查询词典 collagen
- 与 collagen 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Results Blood sugar level was decreased by Merine collagen peptide ,but The blood sugar did not change significatively during the experiment .
目前认为,糖尿病的发生机制与胰岛素抵抗和机体高血糖、高血脂等因素诱导的抗氧化应激功能失调有显著的相关性。
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Objective: To study age-related variation of ratio and type of collagen fibers in human sinoatrial node.
目的了解窦房结的胶原纤维年龄变化及分型。
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Results: The ratio of I and Ⅲ collagen fibers in the sinoatrial nod increased with aging. Type I was the majority while type Ⅲ decreased during aging.
结果:窦房结胶原纤维随年龄的增高有增高的趋势,发现胶原纤维以1型为主,3型胶原纤维随年龄的增高有减少的趋势。
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Objective: To investigate the reconstruction of type Ⅰ and Ⅲ interstitial collagen of sinuatrial node and the intervention role of insulin in type Ⅱ diabetes mellitus rat.
目的:探讨Ⅱ型糖尿病大鼠窦房结内Ⅰ、Ⅲ型间质胶原重构及胰岛素对其的影响。
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Lobulatedcharacter,spiculated sign,significant enhancement and nodal metastasiscorrelated well with discontinuous type Ⅳ collagen,predicting high risk ofmetastasis and poor prognosis.
周围型肺癌的大小、毛刺征、强化显著以及淋巴结转移等都和Ⅳ型胶原异常表达关系密切,说明这些征象提示肺癌恶性度高,预后不良。
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The degrees of fibrosis of sternomastoid muscle in CMT are related to the hyperplasy of collagen type IV.
病变组中纤维型组IV型胶原较肌肉型组有更多的表达,两者比较有显著性差异(P.01),纤维型和肌肉型组与对照组比较均有显著性差异(P.01)。
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Objective To investigate the expression and hyperplasy of Collagen Type Ⅰand Ⅲ in sternomastoid muscle of congenital muscular torticollis and to explore the pathogenesis of sternomastoid muscle fibrosis.
目的研究先天性肌性斜颈胸锁乳突肌Ⅰ、Ⅲ型胶原的增生和表达情况,探讨先天性肌性斜颈胸锁乳突肌纤维化的发生机制。
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Uses: Cosmetic Treatment: Rejuvenates the skin, attenuates the wrinkles and the expression lines, natural generator of collagen, nourish and hydrates, skin regenerator, prevents the striae, attenuate the spots of skin, protects the dehydration by wind or dry atmosphere.
用途: 美容治疗: rejuvenates皮肤,减轻皱纹,并表达线,自然生成胶原蛋白,营养和水合物,皮肤再生,防止皮纹,衰减点的皮肤,也保障了脱水风或干燥的气氛中进行。
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Objective: To explore the mechanisms resulting in the recurrence of urethral scar which make urethral strictures difficult to be cured, a series experiments were conducted to find potential effective factors involved in urethral scar formation and degradation, including the studies of extracellular matrix component of urethral stricture scar, the characteristics of urethral scar fibroblast, and the effects of urine on urethral fibroblast in vitro, as well as the studies to compare the difference of collagenase activity, type Ⅰ collagen and tissue inhibitor of metalloproteinase-1 (TIMP-1) in the tissues and cultured fibroblasts from normal urethra and strictured urethra respectively, and the studies to investigate the effect of antisense TIMP-1 oligodeoxyonucleotide on cell proliferation and collagenase activity of urethral scar fibroblast.
中文题名尿道瘢痕基础研究副题名胶原酶活性,TIMP-1的表达及其反义基因治疗外文题名 Experimental study on urethral scar-activity of collagenase,expression of TIMP-1,and antisense TIMP-1 gene transfection of urethral scar fibroblast 论文作者黄翔导师杨宇如教授学科专业外科学研究领域\研究方向学位级别博士学位授予单位四川大学学位授予日期2002 论文页码总数104页关键词尿道手术瘢痕胶原酶成纤维细胞尿道瘢痕馆藏号BSLW /2003 /R699 /12 目的:研究尿道瘢痕的细胞外基质的组成,尿道瘢痕成纤维细胞的生物学特性以及尿液对其生长的影响;比较胶原酶活性,金属蛋白酶组织抑制因子-1(TIMP-1)以及Ⅰ型胶原含量在尿道瘢痕和正常尿道组织及体外培养的成纤维细胞中的差异;研究反义TIMP-1寡核苷酸对尿道瘢痕成纤维细胞增殖以及胶原酶活性的影响。
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P<0.01. 28d, q=4. 0452, p<0.05 . Corresponding pathologic result shows that the corneal stromal of height edema increase thickness to have infiltration of inflammation cells with hyperplasia of fibroblast cell and large vessel. The collagen fiber in stromatic layer is dissolved and destroyed seriously, suggesting that in addition to HSV-1's direct destructuring effect, inflammatory cells that might play an important role in the pathology of HSK induce the immune-pathological reaction.
相应的组织病理学研究结果显示(UV-B 14天):病毒对照组角膜基质层高度水肿、增厚,并有大量中性粒细胞等炎性细胞浸润、成纤维细胞和新生血管增生,胶原纤维肿胀、分解等,说明除HSV-1对角膜组织直接破坏外,炎性细胞介导的免疫病理反应在复发性HSK发病机理是重要的因素。
- 推荐网络例句
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。