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camp-out相关的网络例句

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与 camp-out 相关的网络例句 [注:此内容来源于网络,仅供参考]

Objective:To study Effects of dehydrocorydaline on cAMP and cGMP concentration in rabbit platelets in vitro.

目的:研究脱氢紫堇碱对兔血小板中cAMP 和cGMP含量的影响以探讨其抑制血小板聚集的作用机制。

RESULTS: The relative levels of COX2 mRNA as well as the contents of PGE2 and cAMP were elevated after fracture, and were significantly higher in study group than those in control group at 3 and 4 weeks postoperatively. Histological observation revealed that the mineralization of cartilage matrix, the endochondral bone formation and the callus remodelling took place earlier in study group.

结果:环氧化酶2(COX2) mRNA的表达量以及PGE2和cAMP的含量均较骨折前显著上升,且实验组在术后3~4 wk时以上指标明显高于对照组,组织学结果则显示实验组骨折间隙内软骨内成骨和编织骨向板层骨的改建均领先于对照组。

Neurons treated with 20-mmol/L-theophylline did not show 180bp ladder characteristic of endonuclear activation on its DNA gel electrophoresis. The [cAMP]i of the neurons rose after being incubated together with different concentration theophylline (5-80 mmol/L) for 15 minutes in CO2 incubator and the prometed range relied on the dose of theophylline.

100%,量效关系有高度显著性(P.01),EC50=2.97mmol/L;经茶碱(20mmol/L)处理的细胞,其DNA凝胶电泳带没有&梯子&状分布;与不同浓度茶碱(5-80 mmol/L)在CO2培养箱孵育15 min,小脑颗粒神经元内cAMP均升高,且是剂量依赖性的。

E. , ACTH antagonizes the analgesia mediated byμand δ opioid receptors, but notκreceptor;(2) The antagonizing effect of ACTH on opioid analgesia is proposed to be mediated by ACTH receptors, although the latter has not been characterized;(3) A contradictory interaction on intracellular cAMP content may constitute one of the postreceptor mechanisms underlying ACTH antagonism of opioid analgesia;(4) Another proposed mechanism of the anti-opioid effect of ACTH is that ACTH can modulate opioid-induced suppression of calcium influx;(5) ACTH has been shown to induce Fos protein expression in selected areas of the rat brain including the nuclei involved in pain regulation as well as the ependyma of the third ventricle and the aqueduct.

根据以上实验结果,本论文首次提出以下论点:(1)ACTH在脊髓水平对抗阿片镇痛具有受体选择性,即ACTH可对抗μ受体和δ受体介导的镇痛,不对抗κ受体所介导的镇痛;(2)由于ACTH与阿片μ受体的肽类配体的结合位点仅有很低的亲和力,与μ受体的非肽类配体的结合位点以及δ受体无亲和力,推测ACTH是通过中枢内的ACTH受体介导发挥抗阿片效应的;(3)ACTH抗阿片作用的受体后作用机制之一是在cAMP信使通路水平与阿片发生相互作用;(4)ACTH抗阿片作用的另一受体后机制是在〓水平影响阿片的效应;(5)通过Fos蛋白的诱导揭示:ACTH可以作用于脑内多个核团,其中包括许多与痛觉调制有关的核团,推测ACTH可能通过激活这些核团的神经元而发挥其中枢效应。

Result:There were IR and DCI existed in the patients which appeared as the elevation of the absolute value of ISI,〔Ca2+〕t and CE-cAMP and decrease of the activity of the ion pump in the erythrocytic membrane. 15 of the 31 patients had hyperinsulinemia. The indexes mentioned above were almost the same in CHDs with hypertension and those without hypertension.

结果:CHD患者存在IR和钙离子代谢障碍,表现为ISI绝对值、红细胞总钙浓度、cAMP浓度升高和红细胞膜离子泵活性下降;31例CHD中15例(48%)有高胰岛素血症;上述指标在有、无并发高血压者间相比无显著性差异。

All of these three factors could decreased the cAMP level of cultured granulosa cells.

EGF、胰岛素和孕酮均可使培养颗粒细胞内的cAMP水平下降。

There was no significant difference in cAMP production of these two kinds of thyroid cells,suggesting that both the hyperfunctioning and normal thyroid cells react to TSH after the same fashion ...

在相同实验条件下,两种细胞释放cAMP的值无显著性差异。提示甲亢甲状腺细胞与正常甲状腺细胞相似,对TSH具有良好的反应性。

The effect of tumor necrosis factor on human normal and hyperfunctioning thyroid cells were studied as expressed by cAMP release from cultured thyrocytes.

采用细胞单层培养方法,以cAMP为观察指标,研究肿瘤坏死因子对正常和甲亢甲状腺细胞功能调控的特征。

2TNF-α in the concentration of 10-3-10u/ml significantly inhibited TSH-induced cAMP release by both normal and hyperfunctioning thyroid cells.

2在10~(-3)~10U/ml的浓度区间,TNF-α显著抑制TSH对正常和甲亢甲状腺细胞生成cAMP的刺激效应,10~3U/ml的TNF-α仅抑制TSH对正常甲状腺细胞的刺激作用,对甲亢甲状腺细胞无明显影响。

ResultsCompared with those in the control, urine volume was decreased markedly, plasma AVP and cAMP levels in renal medullar tissue were decreased obviously, and AQP2 expression was decreased significantly in model group.

结果与对照组相比,模型组大鼠的尿量明显增加,血AVP和肾髓质cAMP水平下降,集合管上皮细胞AQP2表达减少。

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