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cristae相关的网络例句

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The mitochondria were highly bloated, accompany with disappeaance of the cristae and vacuolation of matrix.

鼻内镜术中光和热的作用可以认为是对鼻腔粘膜组织的一种创伤,这种创伤可以引起相应组织内与创伤修复有关的因子改变。

In group B, a large amount of inflammatory cell infiltration in liver tissues were noted and hepatic cells showed spotty necrosis and bleeding under the light microscope; the electron microscope showed a significant decrease of mitochondria cristae, the broadness of endoplasmic reticula, hepatic cell necrosis and karyopyknosis.

B组光镜下可见肝组织大量炎性细胞浸润,肝细胞点状坏死及出血;电镜下可见线粒体嵴明显减少,内质网扩张,出现凋亡小体及核固缩。C组上述病理变化明显减轻。

Light microscope and transmission electron microscopy showed that SMMC-7721 cells induced by SAHA had undergone the restorational alteration in morphology and ultrastructure, which were different from those of nontreated cells but were similar to those of normal cells, and the changes were as follows: the cells turned to be flat and spread; the nucleo-cytoplasmic ratio lessened and nuclear shape became rather regular; the number of nucleolus reduced and its volume lessened; euchromatin increased while heterochromatin decreased in nucleus; in the cytoplasm, mitochondria grew in number with relatively consistent structure and well-developed mitochondria cristae; Golgi complex turned to be well-developed and typical; rough endoplasmic reticulum increased. Immunocytochemistry assay showed that the expression of AFP and PCNA were declined significantly. FCM analysis showed SAHA could arrest SMMC-7721 cells in G0/G1 phase, with an accumulation of the cells in G0/G1 phase while a decrease of cells in S phase. Semi-quantitative RT-PCR detection revealed that the expression of p21WAFl mRNA was upregulated remarkably in the cells treated with SAHA.

结果:倒置显微镜和透射电镜观察显示,经SAHA处理的细胞增殖速度显著减慢,细胞体积增大,细胞核较小,形状较为规则,核仁数量减少、体积变小,核内常染色质增多而异染色质减少,核质比例减小,细胞质内线粒体数量增多、线粒体嵴发达,高尔基体较为典型,粗糙型内质网增多,呈现出与正常上皮细胞相似的形态变化;MTT比色法测定结果显示不同浓度(2.5、5.0、7.5、10.0uM)SAHA对SMMC-7721细胞的增殖均有抑制作用,并有明显的剂量依赖和时间依赖关系;免疫细胞化学检测显示SAHA能显著降低PCNA和AFP在SMMC-7721细胞中的表达;流式细胞仪检测结果显示,SMMC-7721细胞经SAHA处理后,G0/G1期细胞明显增加,S期细胞则明显减少,细胞被阻滞于G0/G1期;RT-PCR检测结果表明,SAHA作用12h后SMMC-7721细胞中p21WAF1 mRNA的表达即有增加,24h后更为明显。

The transmission electron microscope revealed endoplasmic reticula were dilated, the matrix of mitochondria became pale and the cristae of mitochondria shorter in stromal cells from irradiation mice compared with those of controls.

提示:放射复合伤骨髓基质细胞的细胞周期比例、DNA含量和形态的改变可能是导致放射复合伤造血微环境损伤的重要原因之一。

Electronic microscopic observations showed that the swollen mitochondria with cristae blurred appeared in some pyramidal cells. Some pyramidal cells showed degenerative changes with some disappeared endoplastic reticula and ribose nuclear protein and increased lysosomes. The amount of extends endothelial projections going to the lumen of capillaries were increased.

同时伴有超微结构的改变,如神经细胞线粒体肿胀,内质网扩张,染色质凝集成块状,毛细血管外间隙扩大,髓鞘变性坏死,微管排列紊乱,小胶质细胞有吞噬变性坏死的神经纤维成分等。

Most of mitochondria in renal proximal tubule swelled with fragmentized cristae, and some appeared vacuolar degeneration.

恢复供氧48小时后,ATP、AXP含量为正常对照组的76%、79%,ADP、EC水平恢复正常。

Ultrastructural observation showed that there was a great deal of damage by Cd2+ pollution:The thylakoids of chloroplast and cristae of mitochondria became swollen or vesiculate and disintegrated finally;The nucleus were distorted and the nuclear membrance was disrupted.

但随着毒害的加重,当超出细胞的保护限度,将会导致保护酶活性降低,抗氧化物质含量下降,而丙二醛等膜脂过氧化产物大量产生,膜透性增加,使植物体受害加剧。

It is on the large surface area provided by the inner cristae of mitochondria that ATP-generating

线粒体自我复制,并且可能是自由生活的原核生物在进化中形成的后代。

When the hearts were preserved at the time of 144 hours, the mitochondrial cristae of CM were reduced or disappear.

本研究旨在从形态学的角度,探索对供心具有长期保护作用的保存液,以期延长供心的保存时间。

It is on the large surface area provided by the inner cristae of mitochondria that ATP-generating enzymes are located.

ATP 产生酶存在于线粒体上内质体的巨大表面上。

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