查询词典 elastase

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活，ICAM-1表达升高，与被激活的PMN表面表达的LFA-1相结合，"PMN-EC"相互作用加剧，一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍；另一方面粘附的PMN过度吞噬或激活，当白细胞吞噬的颗粒不能被封闭隔离，连同细胞内的酶被释放出来，其中的PMN-elastase能够降解细胞基质中各种成分，水解多种蛋白，加重胰腺的毛细血管内皮细胞和腺泡的损伤；释放的溶酶体酶使组织蛋白水解，产生的不饱和脂肪酸引发脂质过氧化方应，破坏细胞膜的结构和功能；释放的炎性细胞因子，激发其他的免疫细胞的功能，导致进一步的组织损伤和坏死，加重SAP的病理损伤，最终导致休克、脓毒血症及多器官功能障碍等严重后果。

These include lactoferrin ; elastase ; and cathepsin G .

这些措施包括乳铁蛋白；弹性蛋白酶和组织蛋白酶G 。

The pancreatic proterses trypsin,chymotrypsin and elastase are all derived from zymogen precursors by proteolytic activation.

胰腺的蛋白水解酶—胰蛋白酶、胰凝乳蛋白酶和弹性蛋白酶都从酶原前体衍生而来，即由蛋白水解而活化的。

Results Eleven proteins, including chymotrypsinogen B1 precursor, carboxypeptidase A1, trypsinogen 2, chymotryptic peptide C, trypsin 1, carboxypeptidase B1, kinesin superfamily proteins 3B, trypsin 2, mitochondria protein gene, elastase 3A and colipase were found to be able to bind to HCV core protein. Conclusions Proteins related with metabolism of glucose and lipid may bind to HCV core protein.

因此我们应用酵母双杂交技术筛选人胰腺细胞 cDNA 文库中的 HCV 核心蛋白结合蛋白基因，为研究 HCV 慢性感染与胰岛素抵抗（ insulin resistance ， IR ）、代谢性疾病之间的相关性及其作用机制，以及进一步研究 HCV 影响糖、脂类代谢的分子生物学机制提供一定的思路和方向。

Firestone showed that I3C prevents the elastase shortening of cyclin E, thereby arresting development of breast cancer cells.

火石表明， I3C防止弹性缩短周期蛋白E ，从而逮捕了发展的乳腺癌细胞。

It is a new discovery to prove the expression of hepatocyte elastase mRNA from morphology. It can be infered that it is related to metabolism of hepatic ground substance. It may be of value to study the development and degradation of hepatic fiber.

从形态学证实肝脏细胞有弹力蛋白酶mRNA的表达，丝氨酸弹力蛋白酶有可能参与肝纤维化细胞外基质代谢

Special enzymes may be used for certain tissues e.g. elastase for lung tissue, hyaluronidase for connective tissues.

特别的酵素可能为肺薄的纱织品举例来说作为特定的薄纱织品 elastase，对于连接着薄的纱织品的玻尿酸酵素。

MPS inhibits various aggressive lysosomal enzymes that degrade important constituents of connective tissue, such as elastase, collagenase and hyaluronidase.

MPS抑制了各种参与分解代谢的酶，这些酶用于降解结缔组织的重要组成元素，如弹性蛋白酶，胶原酶和透明质酸酶。

Objective The aim of this study was to evaluate the reaction of the rat－ lung to elastase by a single intratracheal instillation of elastase.

目的在 SD大鼠气管内滴入猪胰弹性蛋白酶，复制肺气肿模型，观察鼠肺对弹性蛋白酶的反应。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活，ICAM-1表达升高，与被激活的PMN表面表达的LFA-1相结合，&PMN-EC&相互作用加剧，一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍；另一方面粘附的PMN过度吞噬或激活，当白细胞吞噬的颗粒不能被封闭隔离，连同细胞内的酶被释放出来，其中的PMN-elastase能够降解细胞基质中各种成分，水解多种蛋白，加重胰腺的毛细血管内皮细胞和腺泡的损伤；释放的溶酶体酶使组织蛋白水解，产生的不饱和脂肪酸引发脂质过氧化方应，破坏细胞膜的结构和功能；释放的炎性细胞因子，激发其他的免疫细胞的功能，导致进一步的组织损伤和坏死，加重SAP的病理损伤，最终导致休克、脓毒血症及多器官功能障碍等严重后果。

We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称；三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起，称之为挽歌体诗人或者史诗诗人，他们被称为诗人，似乎只是因为遵守格律写作，而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。