查询词典 d-quercitol

Dipelta, an endemic genus to China, belongs to the family Caprifoliaceae, comprised with four species: D. floribunda, D. elegans, D. yunnanensis, D. wenxianensis.

双盾木属隶属于忍冬科，是我国古特有属，属下有4个种：双盾木(D.floribunda)、优美双盾木，云南双盾木(D.yunnanensis)和文县双盾木(D.wenxianensis)。

objective:our aim is to investigate the relationship of vegf c,vegf d and lymph node metastasis in pancreatic cancer,and to elucidate the role and significance of the cancerous peripheral lymphatic tissue.methods: the expression of vegf c and vegf d was assayed by means of immunohistochemistry in 30 pancreatic carcinomas.results:the positive rates of vegf c、vegf d were 73%(22/30)、57%(17/30) respectively in pancreatic cancer.the expression of vegf c、 vegf d in cancerous invasive edge was significantly higher than that in the center of cancerous tissues.there was no correlation between the expression of vegf c、vegf dand the site,differentiation,histology types.

目的：探讨胰腺癌中vegf c、vegf d蛋白表达与淋巴结转移之间的相关关系，阐明癌周淋巴管增生在胰腺癌淋巴转移中的作用及意义。方法：免疫组化检测30例胰腺癌组织中vegf c、vegf d的表达及其与临床病理、淋巴结转移的关系。结果：胰腺癌vegf c、vegf d蛋白表达阳性率分别为73%(22/30)、57%（17/30），肿瘤周边部位显著高于肿瘤中心部位，其表达与肿瘤的部位、分化程度、组织学类型无关，与肿瘤的tnm分期有关，ⅲ~ⅳ期显著高于ⅰ~ⅱ期。

First,the determinant is regarded as a function of or- der n and denoted by D;Second,the determinant is expanded by row or by column,then the relation in both of Dand subdeterminants will be examined in details to set up certain a recursion,generally speaking,it must be a homogenous or a non homogenous recursion;fi- nally the coefficients of the general solution are found out with the aid.

给出了用递归关系方法求任意 n 阶行列式的值的一般方法：首先，把已知的 n 阶行列式看作为阶数 n 的一个函数，记为 D；其次，按行或按列展开这个行列式，并仔细观察存在于余子式及 D里的关系，建立关于 D的某一递归关系，此关系总为一个齐次的或非齐次的递归关系；最后，借助于 D(0)、D(1)和D(2)等求出递归关系的通解的系数。

There are several D-amino acid itself is toxic to plants even in the relative low concentration of D-amino acids because many plants lack the normal channels of D-amino acid oxidation deamidation, Therefore, we can use D-amino acid lyase having the toxicity to plant as a safe selectable marker.

对植物来说，很多组织缺乏正常的途径对D-氨基酸进行氧化脱氨基作用，由于这种消化系统的不同导致的结果是，有几种D-氨基酸本身对植物是有毒性的，即使在D-氨基酸相对浓度较低的情况下。

Results: Tyroserleutide can significantly increase the life span of H22 tumor-bearing mice by 50-70% in dosages of 20ug/kg/d-80ug/kg/d,specially the high dosage of 80ug/ml can significantly increase the life span by 69.24%; Tyroserleutide can inhibit the growth of transplanted hepatocellular tumor BEL-7402 in nude mice,the rate of tumor inhibition was25-50% in dosages of 40-320ug/ml ,the inhibition rate of 160ng/ml was 44.03%; Tyroserleutide could inhibit the growth of H22 and BEL-7402 tumor in a dose-dependent manner. Simultaneously, tumoricidal activity of tyroserleutide against BEL-7402 cell line in vitro was observed hinger when compared with the control group(P.05).The inhibition effect of 72hrs was higher than 24hrs,48hrs,96hrs.And specially the high dosage of 160ug/ml can significantly inhibit growth of tumor cell by 19.36%. Tyroserleutide can activated PEM and marked enhance cytotoxicity andphagocytosis functions in vitro and in vivo. The OD values of cytotoxicity were observed hinger when compared with the control group(P.05).The cytotoxicity of macrophages activated by tyroserleutide against BEL-7402 and B16-F10 was 35.58%,61.2% in vitro and21.39%,47.63% in vivo. The cytotoxicity rate of nude mice PEM was 32.86%,73.07% in vivo. Furthermore, tyroserleutide alone could stimulated the production of IL-1B TNF- a and NO by M . Tyroserleutide and LPS could synergistically activated M producing more cytotoxicity effectors. Conclusion: Tyroserleutide had inhibition functions against hepatoma carcinoma .Its possible mechanisms were related to the affect that Tyroserleutide could inhibit tumor cell directively and induce tumor cells apoptosis or death effectively.

结果：酪丝亮肽能显著延长腹水型肝癌H_(22)小鼠的生存时间，给药剂量为80μg／kg／d时疗效最显著，达到69.24％，在20μg／kg／d-80μg／kg／d剂量范围内生命延长率为50-70％，给药剂量与荷瘤鼠生存时间呈现一定量效关系；酪丝亮肽能显著抑制人肝癌BEL-7402移植瘤裸鼠的肿瘤生长，给药剂量为160μg／kg／d时疗效最显著，抑制率为44.03％，并且在40-320μg／kg／d剂量范围内抑制率为25-50％，给药剂量与肿瘤抑制率呈现一定量效关系；酪丝亮肽体外对人肝癌BEL-7402细胞生长有一定的抑制作用，在作用72hrs时各浓度酪丝亮肽对肿瘤细胞的抑制作用较24hrs、48hrs、96hrs明显，其中浓度为100μg／ml时抑制率达19.36％；酪丝亮肽体内外均能增强小鼠腹腔巨噬细胞对肿瘤细胞的杀伤：体外作用中巨噬细胞对BEL-7402、B16-F10的杀伤功能明显增强，与效应细胞对照组相比有显著性差异(P＜0.05)杀伤率分别达到35.58％、61.2％；体内作用中巨噬细胞对BEL-7402、B16-F10的杀伤功能明显增强，与生理盐水对照组相比有显著性差异(P 。05)，杀伤率分别达到21.39%、47.63%；裸鼠腹腔巨噬细胞经酪丝亮肤作用后对BEL一7402、B 16一F10杀伤功能明显增强，与生理盐水对照组相比有显著性差异(P.05)，最高杀伤率分别达到32.86%、73.07%；酪丝亮肤能增强单核巨噬细胞系统的吞噬功能，吞噬指数与生理盐水组比较有显著性差异(P.05)；酪丝亮肤体外作用能促进小鼠腹腔巨噬细胞分泌合成细胞毒效应分子IL一lp、TNF一Q和NO，与效应细胞对照组相比有显著性差异(P.05)；酪丝亮肤体内作用能促进小鼠腹腔巨噬细胞分泌合成细胞毒效应分子IL一lp、TNF一Q和NO，与生理盐水对照组相比有显著性差异(P.05)；酪丝亮肤能促进鼠巨噬细胞株R戌W264.7分泌合成IL一1p和NO，IL一1日、NO水平分别在酪丝亮肤作用24hrs、12hrs时达到高峰，酪丝亮肤单独应用能提高巨噬细胞的分泌合成功能，而且酪丝亮肤能与LPS协同作用刺激巨噬细胞的细胞毒效应分子分泌合成。

Based on the energy budget equation, the optimum feeding rate for gibel carp was C〓=G〓+F〓+U〓+R〓(G〓, maximum growth energy, KJ/d; F〓, fecal energy at the optimum feeding rate, KJ/d; U〓, excretion energy at the optimum feeding rate, KJ/d; R〓, metabolism energy at the optimum feeding rate), and F〓=0.077C〓, U〓=0.052C〓 and R〓=k〓 and k is adjusted coefficient.

根据能量收支方程，异育银鲫的最适摄食率可表述为：C〓=G〓+F〓+U〓+R〓（G〓：最大摄食率条件下的生长能，KJ/d；F〓：最适摄食率条件下的排粪能，KJ/d；U〓：最适摄食率条件下的排泄能，KJ/d；R〓：最适摄食率条件下的代谢能，KJ/d），其中F〓=0.077C〓，U〓=0.052C〓，R〓=k*，k为校正系数。

Result 1-3-β-D glucosan was found exceeding the diagnostic criteria (10pg/ml) of the kit in 18 cases of fungal rhinosinusitis; and it was found no more than 10pg/ml in all 5 cases of purely nasal septum deviation patients. One of the 18 cases of fungal rhinosinusitis patients is chronic invasive fungal rhinosinusitis, and its level of 1-3-β-D glucosan reaches to 21.82 pg/ml. We compared the result of the study group to the control groups, and found that there is significant deviation beteen the two groups.

结果 经过实验检测20例真菌性鼻窦炎(19例为真菌球型，1例为慢性侵袭性真菌性鼻窦炎)及5例对照组患者血浆中1-3-β-D葡聚糖的含量，可以发现其中18例真菌性鼻窦炎患者血浆中1-3-β-D葡聚糖的含量大于试剂盒的诊断标准10pg/ml，其中一例慢性侵袭性真菌性鼻窦炎患者血浆中1-3-β-D葡聚糖的含量达到21.82pg/ml，2例真菌性鼻窦炎患者血浆中1-3-β-D葡聚糖的含量小于10pg/ml，而对照组患者血浆中1-3-β-D葡聚糖的含量均小于10pg/ml，病例组与对照组间采用卡方检验测得卡方值为16.071，(P＜0.05)两组间有差异，有统计学意义。

Methods Eighty Kunming mice were equally randomized into 8 groups: normal group, model group, vitamin E (90mg.kg-1.d-1) group, highand lowdose RPM protein (20 and5 mg.kg-1.d-1 respectively) groups, high and lowdose RPM anthraquinone glycoside (28 and 7mg.kg-1.d-1 respectively) groups, combination (lowdose RPM protein and lowdose RPM anthraquinone glycosides)group. Except the normal group,the mice in other groups received subcutaneous injection of Dgalactose to indue the aging model.

选用昆明种小鼠80只，随机分为8组：正常组，模型组，维生素E组（剂量为90mg·kg-1·d-1），何首乌蛋白质高、低剂量组（剂量分别为20、5mg·kg-1·d-1），何首乌蒽醌苷高、低剂量组（剂量分别为28、7mg·kg-1·d-1），AGRPM与PRPM混合组（混合组，均取两者低剂量）；除正常组外，其他组均采用长期（7周）皮下注射D半乳糖复制衰老小鼠模型。

ResultsIn group C and D hyperplasia of epithelium cell and infiltrations of inflammative cell in day 14 that were severe than that of day 7,ciliate cell was piece loss,the glands were hyperplasia（≥40% of group C,≤20% of group D）in windpipe,and pulmonary alveolus were congestion and dropsy.In group C the epidermis were pucked and connective tissue was hyperplasia in the skin,the quantity of hair ball and the sweat glands of group C and D in the food were significantly reduced than that of group A.

结果第14天时，C组、D组气管上皮鳞状化生，C组气管纤毛多呈片状缺损，而D组多呈灶状缺损；两组气管黏膜炎性细胞浸润均较第7天为重，伴气管浆液腺上皮黏液腺化生（D组≥40%、C组≤20%）、肺泡淤血、水肿。C组皮肤表皮呈不规则皱缩、毛球数明显减少；C组、D组足垫部皮肤表皮增厚、汗腺减少伴结缔组织增生。

Results The neurological defects score was singificiant lower in D group (41.50±8.18) than that in group A(82.20±8.08), B(63.60±6.22) and C (61.00±6.58)(all P.01). Compared with groups A, C, group D showed that the cellular nucleus was shrinkaged slightly, the gap of cells was nomal roughly and the degree of rarefaction in neurofibra net was not serious under the light microscope. Under the electron microscope, the neuron was degenerated, the cellular membrane was integrated and the plastosome was swelled lightly in the group D. The physiological demage were slighter in D group.

结果 D组神经功能缺损评分(41.50±8.18)明显低于A组(82.20±8.08)、B组(63.60±6.22)和C组(61.00±6.58)（均P.01）；光镜下D组神经细胞核轻度皱缩，细胞周围间隙大致正常，神经纤维网疏程度不严重；电镜下D组神经元轻度变性，细胞核无皱缩，核膜完整，线粒体轻度肿胀；脑组织病理损害程度较B组和C组明显减轻。

Lugalbanda was a god and shepherd king of Uruk where he was worshipped for over a thousand years.

Lugalbanda 是神和被崇拜了一千年多 Uruk古埃及喜克索王朝国王。

I am coming just now,' and went on perfuming himself with Hunut, then he came and sat.

我来只是现在，'歼灭战perfuming自己与胡努特，那麼，他来到和SAT 。