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cerebral相关的网络例句

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Object: Cerebral vasospasm constitutes a major complication of subarachnoid hemorrhage. The pathogenesis of cerebral vasospasm is unclear, in spite of intensive investigations conducted over the past years. Even though multiple factors are proposed to be involved in cerebral vasospasm, oxyhemoglobin is one of the most important spasmogens for cerebral vasospasm that fallows aneurismal SAH.

目的:脑血管痉挛是蛛网膜下腔出血后最常见和最严重的并发症,近年来人们对SAH所致脑血管痉挛的发生机制进行了大量的研究,但迄今尚未完全阐明,众说纷纭,甚至有些观点相反,但几乎所有关于脑血管痉挛原因的报告都认为,脑血管痉挛是多种原因造成的,其中氧合血红蛋白是SAH后血管痉挛的主要启动因素之一,它对内皮细胞及平滑肌的细胞毒作用已经被报道。

Between 6th and 14th weeks afteconstricted bilateral renal arteries, the blood pressure in the majority of RHRSP with cerebral infarction was obviously higher than in RHRSP without cerebral infarction (28. 8±2. 24kPa~32. 3 ±2. 88kPa, 26. 2±2. 01~28. 3±2. 33kPa respectively). Severe hyalinosis, fibrosis, wall hypertrophy, lumen stenosis, and even microaneurysm formation in cerebral small arteries in RHRSP with cerebral infarction were found, while slight to mild arteriolosclerosis in RHRSP was found without cerebral infarction.

本实验发现,RHRSP在经受不同水平的低血压后,产生脑梗塞的大多数(32/38只)在肾动脉狭窄术后第六周至第十四周,血压为28.8±2.24 kPa~32.3±2.88 kPa(216±17mmHg~242±22mmHg),显著高于无脑梗塞的RHRSP,并且发现这些RHRSP的脑内小血管呈严重的透明样变、纤维素样变性,管壁增厚,管腔狭窄,部分有微动脉瘤形成;而无脑梗塞的RHRSP的血压为26.2±2.01kPa~28.3±2.33 kPa(200±15mmHg~212±17.5mmHg),其脑内小血管呈轻至中度的动脉硬化改变。

Results: Compared with IR group , XSOL could significantly decrease the cerebral water content, the cerebral index, the brain vascular permeability in rats of acute incomplete brain ischemia-reperfusion (P.01,P.05). Cerebral tissue morphology showed that neuronic damage of rats subjected to acute incomplete brain ischemia-reperfusion was ameliorated following administration of XSOL (2.4, 9.6g/kg). Compared with IR group , XSOL could significantly decrease the content of MDA and NO in brain tissue(P.01,P.05)and increase SOD,GSH-px activity. The levels of cerebral Glu,Asp were cut down(P.01,P.05). Conclusion: XSOL shows protective effects on cerebral ischemia-reperfusion.

结果:与缺血再灌注组比较,XSOL大、小剂量组可以显著降低急性不完全性脑缺血再灌注模型大鼠的脑含水量(P.01,P.05)、脑指数(P.01,P.05)、脑毛细血管通透性(P.01,P.05);光镜下脑组织形态学显示各剂量组可不同程度的减轻急性不完全性脑缺血再灌注大鼠脑组织的神经元受损形态;与缺血再灌注组比较,XSOL大、小剂量组可以显著降低缺血再灌注大鼠脑组织中的MDA的含量(P.01,P.05),升高SOD、GSH-Px在脑组织中的活性(P.01,P.05);XSOL大、小剂量组可显著降低脑组织中NO的含量(P.01,P.05),降低脑组织中Glu、Asp含量(P.01,P.05)。

Methods 50 patients with diabetes and contrast group were observed, with transcrianial color Doppler flow imaging, cerebral blood stream velocity; pulsative index,resistive index and S/D were calculated of middle cerebral artery, anterior cerebral artery and posterior cerebral artery Results (1)Changes of cerebral blood stream velocity were non-specifitical in blood waves of TCDFI, but VE was reduced.

经临床诊断糖尿病50例,行TCDFI检测大脑中动脉、大脑前动脉、大脑后动脉等血管的血流速度指标及搏动指数、阻力指数,收缩期血流速度/舒张末期血流速度及频谱形态分析与健康组对照比对分析。结果(1)TCDFI频谱中,血流速度的变化,Vmax是非特异性的,而VE是明显下降的。

Methods 50 patients with diabetes and contrast group were observed, with transcrianial color Doppler flow imaging, cerebral blood stream velocity; pulsative index,resistive index and S/D were calculated of middle cerebral artery, anterior cerebral artery and posterior cerebral artery Results (1)Changes of cerebral blood stream velocity were non-specifitical in blood waves of TCDFI, but VE was reduced.

结果 (1)TCDFI频谱中,血流速度的变化,Vmax是非特异性的,而VE是明显下降的。

Methods 50 patients with diabetes and contrast group were observed, with transcrianial color Doppler flow imaging, cerebral blood stream velocity; pulsative index,resistive index and S/D were calculated of middle cerebral artery, anterior cerebral artery and posterior cerebral artery Results (1)Changes of cerebral blood stream velocity were non-specifitical in blood waves of TCDFI, but VE was reduced.

目的 为探讨糖尿病对脑血管的,而对脑动脉血流动力学进行对比。经临床诊断糖尿病50例,行TCDFI检测大脑中动脉、大脑前动脉、大脑后动脉等血管的血流速度指标及搏动指数、阻力指数,收缩期血流速度/舒张末期血流速度及频谱形态分析与健康组对照比对分析。结果(1)TCDFI频谱中,血流速度的变化,Vmax是非特异性的,而VE是明显下降的。

Methods The rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

采用大鼠大脑中动脉缺血2 h/再灌注22 h模型,神经病学评分,脑梗塞范围及脑组织水含量变化,观察脑康灵胶囊抗脑缺血/再灌注损伤的效应;通过测定大鼠脑组织中一氧化氮合酶(nitric oxide synthase,NOS),超氧化物歧化酶(superoxide disumtase,SOD)活性及丙二醛(malondialdehyde,MDA)含量的变化以探讨药物作用的机制。

MethodsThe rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

方法采用大鼠大脑中动脉缺血2 h/再灌注22 h模型,神经病学评分,脑梗塞范围及脑组织水含量变化,观察脑康灵胶囊抗脑缺血/再灌注损伤的效应;通过测定大鼠脑组织中一氧化氮合酶(nitric oxide synthase,NOS),超氧化物歧化酶(superoxide disumtase,SOD)活性及丙二醛(malondialdehyde,MDA)含量的变化以探讨药物作用的机制。

The early intervention on haematoma surround tissue by early input oxygenised liquid during hypertensive cerebral haemorrhage surgery that could effectively relieve cerebral oedema and improve cerebral ischemic hypoxia to save reversibly injured neurons surrounding haematoma,is an effective intervention, which is helpful to the cerebral protection in the patients with hypertensive cerebral haemorrhage.

在高血压脑出血术中早期输入携氧液对血肿脑周围组织提供早期干预,有效减轻脑水肿,改善脑组织缺血缺氧,挽救血肿周围可逆性损伤的神经元是一项有效的措施,有利于高血压脑出血患者的脑保护。

Thus, too cerebral infarction patients, must be the doctor's advice and timely basis of the factors for cerebral thrombosis formal system of treatment, the only way to effectively prevent the recurrence of cerebral infarction, in particular, is suffering from cerebral infarction, in the effective melting suppository within the time (6 ~ 24 hours), timely treatment, thrombolysis, recanalization, since the view that patients return to normal, often because of financial, human and other factors, eager to hospital after reperfusion, non-systematic formal and effective cause of treatment, which led to the repeated recurrence of cerebral infarction, regret.

因此,得过脑梗塞的病人,一定要遵医嘱,及时针对脑血栓形成的基础因素进行正规系统的治疗,只有这样,才能有效地防止脑梗塞的再发,特别是患脑梗塞,在有效溶栓时机内(6~24小时),及时治疗,血栓溶解,血管再通,自认为恢复正常的病人,往往因经济、人力等因素,再通后急于出院,不进行系统正规的、有效的病因治疗,而导致脑梗塞的反复再发,后悔莫及。

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