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Results The weight of tumor was reduced and the inhibitory rate of tumor weight was 36.83% in group B (P.05 compared with group A); incidence of metastatic carcinoma of lung was 100% in group A and metastatic nodules of lung were decreased in groups B and C (P.05 or P.01 compared with group A); lymph node weight in homolateral popliteal fossa and iliac periartery was lighter in group B and C than that of group A (P.05); life-span prolongation rate was 37.77% and 36.77% in groups B and C respectively and survival time did not differ much from that of group A.

结果］髓清丸低剂量组的瘤质量较模型组减少(P.05)，抑瘤率达到36.83%；模型组肺部转移率达100%，髓清丸高、低剂量组的肺部转移结节均较模型组减少，转移率较模型组低（P.05或P.01）；髓清丸高、低剂量组同侧窝、髂动脉旁淋巴结质量均较模型组减小(P.05)；髓清丸高、低剂量组小鼠生命延长率分别为37.77%、36.77%，但生存时间与模型组比较未见显著性差异。

Based on this,We designed a series of experiments to study the treatment and its mechanism of Iridoids composition of Valeriana jatamansi Jones of the IBS animal model.1 Research on Iridoids composition of Valeriana jatamansi Jones in the role of gastrointestinal function.1.1 Effection of Iridoids composition of Valeriana jatamansi Jones on gastrointestinal function of normal miceMethods:Compared with normal group and YJBH group,mice which has given Iridoids composition of Valeriana jatamansi Jones as 71.2mg/kg,35.6mg/kg,17.8mg/kg,8.9mg/kg was detected gastric emptying and small intestinal propulsion after 4 days drug given.Results:Compared with normal group,gastric emptying and small intestinal propulsion in 35.6mg/kg and 17.8mg/kg group was inhibited.

课题通过观察蜘蛛香环烯醚萜对正常及IBS模型动物的胃肠道功能、精神活动的影响，以及蜘蛛香环烯醚萜干预下IBS大鼠结肠、血清、中枢5-HT含量，结肠5-HTR3A受体的mRNA和色氨酸羟化酶(TPH1)的mRNA表达，结肠和血浆中SP和VIP含量以及结肠中肥大细胞分布，探讨蜘蛛香环烯醚萜对腹泻型IBS的治疗作用及其作用机理，为临床治疗IBS提供实验依据。1蜘蛛香环烯醚萜对胃肠功能的作用目的：探讨蜘蛛香环烯醚萜对胃肠功能的影响1.1蜘蛛香环烯醚萜对正常小鼠胃肠功能的作用方法：正常小鼠灌胃给予蜘蛛香环烯醚萜71.2mg/kg、35.6mg/kg、17.8mg/kg、8.9mg/kg与空白组和越鞠保和丸组对照，给药4日后测定动物的胃排空率和小肠推进率。

This classifier finished TM landcover by fuzzy logic reasoning, and the unclassified pixels increase K adaptively to be classified; It improved 2.7% and 2.9% in overall accuracy and Kapp coefficient compared with MLC, deceased 1% in Kapp coefficient and no change in overall accuracy compared with self-organizing network.

对拒分类的像元，自适应增加K值使其可分。该方法所得分类精度及Kapp系数与最大似然分类方法结果相比分别提高了2.7%和2.9%；与自组织网络相比，总精度相差不大，而Kapp系数低1%。

ResultsAs for the lengthes of mandibula and mandibula r bodies in experimental group increased compared with control group, and the non mastication side increased compared with mastication side in experimental group

结果 ：下颌骨长度、下颌骨体长度实验组较对照组增长，实验组非咀嚼侧较咀嚼侧增长

In the L-Arg group, the amount of NO and SOD increased significantly (P.01, P.01), whereas that of MDA decreased (P.05), and the damage in the nephric tissue and renal function was alleviated,as compared with the control group.On the contrary, the amount of NO in the L-NAME group decreased significantly (P.05) compared with the control group.

给予L-Arg组大鼠肾组织中NO含量较对照组增加显著（P.01），SOD活性增高（P.01），MDA水平降低（P.05），肾功能改善，肾脏组织结构损伤减轻。

Results: Compared with IR group , XSOL could significantly decrease the cerebral water content, the cerebral index, the brain vascular permeability in rats of acute incomplete brain ischemia-reperfusion (P.01,P.05). Cerebral tissue morphology showed that neuronic damage of rats subjected to acute incomplete brain ischemia-reperfusion was ameliorated following administration of XSOL (2.4, 9.6g/kg). Compared with IR group , XSOL could significantly decrease the content of MDA and NO in brain tissue(P.01,P.05)and increase SOD,GSH-px activity. The levels of cerebral Glu,Asp were cut down(P.01,P.05). Conclusion: XSOL shows protective effects on cerebral ischemia-reperfusion.

结果：与缺血再灌注组比较，XSOL大、小剂量组可以显著降低急性不完全性脑缺血再灌注模型大鼠的脑含水量(P.01，P.05)、脑指数(P.01，P.05)、脑毛细血管通透性(P.01，P.05)；光镜下脑组织形态学显示各剂量组可不同程度的减轻急性不完全性脑缺血再灌注大鼠脑组织的神经元受损形态；与缺血再灌注组比较，XSOL大、小剂量组可以显著降低缺血再灌注大鼠脑组织中的MDA的含量(P.01，P.05)，升高SOD、GSH-Px在脑组织中的活性(P.01，P.05)；XSOL大、小剂量组可显著降低脑组织中NO的含量(P.01，P.05)，降低脑组织中Glu、Asp含量(P.01，P.05)。

Results: Small round or linear haemorrhagic erosions were induced in the oxyntic glandular area after rats exposed to WRS. Meanwhile, GMBF decreased and NO contents in plasma and gastric mucosa increased significangly compared with those without WRS. L-NAME not only further decreased GMBF and inhibited the increases of NO contents but also aggravated the extent of gastric mucosal lesions. In contrast, L-Arg increased both GMBF and NO contents resulting in the lessening of mucosal injuries. All these changes were significant compared with control group.

结果：浸水束缚应激4 h可致严重的胃粘膜损伤，同时粘膜血流量明显下降，血浆及粘膜NO含量明显升高，与正常对照组相比差异非常显著（P＜0.01）；L-NAME明显加重粘膜损伤，并阻抑了应激所致的血浆及粘膜内NO含量的升高，使粘膜血流量更趋下降，与实验对照组相比差异显著（P＜0.05）；L-Arg可明显升高粘膜血流量及血浆、粘膜内NO含量，明显减轻粘膜损伤程度，与实验对照组相比差异非常显著（P＜0.01）。

Results On the basis of the percentage of GPI-anchored PLAP conversion,the sera GPI-PLD activities of total 172 patients,included 26 severe acute viral hepatitis as group A,29 liver cirrhosis as group B,32 chronic viral hepatitis as group C,55 mild acute viral hepatitis as group D,30 primary hepatocellular carcinoma as group E,were measured.As compared with 182 healthy presons as control group,the sera GPI-PLD activities of group A and B were significantly reduced;By contraries,the activities of group D and E were significantly raised.The sera GPI-PLD activities of group C compared with healthy control group were not significantly altered.However,when paired Q-test,the changes of serum GPI-PLD activity between all paired groups among this five groups were remarkable.

结果检测172例肝脏疾病患者(包括A组：26例急性重症病毒性肝炎；B组：29例肝硬化；C组：32例慢性病毒性肝炎；D组：55例急性非重症病毒性肝炎；E组：30例原发性肝癌)血清中GPI-PLD的活性，发现A、B两组患者的该酶活性较正常人(182名)显著性降低，而D、E两组则较正常人显著性增高，C组患者的酶活性与正常人无显著性差异，但A、B、C、D、E两组之间的酶活性水平改变却均有显著性。

Results Protein oxidation and lipid oxidation decreased significantly in HO-2 knockout mice at 72 h compared with that of wild-type mice. Striatal cell viability increased significantly in knockout mice at 72 h compared with that of wild-type mice.

结果 与野生型相比，基因(HO-2)敲除小鼠纹状体内蛋白和脂类的氧化作用显著降低，而纹状体细胞的存活率显著增加；HO-1的表达在两种小鼠注射前后没有明显差异。

Results: The mean UI in Chongqing district were higher than proper iodide standard defined by international Council for control of iodine deficiency disorders,and the UI in HT groups were significantly increased compared with Con(P.05). The thyrocele and ophthalmopathy in AITD became more and severer accompanying UI increasing, the rate of goiter and endocrine ophthalmopathy in high iodide AITD was significantly higher than PI AITD( P.05).In the meantime, the positive percentage of TGAb and TPOAb also increased evidently in HI compared with PI(P.05).Cytokine IL-1?

结果：三组患者的平均UI水平高于国际碘缺乏理事会规定的适碘范围，HT患者的UI水平较正常人显著升高（P.05）；在AITD患者UI水平的升高伴随着甲状腺肿大、眼部并发症等临床症状的加重，高碘组与适碘组相比差异有显著性（P.05），且随着UI水平的提高，TGAb和TPOAb阳性百分率明显提高，不同UI水平各组间和每两组间有显著差异(P.05)。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。