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cell membrane相关的网络例句

查询词典 cell membrane

与 cell membrane 相关的网络例句 [注:此内容来源于网络,仅供参考]

But in fact the cell wall and membrane grow heterogeneously over the whole cell surface.

这表明mukB基因可能是染色体相对外膜自然移动的发动机。

The immature stage pointed to the development of SE before vacuole membrane was ruptured.In this stage,the cell radial expanding,cell wall incrassation,sieve element plastid and P-protein producing of SE occurred.

未成熟期是指筛管液泡膜破裂之前筛管的发育阶段,在这一时期内出现了细胞的径向扩展、壁的增厚以及筛管质体和P-蛋白质的产生。

RESULTS AND CONCLUSION: After exercise, apoptosis occurs in normal muscle or muscle under pathologic state. The apoptosis of skeletal muscle cells is similar to common cell apoptosis, including karyopyknosis, plasma membrane vacuole, cell organ shrinking, and apoptotic body formation.

结果与结论:运动后,正常肌肉中或是病理状态下的肌肉中骨骼肌细胞都会出现凋亡,凋亡的形态学表现与普通凋亡细胞相似,即核固缩、质膜发泡、细胞器紧缩,凋亡小体形成,其凋亡过程大致可分为3个阶段,即启始阶段、效应阶段和降解阶段。

The electron microscope micrograph demonstrated that E.coli has the internal region 11 cytoplasm which and the nuclein two may distinguish, gathers round a thin cytoplasma membrane outside them and the very thick cell wall, adheres to stick cohere some end dissociation outside the cell wall the flagella.

电镜显微照片显示E.coli有两个可以区别的内部区域一一细胞质和核质,在它们外面围着一层较薄的细胞质膜和很厚的细胞壁,在细胞壁外部附着一些一端游离的鞭毛。

A large, membrane-bound, usually spherical protoplasmic structure within a living cell, containing the cell's hereditary material and controlling its metabolism, growth, and reproduction.

细胞核活细胞之内大的、有膜的、常为球形的细胞质结构,含有细胞遗传物质并控制其新陈代谢、生长和繁殖

Biology A large, membrane-bound, usually spherical protoplasmic structure within a living cell, containing the cell's hereditary material and controlling its metabolism, growth, and reproduction.

细胞核:活细胞之内大的、有膜的、常为球形的细胞质结构,含有细胞遗传物质并控制其新陈代谢、生长和繁殖

IRAK2 protein contained the death domain and S_TKc domain and located at lots of cell organs such as cell nucleus, cytoplasm, mitochondrium and protoplasmic membrane. It is inferred that IRAK2 may be very important for signaling and regulating on TLRs signal transduction.

IRAK2蛋白含有一个DD结构域和一个S_TKc结构域,位于细胞核、细胞质、线粒体和质膜上,通过DD和S_TKc结构域对TLRs的信号转导有着重要的作用。

Observe the formula's effects to the diabetic nephropathy rat model cause by small dose STZ injured, high lipin feed and hemiresection, the results showed that can decrease significantly blood sugar after diet 2h, control availably hyperplasia of glomerulus mesenteric cell, inhibite tumefaction of glomerulus capillary endothelial cell, keep integrality of podocytic process and basement membrane, relieve vasal cavum stricture, reduce the GFR and excretory rate of 〓-MG, improve the status of high blood lipin, specially decrease significantly TC, reduce the 〓 and T/K, improve the status of microcirculation obstacle, elevate the activity of 〓-〓-ATP enzyme.

观察降糖益肾方对小剂量STZ诱导并单侧肾切除,高脂饲料喂养的糖尿病肾病大鼠模型的影响实验结果:对2h后血糖有显著降低作用(p.05),能有效抑制肾小球系膜基质的增生,减轻肾小球毛细血管内皮细胞肿胀,保持足突与基底膜的完整性,减轻血管腔狭窄,降低GFR、尿微量白蛋白以及〓-微球蛋白的排泄率(p.05),能改善高血脂状态,降低血TG、TC含量(p.05),降低〓与T/K比值(p.05),改善微血管循环障碍的状态;能升高肾组织中〓-〓-ATP酶的活性(p.05)。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,&PMN-EC&相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

Studies on mechanism of Biosorption and toxicity of chromium indicated that Biosorption was contained of surface adsorption, transport across cell wall and membrane, and bioaccumulation. Further more, bioaccumulation played significant role in total Biosorption capacity, it attainted 70.6%. And the resistance on chromium of cell wall was perfect, breakage exceeded of 0.1 nm was not conduced by 300 mg/L chromium. Detoxifcation ability would be facilitated by activated sludge.

对吸附机理和毒性的初步探讨结果表明,菌体对铬的吸附由表面吸附、跨壁膜运输和体内积累组成,其中体内积累对吸附的贡献值最大,达到总吸附量的70.6%;细胞壁对铬毒性的抗性理想,300mg/L铬不会对细胞壁产生0.1nm以上的形态破坏;活性污泥的联合使用有助于铬的还原。

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