查询词典 edema

The CC MRI findings are characterized mainly by:(1)frequently-encountered multiple circular foci during the cysticercus survival:small excentric spotty images of the mural cysticercus scolex :slightly high signs on T1WI and middle-lower signs on T2WI;lower signs on T1WI and high signs on T2WI of the capsular liquid;lower signs of the cerebrospinal fluid but relatively high signs of the capsular liquid on FLAIR;and unremarkable peripheral edema;(2)scolex disappearance,enlarged capsular cavity and remarkable peripheral edema during cysticercus degeneration and necrosis;(3)lower signs on T1WI and T2WI and peripheral edema disappearance after the focal calcification;(4)findings shown by the enhanced scanning:strengthened (24/36) or non-strengthened (12/36) cyst-wall circular images;strengthened (22/36) or non-strengthened (14/36) small intracapsular spotty images;(5)cerebral-ventricular type often occurring in the third and fourth ventricles:one case in the third ventricle (1/36) and three cases in the fourth ventricle (3/36),all of suffered from obstructive hydrocephalus;(6)hydrocephalus caused by arachnoid adhesion (2/36) in meningeal type.

结果 脑实质型18例，脑室型4例，脑膜型3例，混合型11例，主要MRI特点：(1)囊虫存活期，病灶呈圆型，多发常见，附壁囊虫头节呈偏心型小点状影，T1WI呈略高信号，T2WI呈中低信号；囊液T1WI呈低信号，T2WI呈高信号；在FLAIR上脑脊液呈低信号，而囊液呈相对高信号；周围水肿不明显。(2)囊虫退变坏死期，头节消失，囊腔扩大，周围水肿明显。(3)病灶钙化后，T1WI及T2WI均呈低信号，周围水肿消失。(4)增强扫描表现为：囊壁环状增强(24/36)，或不增强(12/36)；囊内小点状影增强(22/36)，或不增强(14/36)。(5)脑室型：常发生于第三、四脑室，其中第三脑室1例(1/36)，第四脑室3例(3/36)，均引起阻塞性脑积水。(6)脑膜型：因蛛网膜粘连，而引起脑积水(2/36)。

In treatment group, there was no progressive necrosis in stasis zone, and at 24 h post injury, capillary dilation, cell edema and inflammatory infiltration were lessened significantly. In control group, 2 wounds had progressive necrosis (accounted for 10% of the total). Cell edema, inflammatory infiltration and capillary thrombosis were serious, Until 72 hr post injury, cell edema did not subside and necrosis of the dermis worsened. 2. Pathological assessment of the stasis zone tissue: As compared with that in control group, in treatment group the dermis structure in stasis zone was intact and the collagenous fiber bundle was normal.

结果：①创面细胞形态学改变：伤后8h毛细血管扩张、细胞水肿，炎性浸润最明显；治疗组中央淤滞区无进行性坏死，伤后24h后毛细血管扩张、细胞水肿、炎性浸润减轻；对照组2个创面呈进行性坏死（占10%），中央淤滞区细胞水肿、炎性浸润明显，毛细血管血栓形成，伤后72h水肿仍明显，真皮坏死加重；②淤滞区组织病理学评分：治疗组与对照组相比，治疗侧淤滞区的表皮结构相对完整、胶原纤维束相对正常，粒细胞浸润小于5个/400倍视野。

A single systemic injection of resiniferatoxin , an ultrapotent analog of capsaicin, produced a long-lasting, reversible blockade of adjuvant-induced thermal hyperalgesia and reduction of early edema in the adult rat. Depletion of mast cell granules in peripheral connective tissues with secretagogue compound 48/80 also reduced the hyperalgesia and early edema to some extent. Co-pretreatment with RTX and compound48/80 had the similar effects as that of RTX treated alone. After unilateral surgical sympathectomy, the thermal hyperalgasia and edema were both significantly enhanced.

成年鼠一次性注射辣椒素类似物resiniferatoxin后，长时程、可逆性阻断了CFA所致热痛过敏的发展并削弱了早期炎性水肿；用促脱颗粒剂compound48／80慢性耗竭外周结缔组织肥大细胞内分泌颗粒后，也部分削弱了热痛过敏及早期水肿的形成；RTX与compound48／80共同处理对热痛过敏及水肿有与RTX单独处理相近程度及时程的影响；单侧交感链切除显著易化了炎性热痛过敏及水肿。

ObjectiveNon-heparinized autohemic arterial blood of the rat was injected into its caudate nucleus to establish a model of intracerebral hemorrhage. The changes of edema in various regions of the brain were measured at different time after hemorrhage. Meanwhile, the effects of erythrocytes and thrombin on the formation of cerebral edema after cerebral hemorrhage were studied to explore the pathophysiological mechanism of cerebral edema and the roles of erythrocytes and thrombin in this process.

目的 利用自体未肝素化新鲜血回注法建立大鼠尾状核出血模型，观察脑出血后不同时间不同部位脑组织水分含量的变化，并分别研究红细胞、凝血酶对脑出血后脑水肿形成的影响，揭示脑出血后脑水肿形成的病理生理机制，并阐明红细胞、凝血酶在脑出血后脑水肿形成中的作用，从而为临床脑出血病人病情评估及治疗提供理论依据。

MethodsNonheparinized autohemic blood of the rat was taken out from rat"s femoral artery and injected into its caudate nucleus immediately to establish a model of intracerebral hemorrhage.The changes of edema in the ipsilateral bassal ganglia , ipsilateral cortex, contralateral bassal ganglia and contralateral cortex were measured at 1,2,3,5 and 7 days respectively after intracerebral hemorrhage. The rat"s packed red blood cells, lysed red blood cells and hemoglobins at different concentrations were injected respectively into the rat"s caudate nucleus. The changes of edema in various regions of the brain at different time were measured as above. 5U thrombin was injected into the rat"s caudate nucleus and the changes of edema in various regions of the brain at different time were measured as above.

自体股动脉取血，并采用自体未肝素化新鲜血回注法建立大鼠尾状核出血模型，观察脑出血后1，2，3，5，7天血肿同侧基底节区、血肿同侧对应的皮质区、血肿对侧对应的基底节区及血肿对侧对应的皮质区脑组织水分含量的变化；分别以自体浓缩红细胞(PRBC，50％)、溶解红细胞(LRBC，50％)、不同浓度的大鼠血红蛋白注入大鼠尾状核，并观察不同时间不同部位脑组织水分含量的变化；以5U凝血酶注入大鼠尾状核，观察不同时间不同部位的脑组织水分含量的变化。

Electron microscopic examination showed that the spinal cords were normal in group N and A, slight edema of mitochondria and endoplasmic reticulum in group B, loosened fibrous layers in medullary sheath, edema and local degeneration of neuraxis in group C,and shrinkage of nuclear membrane, serious edema of ER, vacuolus change of mitochondria and local demyelination in group D.

电镜结果显示，N组和A组脊髓超微结构正常；B组线粒体、内质网轻度水肿；C组脊髓髓鞘纤维板层结构疏松，轴索肿胀，局灶变性；D组脊髓神经元细胞核膜皱缩，线粒体空泡样变，内质网肿胀严重，局灶性脱髓鞘改变。

Effect of A_3 on dimethylbenzene-induced mice ear edema Method:After peroral A_3 3 days, the mice ear edema models induced by dimethylbenzene were prepared. The mice ear edema was tested at 1 h.

A_3对二甲苯所致小鼠耳廓肿胀的影响方法：连续灌胃给药3天(1次/天)后，采用二甲苯制备小鼠耳廓肿胀的炎症模型，观察二甲苯致炎后1 h药物对小鼠耳肿胀的影响。

Effect of A_3 on carrageenin-induced rats paw edema Method:After peroral A_3 4 days, the rats paw edema models induced by CGN were prepared. The rat paw edema was tested at

A_3对角叉菜胶所致大鼠足肿胀的影响方法：连续灌胃给药4天(1次/天)后，采用角叉菜胶制备大鼠足趾肿胀的炎症模型，观察角叉菜胶致炎后1，2，4，6 h药物对大鼠足趾肿胀的影响。

Piglet edema were first discovered in Britain, is hemolytic E. coli intestinal toxemia, so capillaries or systemic damage to the small blood vessels, increasing permeability and fluid extravasation too much water, to the head, eyelids, Equality ear edema, acute ataxia and death as the main feature of acute infectious diseases, also known as gastrointestinal edema pigs or pig intestinal toxemia E. coli, a highly lethal disease.

仔猪水肿病最早发现于英国，是由溶血性大肠杆菌引起肠毒血症，而使全身毛细血管或小血管受到破坏，通透性增大，水液外渗过多，造成的以头部、眼睑、耳部等处水肿、共济失调和急性死亡为主要特征的急性传染病，又称猪胃肠水肿或猪大肠杆菌肠毒血症，有高度致死性疾病。

Deferoxamine can attenuated the brain edema. Conclusion RBCs play an important role in development of brain edema after ICH and the use of iron chelators can abate brain edema after ICH.

红细胞在脑出血后脑水肿的形成中起重要作用；铁络合剂可减轻脑出血后脑水肿。

With Death guitarist Schuldiner adopting vocal duties, the band made a major impact on the scene.

随着死亡的吉他手Schuldiner接受主唱的职务，乐队在现实中树立了重要的影响。

But he could still end up breakfasting on Swiss-government issue muesli because all six are accused of nicking around 45 million pounds they should have paid to FIFA.

不过他最后仍有可能沦为瑞士政府&议事餐桌&上的一道早餐，因为这所有六个人都被指控把本应支付给国际足联的大约4500万英镑骗了个精光。