查询词典 cerebral

The purpose of the research is to exploring the mechanism of proctoclysis of Tongfuxingshen liquid treating unconscious patients with cerebral hemorrhage and encephaledema, cerebral vessels permeability and expression of HSP70, HO-1mRNA after cerebral hemorrhage in rats by TCM theory and clinical, experiment. Providing the theoretic and clinical, experimental evidence for it, and exploring the new administration ways to treating acute intracerebral hemorrhage.

本文试图从理论、临床和实验研究探讨导师经验方——通腑醒神胶囊直肠滴注治疗出血中风的机理，通过观察对脑出血神昏病人意识状态及对脑出血大鼠脑水肿及脑血管通透性、脑组织HO-1mRNA和HSP70表达的影响，探讨通腑醒神液直肠滴注治疗出血中风的作用机制，为通腑醒神液直肠滴注治疗脑出血提供理论、临床及实验依据，探索抢救脑出血急性期昏迷患者新的给药途径。

However, cysts in the suprasellar cistern, cerebellopontine angle, and sylvian cistern may cause cerebral arteritis with subsequent infarction of the middle cerebral or posterior cerebral distribution.

而位于鞍上池、桥小脑角、以及大脑外侧裂池的囊虫病可以引起大脑动脉炎伴随着继发性的大脑中动脉和后动脉梗塞。

Objective To study the MR findings and its relationship with clinical manifestions of cerebral palsy caused by hypoxic-ischemic brain damage.The pathological mechanisms of MR findings were also explored.Methods The MR findings of14patients with cerebral palsy caused by hypoxic-ischemic damage were studied retrospectively and correlated with clinical findings.All those patients underwent MRI.Cerebral cortex and subcortical white matter,deep white matter,basal regions and thalami,and ventricles and subarachnoid space were observed on T1WI and T2WI.

目的 通过分析14例缺氧缺血性脑损伤所致脑瘫的MR成像特点及临床表现，讨论了该病的MRI表现与临床表现的相关性，以及MRI表现的病理损伤机制方法对14例缺氧缺血性脑损伤所致脑瘫患儿行MR检查，在T1WI及T2WI上对脑皮层及皮层下白质、深部白质、基底节、丘脑、以及脑室和蛛网膜下腔等部位进行观察。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后，血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少，Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少，血小板聚集程度及红细胞聚集指数显著上升，病理切片显示胸主动脉内皮细胞破坏明显，脑心肾组织内的血管进一步增厚甚至几乎阻塞，导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR，脑体重比显著升高；心肌内出现明显的缺血梗塞疤痕灶，心体重比显著升高；肾小球明显萎缩、玻璃样变性，肾小管也变性甚至坏死，使肾硬化程度显著增加呈颗粒性固缩肾，肾体重比也显著减少。

Objective To explore the association between the level of folate,VitB12 and acute cerebral infarction disease.Methods 88 acute ischemic cerebrovascular stroke disease events were disparted in two subgroups,32 of arteriosclerotic cerebral infarction,56 of lacunar cerebral infarction,and 35 contrast group were set up.All the participants' serum folate,VitB12 were measured when they entered the hospital.

目的 探讨急性脑梗死患者血清叶酸、VitB12水平及其临床意义方法对88例首次发病的急性缺血性脑中风患者分两亚组，动脉硬化性脑梗死组32例，腔隙性脑梗死组56例，并设对照组35例，入院后用罗氏公司Elecsys 2010型全自动电化学发光免疫分析仪测定血清叶酸、VitB12浓度。

Every group rats were divided into two parts with the same rats. At last the rats were redivided ,then them were killed and their brains were excluded after one hour.4 hours or one week.Results PO2 and cerebral blood flow of E-selectin 2mg/kg group were higher than that of saline and lower than that of l0mg/kggroup(p.01).The volume of cerebral infarct and blood speed of E-selectin 2mg/kg group was smaller than that of saline and larger than that of l0mg/kg group(p.01).At the same time PCh.speeds.flows and the volume of infarct were related with the time (p.01).Conclusion E-selectin can effectively increase PC2 and rCBF after ischemia and reperfusion .reducing infarct volumes and speeds of blood at the same time . Artificial synthetic E-selectin can effectively reduce the reperfusion injury following cerebral ischemia.

结果 生理盐水组，2mg／kg组，10mg／kg组PO_2值依次提高(p＜0.01)，脑皮质血流量依次提高(p＜0.01)；脑血流速度依次减低；脑梗死体积依次减少(p＜0.01)；同时在测定的时间段里PO_2和脑组织血流量值与时间成正相关(p＜0.01)；脑梗死体积和脑组织血流速度值与时间成正相关(p＜0.01)。E一选择素在脑缺血再灌注损伤中表达的实验研究中文摘要结论E一选择素能增加脑缺血再灌注损伤的脑组织氧饱和度，脑血流量，减低脑血流速度；减少脑损伤体积；从而保护了脑缺血再灌注损伤的脑组织。

To approach its" mechanism of action, we carded out this experiment. The SD rats were objects in this experiment. We made rat focal cerebral ischemia model with middle cerebral artery occlusion using a thread block technique. We gave a mark of the neurologic impairment in rat and detected the contents of calcitonin gene-related peptide and neuropeptide Y in rats" cornu ammonis and brain mantle respectively by electro-acupuncture on shuigou(DU 26) acupoint. In this way, we are trying to explore the mechanism of acupuncture therapy in cerebral ischemia.

为了进一步探讨其作用机理，本实验以SD大鼠为研究对象，采用线栓法制作大鼠局灶性脑缺血模型，运用电针水沟进行治疗，并对大鼠神经功能缺损进行评分，检测了大鼠海马及大脑皮质降钙素基因相关肽以及神经肽Y含量，从中枢神经肽类递质的变化探讨电针抗脑缺血损伤的作用机理，为临床治疗缺血性脑血管疾病提供实验依据。

Methods: The cerebral cortical astrocytes and neurons of rats were cultured and the model of simulated cerebral ischemia and reperfusion was set up in vitro. The activity, survival rate, death rate of the neurons, leakage rate of lactate dehydrogenase and the activity of nitric oxide synthase of cerebral cortical neurons were measured after 18 boors of reperfusion.

分离纯化培养大鼠大脑皮质Ast和神经元，体外模拟脑缺血再灌注损伤模型，用ACM培养损伤后的神经元，测定其神经元的活性、存活率、死亡率、培养液乳酸脱氢酶的漏出率和NOS强阳性细胞的表达量。

Results The main injury characteristics of HDI were as follows: scalp contusion,cranial fracture,epidural hematoma,subdural hematoma and cerebral contusion at impact position;subdural hematoma,basal fracture and cerebral contusion at contrecoup site;skull fracture mainly at impact position,subdural hematoma mainly at contrecoup site,subarachnoid hemorrhage mainly at the base of brain and cerebral contusion regions;severe contrecoup injury at frontal and temporal lobe.

结果 颅脑减速伤损伤的主要特点为：撞击部位头皮损伤、颅盖骨折、硬膜外血肿、硬膜下血肿和脑挫裂伤，对冲部位硬膜下血肿、颅底骨折和脑挫裂伤；颅骨骨折以撞击部位多见，硬膜下血肿以对冲部位多见，蛛网膜下腔出血主要位于脑底部及脑挫裂伤区；额、颞叶严重对冲伤是常见颅脑减速伤的重要特征。

Abstract］ objective to investigate the clinical featuremnursing and prognosis for cerebral infarction of aged people above 80 years old.methods selected 50 patients of aged people above 80 years old（maximun age 93）dured 2004 to 2006,who had been conformed cerebral infarction with mri or ct.results there were 38 patients improving and 11 patients died,who suffered from multi-focus of infection,large area and serious complication.conclusion aged people above 80 years old suffered from cerebral infarction with serious symptom,multi-complication and high mortality rate.

目的 探讨80岁以上老年人脑梗死的临床特点、护理及预后。方法取2004～2006年收治的50例80岁以上脑梗死患者（其中最大者93岁），均用头颅mri或ct证实。结果 50例中多数为多病灶、大面积和伴有并发症，治疗后好转38例、死亡11例。结论 80岁以上老年人患脑梗死后症状重、并发症多、死亡率高。

I didn't watch TV last night, because it .

昨晚我没有看电视，因为电视机坏了。

Since this year, in a lot of villages of Beijing, TV of elevator liquid crystal was removed.

今年以来，在北京的很多小区里，电梯液晶电视被撤了下来。