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cardiac rupture相关的网络例句

查询词典 cardiac rupture

与 cardiac rupture 相关的网络例句 [注:此内容来源于网络,仅供参考]

It was available that the method of pressing and threshing on precardiac region was used in the cardiac sudden death resulting from cardiac dysrhythmia and torpidity cardiac dysrhythmia.

快速性心律失常与缓慢性心律失常引起的心源性猝死用胸前捶击及胸外按压方法抢救是有效的。

The input parameters for cardiovascular model are heart and vascular network parameters such as cardiac cycle, heart elastance, vascular compliance and vascular resistance, while the outputs are the blood pressure and flow wave on each segment of vascular, and the cardiovascular parameters such as stroke volume, cardiac output, eject fraction, energy and oxygen consuming in each cardiac cycle, and so on.

模型的输入是心脏和血管网络的参数如:心动周期、心肌弹性度、血管顺应性、血管阻力等,模型的输出是各血管段的压力、流量波以及反应心血管系统功能状况的生理参数如每搏输出量、心输出量、射血分数、每搏耗能和耗氧量等。

Background— The hallmarks of diabetic cardiomyopathy are cardiac oxidative stress, intramyocardial inflammation, cardiac fibrosis, and cardiac apoptosis.

背景——糖尿病性心肌病的标志是心脏氧化应激、心肌内炎症、心脏纤维化、心脏凋亡。

In contrast, exposure to ectopically expressed noncanonical Wnt11 markedly decreased the expression of Oct-4 and Nanog and induced mRNA expression (quantitative real-time reverse-transcription polymerase chain reaction) of cardiac-specific genes (Nkx2.5, GATA-4, atrial natriuretic peptide,- and -myosin heavy chain, and cardiac troponin T) by day 3 with subsequent progression to a pattern characteristic of the cardiac fetal gene program.

对比之下,接触到非典型wnt11能明显降低Oct-4 和 Nanog表达和在第3天出现诱导心脏特异性基因(Nkx2.5,GATA - 4,心房利钠肽,- and -肌球蛋白重链,肌钙蛋白T)mRNA的表达(应用反转录聚合酶链式反应实时定量),随后逐渐表达为心脏胎儿基因。

Methods: a comprehensive analysis of 20 cases with acute left ventricular failure as the first manifestations of acute subendocardial myocardial infarction patients with a history of 20 cases and clinical characteristics, according to ECG, dynamic changes of enzymes, Killip classification of cardiac function and cardiac color report indicators of cardiac function, acute acute subendocardial myocardial infarction with acute left heart failure occurred in the characteristics and causes.

综合分析20例以急性左心衰竭为首发表现的急性心内膜下心肌梗死20例病人的病史和临床特点,根据心电图、心肌酶的动态变化、 Killip 心功能分级和心脏彩超报告的心功能指标,探讨急性急性心内膜下心肌梗死发生急性左心衰竭的特征和原因。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

Background— Protein kinase A signaling has long been known to play an important role in cardiac function. Dysregulation of the protein kinase A system, caused by mutation of the protein kinase A regulatory subunit gene PRKAR1A, causes the inherited tumor syndrome Carney complex, which includes cardiac myxomas as one of its cardinal features. Mouse models of this genetic defect have been unsatisfactory because homozygote null animals die early in development and heterozygotes do not exhibit a cardiac phenotype.

背景——蛋白激酶A信号传导在心功能中发挥了重要作用早已为人们所知蛋白激酶A调节亚基因PRKAR1A的变化引起蛋白激酶A系统失调,导致了遗传性卡尼复合肿瘤综合症,其中包括的心脏粘液瘤作为它的一个主要特征此种遗传缺陷的小鼠模式仍然不能令人满意,因其纯合体空白动物在发展中早期死亡,而杂合体不能显示心脏表型

Methods Retrospective review of 71 patients with cardiac injury was carried out including 21 blunt wounds,40 penetrating wounds and 10 iatrogenic wounds.Conservative therapy was performed on 8 myocardial contusions,2 hemopericardiums,2 small metal foreign bodies retention in myocardium and also 2 cardiac tamponades with iatrogenic left atrium perforation.The acute or delayed thoracotomies for repairing cardiac lesion according to the injury condition were performed in remainders.

对21例钝性心脏伤、40例穿透伤和10例医源性损伤的临床资料进行了回顾性分析;全组除心肌挫伤8例,血心包、心脏细小金属异物和医源性左心房穿透伤导致急性心包填塞各2例行保守治疗外,其余病例均根据心脏损伤情况,分别进行了急症或延迟性开胸手术,修复心脏损伤。

OBJECTIVE: To investigate the effect of mitogen activated protein kinase on the role of cardiac hypertrophy induced by Ang Ⅱ in cardiac myocytes so as to provide theoretical basis for clinical prevention and cure of cardiac hypertrophy.

目的:探讨丝裂素活化蛋白激酶在AngⅡ致心肌细胞肥大的作用,为心肌肥大的防治提供理论依据。

Cardiac toxicity was minimal in both groups, the editorial notes, adding that this is a major adverse effect of trastuzumab. However, the trialists point out that there was a bias, as they had selected women who had normal cardiac function after having received various other therapies (including anthracyclines and taxanes) as well as trastuzumab. Also, since the duration of observation was limited, the possibility of late events cannot be excluded."Nevertheless, the low incidence of adverse cardiac events is reassuring," they comment.

主编表示,两组之间发生心脏毒性的比例相似,且这是trastuzumab主要的副作用;然而,研究者指出其中有误差,因为他们挑选已经接受其它不同治疗(包括anthracyclines类药物与taxanes类药物)以及trastuzumab后心脏功能正常的病患;同时,因为试验观察时间长度有限,晚期发生不良反应的可能性并不能排除;他们的评论是,不管如何,心脏不良反应发生率低是可靠的。

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