查询词典 cerebral cortex

In order to investigate the effects of cholecystokinin octapeptide(CCK8) on calmodulin activity in rat cerebral cortex,the cerebral cortex neurocytes was used as a model.

为了探讨胆囊收缩素受体在中枢神经系统中的信号传递机制，观察了CCK8和CCKA受体拮抗剂L-364，718、CCKB受体拮抗剂L-365，260对大鼠大脑皮质钙调素活性的影响。

This disease basically is the backbone branch that kills pear tree, come on earlier bark appearance does not have apparently ill spot, from the place such as leather aperture, cicatricle, cut oozy rust lubricious drip or water having rust is abrupt and oozy, right now only useful knife cuts cerebral cortex, just see cerebral cortex show damask have Brown flyspeck or hematic filiform streak, corrupt skin is loose fill water, have lees taste, embedded and many bacterium.

此病主要为害梨树的骨干枝，发病初期树皮外表无明显病斑，从皮孔。叶痕。伤口等处渗出锈色水珠或有锈水忽然渗出，此时只有用刀削开皮层，才可见皮层呈淡红色并有褐色小斑或血丝状条纹，腐皮松软充水，有酒糟味，内含大量细菌。

In normal group and sham lesion group, BDA positive neurons were distributed symmetrically in the pyramid cells of sensorimotor cortex, and BDA positive fibers of pyramid cells passed through the cerebral cortex, internal capsule, cerebral peduncle of midbrain, basilar portion of pons to the pyramid of medulla oblongata. At the lower part of the medulla oblongata, most of the BDA positive fibers passed through the pyramidal decussation to the contralateral side, continously descended at the ventral part of posterior funiculus just dorsal to the posterior gray commissure and ended at sacral spinal segments.

正常组和假损伤组大鼠BDA示踪显示BDA标记阳性细胞定位于双侧感觉运动皮层的锥体细胞，锥体细胞发出的BDA阳性纤维束对称分布于双侧皮层、内囊、大脑脚、脑桥基底部、延髓锥体中的皮质脊髓束内，继而经锥体交叉到脊髓后索，在双侧后索最深层、灰质后连合背侧下行至脊髓骶段。

Condition. Regarding this, the psychology research gives the explanation is: Happy can cause the cerebral cortex to appear a new excited stove, according to negative induction rule, when tense mental labor causes the cerebral cortex excited region obtained the rest, can play the elimination to be weary,adjustment nerve activity good role.

对此，心理学研究给予的解释是：欢愉会使大脑皮层出现一个新的兴奋灶，根据负诱导规律，紧张脑力劳动时所引起的皮层兴奋区域得到了休息，就会起到消除疲劳，调节神经活动的良好作用。

Results: Neonatal rats in hypoxic-ischemicgroup behaved restlessness, cyanosis, deep and rapid breath, astasia, lethargy, irritation and spasm; There were local pyknosis, fragmentation, dissymmetry, blur or disappearance of nucleoli, raritas of ground substance in left cerebral cortex; but there was no statistic significance in chinese traditional medicine group and sham group. The expression of HIF-1α mRNA in brain tissue of experimental group and chinese traditional medicine group were higher than that in sham group, especially chinese traditional medicine group at 3d; HIF-1α positive cells were found in cerebral cortex and hippocampus. There were more HIF-1α positive cells in experimental group and chinese traditional medicine group and mainly in vascular endothelial cells.

结果 缺氧缺血后大鼠相继表现为烦躁不安、全身发给、呼吸加深加快、站立不稳、嗜睡、激惹或间断发作的痉挛和抽搐；HE染色显示实验组大鼠左侧大脑皮层出现局灶性神经元核固缩，核碎裂，核仁偏位，不清或消失，基质疏松；中药组大鼠脑组织结构基本接近假手术组，无明显水肿和坏死表现；实验组和中药组大鼠脑组织HIF-1α mRNA表达较假手术组明显增加，尤其是中药组实验第3日者；免疫组化显示实验组和中药组各时间点大鼠大脑皮层和海马区均可见不同程度的HIF-1α表达，明显高于假手术组，阳性表达主要在血管内皮细胞，海马和皮层的锥体细胞亦有HIF-1α阳性细胞分布。

Expression of nNOS mRNA, eNOS mRNA and iNOS mRNA in the E 20 cerebral cortex: There were no obvious changes of expression of nNOS mRNA in ETU-injected and control groups. Expression of eNOS mRNA increased significantly in cerebral cortex of the encephalocoele group, And there were no obvious changes of expression of eNOS mRNA in cerebral cortex of the control group and the other ETU-injection groups.

六、不同组E20胎鼠大脑皮质中nNOS mRNA、eNOS mRNA和iNOS mRNA的表达 1、实时定量PCR检测发现对照组、给药无畸形组、脊柱裂组和脑膨出组E20胎鼠大脑皮质中nNOS mRNA表达无明显改变； 2、给药无畸形组、脊柱裂组和对照组胎鼠大脑皮质中eNOSmRNA表达无明显改变，而脑膨出组胎鼠大脑皮质中eNOS mRNA表达明显增多； 3、在各组未检测到iNOSmRNA表达。

Outcome:(1) Acute ischemic damage may induce the expression of c-fos protein in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia. And the expression level of c-fos can be up-regulated by putting blood of 12 hand Jing Points. So we think that treated by this therapy can rise the irritability of brain to resist the development of ischemia and enhance the ability to plerosis.(2) Acute ischemic damage may induce the expression of HspVO in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia.

三 结果(1)急性缺血性脑损伤可快速诱导c-fos蛋白在皮层缺血区脑组织的表达，其表达有随缺血时间延长而逐渐增多的趋势，提示脑的缺血性损伤可应激性地增强神经细胞对缺血、缺氧的耐受和适应能力，而应用井穴放血的干预治疗可明显上调缺血区皮层脑组织c-fos的表达水平，进一步增强脑组织对抗缺血性损伤的应激能力，抵抗缺血性脑损害的进一步发展。

There were no statistically differences between the 8th~ 14thday, the 15th - 21th day post-BC and the control group. During the rt~24tkday post-BC , pycnosis degeneration or necrosis neurons of the locus cerebral cortex, dorsal hippocampus, dentate fornix were significantly increased, then decreased gradually, but recovered to normal by the 24thday after BC, and especially in parietal cortex and piriform cortex the necrosis neurons were significantly increased than temporal cortex , and there were no statistically difference berween the left and the right side. Pycnosis degeneration or necrosis neurons in the brainstem reticular formation were markedly increased in the 4thday after BC, and there were no statistically difference among the other groups and the control group.

与对照组相比，BC后在大脑皮质、背侧海马和齿状回部位，固缩变性和不完全坏死细胞数先显著增加(P＜0.05)，然后逐渐减少，至24d基本恢复正常；顶叶、梨状皮质比颞叶皮质变性坏死细胞多，差异有高度显著性(P＜0.01)；大脑左侧比右侧变性坏死细胞稍少但差异无显著性；在BC后4d，脑桥核、斜方体核平面的脑干网状结构中固缩变性和不完全坏死细胞数明显增多(P＜0.05)，其它组间无显著性差异。

The density of PTEN inthe cerebral cortex, caudate putamen and thalamencephalon was different. The density ofp-Akt in the cerebral cortex, septal nuclei and tuber olfactorium was also different.

4PTEN在大脑皮质、尾壳核和丘脑以及p-Akt在大脑皮质、隔核和嗅结节的表达变化程度是不一致的。

The rats were immediately debrided after injury, then NSCs combined with the chitosan porous scaffold were transplanted into injured cerebral cortex in NSCs+ scaffold group, NSCs combined with the chitosan porous scaffold and NGF were transplanted into injured cerebral cortex in NSCs+ scaffold +NGF group.

Feeney法制备SD大鼠TBI模型，随机分为3组：损伤对照组清创后不做移植；NSCs+支架移植组行壳聚糖作载体的NSCs移植；NSCs+支架+ NGF移植组行壳聚糖作载体的NSCs移植，并在其中加入NGF。

With Death guitarist Schuldiner adopting vocal duties, the band made a major impact on the scene.

随着死亡的吉他手Schuldiner接受主唱的职务，乐队在现实中树立了重要的影响。

But he could still end up breakfasting on Swiss-government issue muesli because all six are accused of nicking around 45 million pounds they should have paid to FIFA.

不过他最后仍有可能沦为瑞士政府&议事餐桌&上的一道早餐，因为这所有六个人都被指控把本应支付给国际足联的大约4500万英镑骗了个精光。