查询词典 cml.

Some VAI double-current conticaster Anshan frame one shearing force wall concrete structure, 11.43-14.12 (M-1) assembles bigger beam board post shearing force wall surface modulus CML = in 3 ~ 11 centigrade-, for satisfying equipment still needing the early phase intensity suitable.

某VAI双流连铸机鞍山框架一剪力墙混凝土结构，梁板柱剪力墙表面系数较大CML=11.43-14.12(M-1)又处在-3~11摄氏度，为满足设备安装尚需要相当的早期强度。

The protein product of BCR-ABL is a constitutively active tyrosine kinase that drives the abnormal proliferation of CML cells.

急变期是CML的晚期，在此期间常常出现其它附加染色体和分子的改变。

It is concluded that the value of routine cytogenetical and molecular biological analysis in diagnosis for potential CML cases,which mimicked ET as in this presentation,is very distinctive,and the importance is magnified by the recent availability of imatinib,a specific inhibitor of the bcr/abl tyrosine kinase produced by the Philadelphia chromosome.Every case of

对血小板明显增多的患者应及时进行Ph染色体及bcr/abl融合基因表达水平的检测，这对于ET及CML的诊断和鉴别诊断极为重要，以避免误诊、误治。

Altogether 60 cases of CML were studied from August 1993.They were hematologically typical and cytogenetically ph1+.

从1993年8月起一共研究了60名 CML 患者，他们为血液学典型病例，细胞遗传学 PH1+。

Imatinib, a BCR-ABL tyrosine kinase inhibitor, is able to induce a complete cytogenetic response in more than 85% of patients with CML. However, the researchers note that patients with a complete cytogenic response tend to relapse when imatinib is discontinued, and less than 10% of patients achieve a molecular remission, defined by an undetectable residual disease using real-time quantitative–polymerase chain reaction.

Imatinib是一种BCR-ABL酪胺酸激酶抑制物，可以在85%以上的CML病患诱导完整的细胞生成反应；不过，研究者指出，完整细胞生成反应的病患倾向在停用imatinib时复发，不到10%的病患达到分子生物学上的缓解，其定义为以即时定量聚合酶连锁反应侦测不出残余疾病。

Imatinib, a BCR-ABL tyrosine kinase inhibitor, is able to induce a complete cytogenetic response in more than 85% of patients with CML. However, the researchers note that patients with a complete cytogenic response tend to relapse when imatinib is discontinued, and less than 10% of patients achieve a molecular remission, defined by an undetectable residual disease using real-time quantitative–polymerase chain reaction.

Imatinib是一种BCR-ABL酪胺酸激酶抑制物，可以在85%以上的CML病患诱导完整的细胞生成反应；不过，研究者指出，完整细胞生成反应的病患倾向在停用imatinib时復发，不到10%的病患达到分子生物学上的缓解，其定义为以即时定量聚合酶连锁反应侦测不出残餘疾病。

RESULTS: The Bcl-x deamidation pathway was inhibited in myeloid cells, but not T cells, in patients with CML or polycythemia vera.

结果：CML患者或真性红细胞增多症患者的Bcl -×脱酰胺途径受抑制是在髓系细胞中，而不是T细胞。

BCR-ABL and mutant JAK2 inhibit the Bcl-x deamidation pathway and the apoptotic response to DNA damage in primary cells from patients with CML or polycythemia vera.

BCR-ABL和突变的JAK2可抑制CML或真性红细胞增多症患者Bcl -×的脱酰胺途径和原代细胞DNA损伤诱导的凋亡反应。

Subsequent molecular studies discovered that this mutation led to gain-of-function of GATA 2, partly blocking myelo-monocytic differentiation and resulted to acute transformation of CML.

随后的分子生物学研究结果提示该突变导致获得性GATA-2功能增强，部分阻滞了CML髓系祖细胞的分化，为慢粒发生恶性转化提供了重要的分子学证据。

Gleevec is safe in the CML patients and dont delay the engraftment or hemapoietic reconstruction.

对于CML进展期患者，先用格列卫治疗然后行移植，可以降低体内的Ph细胞负荷。

And Pharaoh spoke to Joseph, saying, Your father and your brothers have come to you.

47：5 法老对约瑟说，你父亲和你弟兄们到你这里来了。

Additionally, the approximate flattening of surface strip using lines linking midpoints on perpendicular lines between geodesic curves and the unconditional extreme value method are discussed.

提出了用测地线方程、曲面上两点间短程线来计算膜结构曲面测地线的方法，同时，采用测地线间垂线的中点连线和用无约束极值法进行空间条状曲面近似展开的分析。