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cell sap相关的网络例句

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与 cell sap 相关的网络例句 [注:此内容来源于网络,仅供参考]

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,"PMN-EC"相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

We will carry on further calculation and expect to get dielectric characters of complex practical cell sap.

计算得到的结果对已有的研究起到很好的补充作用。

Because water is one of the most important molecules for the life and is the main component of cell sap, it is fundamental to research dielectric characters of H_2O.

水是生命中最重要的分子之一,也是细胞溶液的主要组成部分,对它的介电性质的研究有着基础而重要的意义。

The vacuole is filled with cell sap, which contains substances in solution, e.g.

液泡内的液体是细胞液,其内溶解了多种物质,如:糖、盐和有机酸。

It is absorbed by plant roots as the potassium ion, K+, and in plants is the most abundant cation in the cell sap.

以钾离子 K+的形式通过根系吸收,在植物中是细胞液里最丰富的阳离子。

Results: Obvious pathologic changes were observed in pancreas and the apoptotic rate of pancreatic cell in SAP groups were much higher than that in the control group(P.01). The mortality rate was 12.5% in 24h, 90% in a week.

结果:SAP组大鼠胰腺发生明显病理损害,SAP时各时点胰腺细胞存在胰腺细胞凋亡,SAP大鼠1周死亡率为90%。

Active oxygen is found to be involved in apoptotic progress affected by exogenous SOD and measurement of SOD in cell sap. Arsenic compounds cause a decrease of SOD in cell. It is exactly the decrease that make plenty of active oxygen gather in mitochondria, and starts the apoptotic progress. The analysis of SOD values shows that active oxygen has an effect on apoptosis.

通过加入外源性SOD和对胞液SOD的测量,发现胂化物引起细胞凋亡的过程,有活性氧的参与,并且胂化物还导致细胞内SOD值的减少,正是由于这种降低,才导致了细胞内特别是线粒体中活性氧大量聚集,启动了细胞凋亡过程,通过对SOD值的分析,证明了活性氧在细胞凋亡中的作用。

TLR4 participate in the signal transduction of inflammatory cell of SAP. Sandostation can down regulate the expression of TLR4 level, which may influence on the signal transduction of inflammatory cell of the SAP rats.

TLR4影响SAP大鼠肝脏细胞炎症信号转导过程,善宁可能通过影响TLR4的表达,干预SAP大鼠肝脏细胞炎症信号转导过程。

The contact of bacilluscoli with antibacterial steel makes their cell wall and cytoplasm destroyed and their morphology deflate gradually. Accompaning the loss of cell sap. The contact of staphylococcus aureus with antibacterial steel makes their body quickly deflated.

铁素体抗菌不锈钢在与大肠杆菌的作用过程中,能破坏大肠杆菌的细胞壁和细胞膜,使其形体逐渐消瘦,同时伴有组织液流失;在与金黄色葡萄球菌的作用过程中,金黄色葡萄球菌的形体更快地缩小。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,&PMN-EC&相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

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