查询词典 cardiac

Discussion TNF-αis an inflammatory cell cytokine,with the properties of autocrine, produced by cadiocyte at the early age of MIRI,inducing myocardial function abnormal and cadiocyte necrosis in MIRI、pyaemia、chronic cardiac insufficiency、viral myocarditis and the homograft rejection of heart.

讨论 TNF-α是心肌缺血再灌注早期心肌细胞产生的炎性细胞因子，是一种自分泌的作用因子，在缺血再灌注损伤、脓毒症、慢性心功能不全、病毒性心肌炎以及心脏同种移植排斥反应中均能导致心肌功能能障碍和心肌细胞坏死。

Pyridine obviously a huge potential to make money so that related parties cardiac effects.

吡啶显然是潜力巨大的赚钱，让有关各方心脏的影响。

Objective To establish a new procedure for the quantification of cell proliferation on the cultured cardiac fibroblasts.

目的 在培养的心脏成纤维细胞模型上建立检测增殖的方法。

AIM: To investigate the effect of edaravone, a novel free radical scavenger, on cardiac function and myocardial cell injury in rats with septic shock.

目的：观察氧自由基清除剂依达拉奉（edaravone， ED）对脓毒性休克大鼠心脏功能和心肌损伤的作用。

After simple measurement on Doppler spectrum,the Rapp and the maximal cardiac output achieved by controlling heart rate were obtained according to the appropriate ventricular rate theory.

该系列公式为深化MS并AF血流动力学的认识并估算心脏泵功能提供了新的方法，为临床定量化、个体化地控制心室率提供了科学的依据。

(1)The levels of MCP-1 and VEGF'levels was significant increased in CHD patients,Compared with controls, It was implicated that they might take part in the actions of informing atherosderotic plaques;(2)The serum level of MCP-1 and VEGF were elavated step by stey among the group of controls、SAP、UAP、AMI, It was thought they might develop the matter on the stability of atherosderotic plaques;(3)The serum level of MCP-1 and VEGF were positive correlated with 7-TIMI risk score, It prompt that MCP-1 and VEGF was the reactiveness markers of coronary artery process ,and developed the risk to major acute cardiac events.

(1)血清MCP-1、VEGF水平在冠心病患者中显著升高，提示MCP-1、VEGF可能在促进冠状动脉粥样硬化斑块形成的过程中起重要作用；(2)血清MCP-1、VEGF水平在对照组、各冠心病亚组(SAP、UAP、AMI组)之间逐级升高，提示MCP-1、VEGF可能增加了冠状动脉粥样硬化斑块的不稳定性；(3)联合检测MCP-1、VEGF的灵敏度\特异度上升，证明临床上联合检测二者血清指标意义大；并且血清MCP-1、VEGF水平和7-TIMI积分呈正相关性，7-TIMI积分高的患者血清MCP-1、VEGF水平也高，说明它们能作为冠脉病变的活动性标志物，可能成为评价主要心血管恶性事件的危险性的相关指标。

［Objective］To detect the protection mechanism of Na+/H+ permutoid retarder to diabetic mouse cardiac muscle cell oxygen deficit/reaeration on injury.

［目的］探讨Na+／H+交换体阻滞剂对糖尿病鼠心肌细胞缺氧／复氧损伤的保护机制。

Methods］By observing the cytoplasmic Ca2+ concentration change of single cardiocyte in I/R status and the effect of Na+/H+ permutoid retarder to Ca2+ concentration in different phases (diabetes pure oxygen-lack deficit group called DM group,the oxygen deficit/reaeration on entire journey for medicine group called DM-EIPA group, the first group giringmedicine before reaeration on called DM-EIPA 1 group, and the second group giringmedicine before reaeration on called DM-EIPA 2 group) to detect the protection mechanism of Na+/H+ permutoid retarder to diabetic mouse cardiac muscle cell oxygen deficit/reaeration injury.

方法］ 通过观察糖尿病鼠单个心肌细胞在缺氧/复氧时细胞胞浆Ca2+浓度的动态变化以及Na+/H+交换体阻滞剂在不同时相（糖尿病单纯缺氧组即DM组，缺氧/复氧全程给药组即DM-EIPA组，复氧前给药1组即DM-EIPA 1组，复氧前给药2组即DM-EIPA 2组）对Ca2+浓度的影响来研究Na+/H+交换体阻滞剂对糖尿病鼠心肌细胞缺氧/复氧损伤的保护机制。

Furthermore, the limiter not only improved the recording quality but also protected the cardiac electronic equipment because overdue stimulation signal was limited at a given electronic level during the recording of transesophageal pacing.

此外，在经食管心电生理的记录中，过大的刺激脉冲也因实施了限幅技术而被限制在特定的电平内，不仅明显提高了记录质量，而且也保护了心电记录设备。

Conclutions: Under ischemic condition, the decrease of Iks could prolong the mid-late repolarization phase of APD. It could cause EAD and then arouse TdP. Because the decrease of M cell was significantly greater than that of sub-endocardial myocytes and sub-epicardial myocytes, it could increase TDR. Cardiac arrhythmias could last due to reentrance that secondary the increase of TDR.

缺血时，心肌细胞的Iks电流减弱，使得细胞动作电位复极中后期延长，容易出现EAD并引发TdP；由于M细胞的Iks电流减弱程度明显大于心内、外膜细胞，造成心室跨壁复极离散度增加，有利于折返的发生，使得心律失常得以持续。

In the negative and interrogative forms, of course, this is identical to the non-emphatic forms.

。但是，在否定句或疑问句里，这种带有"do"的方法表达的效果却没有什么强调的意思。

Go down on one's knees;kneel down

屈膝跪下。。。下跪祈祷