ〓 increased than the model group; the density of the HSP70 of the Cholic Acid group is no different and the density of TGF-β〓 increased than the model; the density of the light of HSP70 of the Hefang Group increased and which of TGF-β〓 increased than the model group.

合方治疗脑缺血12小时组缺血脑组织TGF-β1光密度值较缺血12小时模型组明显升高(p<0.01)。大鼠局灶性脑缺血24小时模型组缺血脑组织TGF-β1光密度值含量较正常组明显增高(p<0.01)。珍珠母治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高(p<0.01)。胆酸治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高(p<0.01)。栀子苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组无显著性差异(p>0.05)。黄芩苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高(p<0.01)。

The time of cornea diabrosis, metastases into cervical lymph nodes and lung, survival time and ratio of CD4+ and CD8+ T cells in peripheral blood were observed.RESULTS: The time of cornea diabrosis in GTVC group or GTV group was later than that in PBS group, but no statistical difference between GTVC group and GTV group. Metastatic incidence in cervical lymph nodes of GTVC group mice or GTV group mice was lower than that in PBS group 18 days after the model had been established. There was no statistical difference in metastatic incidence between GTV group and GTVC group.

将荷瘤模型鼠分为磷酸盐缓冲液(phosphatebuffered saline,PBS)治疗组,IL12基因修饰肿瘤细胞疫苗(gene modified tumor cell vaccine, GTV)治疗组及GTV 联合CpGODN治疗组;鼠荷瘤模型建立后的第3d及第10d分别在各组鼠结膜下注射30μL PBS,30μL(3.0×106个细胞)灭活的GTV及含20μg CpGODN灭活的GTV,进行角膜溃破时间、淋巴结和肺部转移瘤发生率、生存时间及外周血CD4+和CD8+T细胞比例的观测。

Objective To study the relationship of endangium proliferation and the expression of metalloproteinases and tissue inhibitors of metalloproteinases after angioplasty of iliac artery in rabbits to investigate the probable mechanism of composite Danshen pill in prevention and treatment of restenosisMethods 30 white male rabbits of Japan (the average body weight was 25~30 kg)were randomly divided into 3 groups:normal control group,model group (intima destroyed by balloon and given hypercholesterol diet),CDP group (intima destroyed by balloon and given hypercholesterol diet plus drug CDP 150 mg/d),10 ones each groupResults The results of iliac artery angiogram and the analysis of pathology:there were lumens stenosis,thinner intima,smaller intima area,in CDP group compared with those in model group,and ratio of intima and tunica media thickness and area among each group (P<001)Immunohistochemistry analysis:the MMP2,MMP9,TIMP1,TIMP2 in model group significantly increased than those in control group (P<001)The CDP group had lower MMP2 and MMP9 expression,higher TIMP1 and TIMP2 expression,and increased MMP2/TIMP1,MMP9/TIMP2 than model group (P<001)The thickening of vascular neointima and stenosis degree of vascular lumen were relevant to MMP2 and MMP9 (r=0896,P<001)Conclusions MMP and TIMP play the very important role in the restenosis process of arteryCDP could inhibit the thickness of vascular neointima after balloon injury,the probable mechanism of which may be inhibiting collagen formation,smooth muscle immigration and decreasing hyperplasia of intima by interfering expression of MMPs and TIMPs

目的 研究兔髂动脉成形术后血管内膜增生和基质金属蛋白酶及其抑制物表达之间的关系,探讨复方丹参滴丸预防再狭窄的可能机制。方法健康成年二级雄性日本大耳白兔30只,平均体重25~30 kg。随机分为3组:正常对照组10只,模型组10只(球囊内膜剥脱加高胆固醇饮食),治疗组10只(球囊内膜剥脱加高胆固醇饮食以及复方丹参滴丸150 mg/d)。结果兔髂动脉造影、血管病理图像分析检测结果:①复方丹参滴丸组较模型组血管造影示管腔直径狭窄、内膜厚度减少、内膜面积减少、内膜厚度和中膜厚度比、面积比,各组之间有显著性差异(P<001)。②免疫组化分析:模型组MMP2、MMP9、TIMP1、TIMP2表达均高于对照组(P<001);复方丹参滴丸组MMP2、MMP9的表达低于模型组,而TIMP1、TIMP2的表达高于模型组;TIMP1/MMP2、TIMP2/MMP9明显增加,差异有显著性(P<001)。③内膜的增生以及管腔的狭窄程度与MMP2、MMP9有很好的相关性(r=0896,P<001)。结论 MMP和TIMP在再狭窄形成中起重要作用;复方丹参滴丸能够明显抑制血管损伤后的内膜增生,可能的机制是通过影响金属蛋白酶MMP2、MMP9及其抑制物TIMP1、TIMP2的表达从而抑制胶原的生成、平滑肌的迁移、增生,而减少内膜的增生。

Results On the basis of the percentage of GPI-anchored PLAP conversion,the sera GPI-PLD activities of total 172 patients,included 26 severe acute viral hepatitis as group A,29 liver cirrhosis as group B,32 chronic viral hepatitis as group C,55 mild acute viral hepatitis as group D,30 primary hepatocellular carcinoma as group E,were measured.As compared with 182 healthy presons as control group,the sera GPI-PLD activities of group A and B were significantly reduced;By contraries,the activities of group D and E were significantly raised.The sera GPI-PLD activities of group C compared with healthy control group were not significantly altered.However,when paired Q-test,the changes of serum GPI-PLD activity between all paired groups among this five groups were remarkable.

结果检测172例肝脏疾病患者(包括A组:26例急性重症病毒性肝炎;B组:29例肝硬化;C组:32例慢性病毒性肝炎;D组:55例急性非重症病毒性肝炎;E组:30例原发性肝癌)血清中GPI-PLD的活性,发现A、B两组患者的该酶活性较正常人(182名)显著性降低,而D、E两组则较正常人显著性增高,C组患者的酶活性与正常人无显著性差异,但A、B、C、D、E两组之间的酶活性水平改变却均有显著性。

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