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dose rate相关的网络例句

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与 dose rate 相关的网络例句 [注:此内容来源于网络,仅供参考]

The uptake of CNTs by Stylonychia mytilus and their distribution during the dividing process of the cells were observed with optical microscope for the first time. The dose-effect relationship between concentration of CNTs and surviving rate of Stylonychia mytilus was measured.

自从1991年11月日本电气公司基础研究实验室的饭岛钝雄发现碳纳米管以来,其作为一种新型的纳米材料,已成为富勒烯领域的一个主要的研究热点,被广泛应用于物理、化学、材料等各个领域。

It is different that different dose of NO influenced on incidence rate of neuronal apoptosis.

不同剂量的NO对神经细胞凋亡的影响不同。

zinc could inhibit the luminescence rate of photoluminescent bacteria and antagonize MeHg toxicity in certain dose and time.

Zn可以抑制MeHg对发光杆菌发光率的影响,拮抗甲基汞的毒性。

Micronuclei in the bone marrow polychromatophilic erythrocytes in each dose group of mice were examined,leucocytes number and 30 day survival rate of mice following whole-body 5.0?Gy γ irradiation were also determined.

通过照射前后给予受试物,检测各剂量组受照小鼠的白细胞数、骨髓嗜多染红细胞微核数,观察照射后30d的平均存活天数。

Furthermore, with only a half -dose of DNA in the current system, by the horizontal system under×100 magnification both the survival rate of pronuclear embryos following microinjection and the integration frequency of exogenous DNA injected into embryos also increased.

此外,利用该系统还可在低倍镜下×100)将DNA常规注射量减半注射,提高了原核胚注射后的成活率,其注射胚的外源基因整合率亦明显提高。

Results The cytopathic effect induced by influenza virus was inhibited remarkedly,with IC50 of 3.33 mg raw material/ml and TI of 7.48. At the dose of 8~20g raw material/kg body weight,pneumonia induced by influenza virus in mice was inhibited significantly by the water extract.The survival rate was increased remarkedly. Conclusion The water extract from Pitecollobium clypearia Benth.

结果 猴耳环水提取物在体外对流感病毒引起的细胞病变有明显抑制作用,其IC50生药为3.33mg/ml,TI为7.48;在给生药药剂量为8~20g/kg时,对流感病毒所致的小鼠肺部炎症有明显抑制作用,并能明显降低流感病毒感染小鼠的死亡率和延长其存活时间。

The result indicated that the rats received LE95 at a dose of 0.4g/kg body weight may act against hemolytic anemia by enhancement of the splenocyte proliferation rate.

然而,CPLE2组出现相对脾重增加的特殊现象,可能是藉由增加脾脏细胞增生的速率抵抗该组溶血性贫血的结果。

Results The high incidence rate of hematoma enlargement was high in the shape of hematoma shape irregularity cases, thalami hemorrhage cases, vascular hypertension cases, using large dose mannitol in super-early period cases.

结果 血肿形态不规则、丘脑部位出血、血压过高超早期应用大剂量甘露醇等脱水剂病例血肿扩大发生率高。

Reactive oxygen species causing DNA oxidative damage comes from two kinds of ways:one is from cellular normal physiological metabolism;the other is from outer environment.Redox-sensitive green fluorescent protein was expressed in Saccharomyces cerevisiae.Recombinant cells were evaluated in monitoring the changes in the redox state of living cells when challenged with toxicologically relevant metal ions NaAsO_2 or Pb(NO_3)_2 by measuring emission intensity at 510 nm with a Hitachi F6500 fluorescence spectrophotometer,roGFP expressed in yeast responded not only to typical membrane-permeant oxidants H_2O_2 and reductants DTT,but also to toxicological metal ion-induced intracellular redox changes in a dose-dependent manner.Moreover,exposure of yeast cells to NaAsO_2 or Pb(NO_3)_2 at concentrations that induced redox changes reported by roGFP caused up to 2~3 fold increases in DNA mutation frequency.This mutagenic effect was largely caused by oxidative stress since blocking the production of hydryl radicals with thiourea significantly reduced the mutation rate as well as delayed the cell death.

本文将对氧化还原状态变化敏感的绿色荧光蛋白roGFP1-R12,在酵母细胞中实现了多拷贝强表达;荧光扫描经强氧化剂H_2O_2和还原剂DTT以及环境中重金属NaAsO_2或Pb(NO_3)_2处理后的酵母细胞悬液,测定510 nm处的荧光发射强度结果显示,表达的绿色荧光蛋白对氧化还原水平敏感,且在510 nm处的荧光强度与一定的重金属浓度呈正相关,即roGFP1-R12在510nm处的荧光发射值随重金属浓度的增高而增强,从而说明重金属对细胞的毒性在一定程度上很可能是通过破坏细胞内的氧化还原平衡发生作用;同时通过该绿色荧光蛋白对胞内氧化还原状态变化的响应情况可以来实时检测环境中的重金属;遗传学的点突变频率及致死率实验数据表明,重金属能导致菌体的点突变频率和致死率升高,且活性氧的清除剂巯基脲能明显降低这种点突变和致死率,说明由重金属引发的这种点突变和致死效应在很大程度上是依赖于重金属对细胞诱导产生的氧化胁迫。

Observe the formula's effects to the diabetic nephropathy rat model cause by small dose STZ injured, high lipin feed and hemiresection, the results showed that can decrease significantly blood sugar after diet 2h, control availably hyperplasia of glomerulus mesenteric cell, inhibite tumefaction of glomerulus capillary endothelial cell, keep integrality of podocytic process and basement membrane, relieve vasal cavum stricture, reduce the GFR and excretory rate of 〓-MG, improve the status of high blood lipin, specially decrease significantly TC, reduce the 〓 and T/K, improve the status of microcirculation obstacle, elevate the activity of 〓-〓-ATP enzyme.

观察降糖益肾方对小剂量STZ诱导并单侧肾切除,高脂饲料喂养的糖尿病肾病大鼠模型的影响实验结果:对2h后血糖有显著降低作用(p.05),能有效抑制肾小球系膜基质的增生,减轻肾小球毛细血管内皮细胞肿胀,保持足突与基底膜的完整性,减轻血管腔狭窄,降低GFR、尿微量白蛋白以及〓-微球蛋白的排泄率(p.05),能改善高血脂状态,降低血TG、TC含量(p.05),降低〓与T/K比值(p.05),改善微血管循环障碍的状态;能升高肾组织中〓-〓-ATP酶的活性(p.05)。

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