查询词典 collagen

The latter is due to adventitial chronic inflammation and excessive perivascular collagen deposition.

而后者主要是因为外膜慢性炎症以及血管周围过多的胶原沉着引起。

Tendon cells 5 x 10(4/ml derived from phalangeal flexor tendon of SD rats were co-culture with CPPF scaffold or CPPF scaffold resurfaced with collagen type-I within 1 week. The co-cultured specimens were examined under optical and electric scanning microscope.

将体外培养 SD大鼠原代趾屈肌腱细胞，以 5×10 6/ ml浓度与 CPPF辫状支架或型胶原涂层的 CPPF辫状支架进行体外混合培养，组织学观察材料与细胞的吸附情况。

Three kinds of molecular weight determination methods, i.e., size exclusition chromatography, sodium dodecyl sulphate-polyacrylamide gel electro phoresis and high performance liquid chromatography / electro spraying ionization-mass spectrometry (HPLC/ESI-MS) are introduced to study of the attribute of collagen hydrolysates.

应用体积排阻色谱、聚丙烯酰胺凝胶电泳、高效液相与电喷雾质谱联用(HPLC/ESI-MS)这三种蛋白质分子量分析测试技术，来表征胶原水解产物的分子量，以寻求较好的胶原水解产物分子量的测试方法

Expression of collagen I,II and alkaline phos┐phatase in osteogenesis by rhBMP┐2in vivo

rhBMP┐2在体内诱导成骨中I，II型胶原及碱性磷酸酶的表达

Photothermic and photochemical action will be triggered by the radiation of IPL. On one hand, IPL will promote collagen reproducing and recombining which makes the skin more flexible and smooth. On the other hand, the light with long wavelength can easily pass through epidermis layer and will be absorbed selectively in priority by pathological pigments which will be destroyed by high heat, and get the effect of remove pigmentations, vascular lesions and unwanted hair.

当强脉冲光照射皮肤时，会产生生物刺激作用和光热解作用，一方面使皮肤中的胶原纤维和弹力纤维再生并重新排列，恢复皮肤弹性，从而减轻皱纹；另一方面，强脉冲光能穿透皮肤，被组织中的病变色素团优先选择性吸收，热能在不破坏正常皮肤组织的前提下，击碎病变色素团，从而达到治疗毛细血管扩张，祛除色斑，和脱除多余毛发的效果。

And Sirius Red in a supersaturated picric acid solution viewed by polarization microscopy. The changes of mRNA expression of collagen type I were analyzed by RT-PCR. The results were compared with those after intervention with Losartan.

染色及苦味酸天狼猩红胶原特异染色偏振光显像进行心脏间质胶原的组织化学观察，RT-PCR观察心脏I型胶原mRNA表达的变化，并与氯沙坦干预后比较。

The skin from the core to solve the problem, repair damaged cells, proliferation of bone collagen and keratosis protein, will have been deformed shrinking and the "plum" shape of the damaged cells back to health, to complete the "bulge pitapat."

由核心解决肌肤问题，修复受损细胞，增生骨胶原和角化蛋白，将已变形萎缩和&话梅&状的受损细胞回复至健康完整的&胀卜卜&。

The human cartilages are composed of chondrocyte and extracellular matrix,the form of chondrocytes are hypertrophy and the quantity are less;the ECM of cartilage are compised of type II collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury,the ommilayer injury ofcartilage can be recovered by marrow,but because of without stimulation mechanism,the new tissues are merely fibrocartilages,they can not be coincide with hyaline cartilage in menchanics;the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation,thus,the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.Ever since,people tried their best to find a way to reconstruct articular cartilage.

造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

Both Fas and NO can lead chondrocyte apoptosis respectively and cause articular cartilage destruction. IGF-Ⅰ, TGF-β, bFGF, BMP and other growth factors are polypeptide agents that can influence cell activity by signal convection. They can accelerate chondrocyte proliferation and proteoglycan synthesis, play the local regulation action on formation and plerosis of bone and cartilage tissue by autocrine or paracrine. They have the ability to induce cartilage formation. Some investigations showed that growth factors can influence chondrocyte metabolism, synthesis of specific Ⅱ type collagen and proteoglycan by co-operation and inhibition. 1. 3 Situation of OA therapeutics The therapeutic methods of OA mainly comprised non-drug treatment, drug treatment, operation treatment, tissue and genetic engineering, et al. Drug treatment is the chief method among them.

若其活性发生改变，则将导致关节软骨基质成分的丢失和进行性破坏；软骨细胞凋亡与OA的发病密切相关，Fas与NO可各自独立介导软骨细胞凋亡，造成关节软骨破坏；IGF-Ⅰ、TGF-β、bFGF、BMP等生长因子是一组通过细胞间信号传递影响细胞活动的多肽因子，具有促进细胞生长、增殖与合成等作用，可通过自分泌或旁分泌方式对骨和软骨的形成和修复起局部调节作用，可促进软骨细胞增殖、分化与蛋白多糖的合成，具有较强的诱导软骨形成的能力，研究表明多种生长因子相互促进、相互抑制，以协同或拮抗方式影响软骨细胞代谢，影响软骨细胞特异性Ⅱ型胶原和蛋白多糖的合成分泌。

The human cartilages are composed of chondrocyte and extracellular matrix , the form of chondrocytes are hypertrophy and the quantity are less; the ECM of cartilage are compised of type Ⅱ collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury, the ommilayer injury ofcartilage can be recovered by marrow, but because of without stimulation mechanism, the new tissues are merely fibrocartilages, they can not be coincide with hyaline cartilage in menchanics; the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation, thus, the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.

造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。