英语人>网络例句>chondriosome 相关的网络例句
chondriosome相关的网络例句

查询词典 chondriosome

与 chondriosome 相关的网络例句 [注:此内容来源于网络,仅供参考]

Because chronic obstructive inflammation of airway severely influences the ventilation and exchange function of COPD patients, The motor ability of patients is limited .Despnoea and fatigue limit patients to sedentary,because of this, skeletal muscle began to analosis,oxygen uptaking capacity of skeletal muscle descends,and concentration of chondriosome degrades.Function limitation of skeletal muscle causes oxygen needs increase,so vicious cycle emerges,and further to restrict patient's respiratory and motor function.

由于气道的慢性炎症性阻塞,严重影响了慢阻肺患者的通气及换气功能,使得患者的运动能力明显受限,因喘息和疲劳而限制了患者的运动能力,使患者出现久坐,由此出现骨骼肌的萎缩,骨骼肌摄氧能力下降,线粒体氧浓度降低,骨骼肌的功能受限又会引起通气量不足,进而引起低氧血症和高碳酸血症,使患者的摄氧需求增加,由此出现恶性循环,进一步制约了患者的呼吸和运动功能。2001年正式提出肺康复作为中重度COPD患者稳定期非药物治疗的首选管理方案。

The chondriosome signal transduction pathway: In the existing of dATP, Cytc combines with Apaf-1, to exposure CARD motif on Apaf-1, and then combines CARD of proaspase-9 into apoptosome, The result is the activation of procaspase-9, and the latter activates Caspase-3, 6s, according induces apoptosis.

已有研究表明,某些细胞在其发生发展过程中Fas的表达不断增加,而凋亡抑制基因Bcl-2的表达逐渐减少,最终导致细胞凋亡的发生,完成细胞代谢过程,达到细胞增殖与死亡的平衡。

Although the pulmonary tissue pathological injuries were also relieved and the rates of pathological changes of chondriosome were comparative in Jinshuibao capsule group as compared with those in the COPD model group, the efficacy of Jinshuibao capsule was not as good as that of Shenqichongcao capsule.

金水宝胶囊组肺组织病理损伤较模型组亦有所减轻,但作用不如参七虫草胶囊组显著;线粒体病变发生率与COPD模型组相当。

The pupae were exposed to 9 krad 60Co-γ rays for two days before emergence.The adults were dissected and many mature sperms could be seen in the male flies.There were no significant difference in the form,number and vigor of sperm between irradiatid and unirradiated male flies,but there were some abnormal ultrastructurail changes in the sperm of irradiated male flies such as the appearence of vacuoles in nutrient cells,disarrangement of sperms,variation of the number of chondriosome derivates,axonemes ction of formation and abnormal cell division of sperm cells.

用9krad辐照羽化前2天的蛹,成虫羽化后解剖观察,雄虫已有大量成熟精子,精子形态、数量和活力与未经辐照处理的雄虫无显著差异,但其超微结构则发生了某些异常变化,如营养细胞出现空泡、精子排列零乱、线粒体衍生体和轴丝增多或减少、构型被破坏、精子细胞分裂异常等。

Results indicated that the variation was significant from treatment to treatment. S-deficiency caused bulging chloroplasts, loosening and dishevelled grana lamella, reducing organelle, and damaged chondriosome in mesophyll cells of the leaves, and empty cells of the roots, while S-excess, however, led to densification, condensation and randomization of grana lamella of the chloroplasts of the leaves, and separation of cytoplasms from walls of the cells of the roots.

结果表明,受S素营养胁迫的水稻,其根系及叶片的超微结构均发生了显著的变化:低S胁迫会造成水稻叶片叶肉细胞内的叶绿体结构肿胀,基粒片层松驰、散乱,细胞器减少,线粒体结构被破坏,根系细胞内几乎没有内含物;S浓度高则会使叶片叶绿体基粒片层致密、浓缩,无规则化,根系细胞发生质壁分离。

The ultramicrostructure in atrial myocytes displayed glycogen aggregation, karyopycnosis, sarcoplasmic reticulum distention and chondriosome swelling at 24 hours after rapid pacing.

经过24 h的快速起搏,心房肌细胞出现糖原聚集、核固缩、空泡样变以及肌浆网扩张、线粒体肿胀、部分线粒体嵴轻度溶解等超微结构的改变。

RESULTS: The transmission electron microscope showed that the retinal photorecepter cells, the retinal pigment epithelium and the retina inner and outer barriers were damaged in the tPVR group. In the tPVR treated with GM 6001 group, the membranous discs of the outer segment of the retinal photorecepter cells were still clear. There were plenty of chondriosome, pinocytosis bullules and smooth endoplasmic reticulum in the cytoplasm. The cell junction was regular.

结果:透射电镜示tPVR组大鼠视网膜光感受器细胞和RPE、视网膜内外屏障受损,而外伤后应用GM 6001组视网膜光感受器细胞外节膜盘结构尚清晰,RPE基底部质膜内褶较tPVR组多,胞质中含大量线粒体、吞饮小泡和滑面内质网,细胞连接规则。

Results By using optical microscope,the neurocytes of hippocampus in experimental group were observed to denaturalize,tumefy,destroy and disappear.And under electron microscope,the karyon was irregular,nucleoles deviated,chondriosome tumefied,crista collapsed,membrane was damaged,substance cavitated.Such changes were more evident with the increased paroxysm of epilepsy,the parameters between control group and operation group had no difference(P>0.01).But make a monofactorial variance analysis among cells' number,area,circumference and integral optical density in different groups,it showed that there were significant differences between optional two groups(P.01;at the same time,the more serious injury in hippocampus,the more powerful GFAP immunoreaction was.

结果: 实验组海马区光镜下可见神经细胞变性、肿胀、坏死及神经细胞脱失改变,电镜可见细胞核形态不规则、有切迹、核仁偏位、线粒体肿大、嵴断裂、膜破损和基质空化等改变,随癫痫发作次数增加上述改变越明显,对照组与手术对照组比较,各参数差异无显著性(P>0.05),将不同组别大鼠细胞数、面积、周长、积分光密度分别作单因素方差分析,不同组别间均差异有显著性(P.01),同时观察到海马区GFAP免疫反应阳性的胶质细胞随海马损伤的加重而GFAP免疫反应增强。

The transmition elec-microcope observation indicated that kangshuai Ⅰ prescription can chang the ultramicro fabric of nerve cell in quatar CA1 in hippocampus of the model rats by affecting the cell organs such as karyon、rough endoplasmic reticulum 、chondriosome、dictyosome and lipofuscin, which has similar effect with the original prescription.

透射电镜结果提示抗衰Ⅰ号各剂量组可以改善衰老模型大鼠海马CA1区细胞核、粗面内质网、线粒体、高尔基体、脂褐素体等细胞器的变化,尤以高剂量改善明显,与强力抗老液作用相仿。

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