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camp-fire相关的网络例句

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与 camp-fire 相关的网络例句 [注:此内容来源于网络,仅供参考]

Yet scheduled university is one of the founders of ngos camp seven camp is scheduled to express a youth organization strength feeling 4 activities of the organization, such as "quickly" activities, the public concert busk, etc.

2009.03-至今大学城NGO crazy camp 七个创始人之一 crazy camp是一个为了抒发青春策划组织劲情四射活动的组织,例如"快闪"活动,大众演唱会,街头卖艺等等。

Selenium chitosan could reduce cGMP level, promote Ca(superscript 2+) and cAMP level and cAMP/cGMP ratio, which are all possible mechanisms that selenium chitosan induces NB4 cell apoptosis and inhibite proliferation.

硒化壳聚糖可通过升高细胞钙离子和cAMP含量,降低cGMP含量,增大cAMP/cGMP比值来抑制NB4细胞生长,诱导其凋亡。

The results suggested that AC-cAMP-PDE pathway in cerebellum did not participate in regulating the anesthetic action, but perhaps correlated with ethological changes (such as behavior mis match and orientating function handicapped) during recovery period.

4临床相关剂量的噻拉唑和QFM合剂对大鼠大脑皮质、海马、脑干和小脑的PDE活性无影响,结果说明,大脑皮质、海马、脑干和小脑AC-cAMP-PDE信息传递系统对噻拉唑和QFM合剂麻醉作用的调控不是通过PDE对cAMP的降解作用,而是通过其它途径(如通过调节AC的活性影响cAMP的生成环节)来调控cAMP的产量实现的。

METHODS: 8-Bromo-cAMP and sodium nitroprusside were added into the media to raise the concentrations of cAMP and cGMP of cultured cardiomyocytes, in situ hybridization and immunohistochemistry methods were used to identify the mRNA and protein expression of bFGF. The amount of bFGF mRNA and protein expression were detected by computor imaging analysis system MIAS exe.

分离培养大鼠心肌细胞,加入8-溴-cAMP和硝普钠,升高细胞内cAMP、cGMP浓度,用原位杂交及免疫细胞化学的方法观察细胞内bFGF的表达,图象分析检测bFGF表达量的变化。

Our results indicated that (1)SO_2 derivatives could dose-dependently relax the isolated aorta rings with or without endothelium precontracted by NE, no difference was found for the rings with and without endothelium;(2)Levels of cAMP and 6-Keto(PGI_2) in isolated aortic rings were increased by SO_2 derivatives in a dose-dependent way, which were independent of endothelium;(3)SO_2 derivatives decreased cGMP levels in the rings, but not significantly; The decrease of cGMP levels was also independent of endothelium;(4)No changes of TXB_2 in rings with or without endothelium were observed;(5)Whether presence or absence of endothelium, cAMP/cGMP and 6-Keto/TXB_2 ratios were increased significantly.

结果:①SO_2衍生物对去甲肾上腺素预收缩的内皮完整和去除内皮的大鼠胸主动脉血管环均呈浓度依赖性舒张作用,且其张力变化在内皮完整与去内皮的血管环之间无显著性差异;②SO_2衍生物可引起血管组织中cAMP、6-Keto(即PGI_2)含量剂量依赖性增加,其增加程度与内皮无关;③SO_2衍生物对血管环cGMP含量呈降低趋势但在统计学上无显著意义,且对去除内皮血管无显著影响;④SO_2衍生物对去内皮或内皮完整血管环的TXB_2含量均无明显影响;⑤无论是否去除内皮,SO_2衍生物均可引起cAMP/cGMP和6-Keto/TXB_2比值显著升高。

The Change of temperature before and after the modal-making and administration, the content of plasma cAMP, quotient of cAMP and cGMP and the peripheral blood picture was examined to study the fever-preducing function of Dihuang on RFEHYDS.

通过测定造模、给药前后体温、血浆cAMP含量、cAMP/cGMP比值以及外周血象的变化,研究地黄对家兔阴虚热盛证型发热的解热作用。

RESULTS:The levels of PGE and cAMP in fever rabbits' cerebrospinal fluid was rised abnormally(P.01), AFMI can lower the abnormally rised levels of PGE and cAMP in the cerebrospinal fluid(P.01), and the use of AFMI ahead of schedule can inhibit the synthesis and release of PGE and cAMP in the cerebrospinal fluid.

结果:发热家兔脑脊液PGE、cAMP含量异常升高(P.01),AFMI可显著降低发热家兔脑脊液中异常升高的PGE和cAMP的含量(P.01),最佳剂量为26.25 g*kg-1,提前应用AFMI,可阻抑感染家兔脑脊液中PGE、cAMP的合成释放。

Bitter compounds in saliva activate particular T2R/TRB isoforms activate gustducin heterotimers, activated α-gustducin stimulates PDE to hydrolyze cAMP, the decreased cAMP may disinhibit cyclic nucleotide-inhibited channels to elevate intracellular Ca2+.

唾液中的苦味化合物激活T2R/TRB同型体,这种激活导致味导素异聚体的激活,被激活的α-味导素又激活PDE,PDE水解cAMP,cAMP水平的降低使环核苷酸抑制的阳离子通道去抑制,从而使细胞内Ca2+升高。

Salty and sour directly transduction by apical channels that are non-gated channel or chemically-gated channel. Bitter compounds in saliva activate particular T2R/TRB isoforms which activate gustducin heterotimers, activated α-gustducin stimulates PDE to hydrolyze cAMP, the decreased cAMP may disinhibit cyclic nucleotide-inhibited channels to elevate intracellular Ca2+.

咸和酸直接通过顶部非门控或化学门控离子通道转导唾液中的苦味化合物激活T2R/TRB同型体,激活导致味导素异聚体的激活,被激活的α-味导素又激活PDE,PDE水解cAMP,cAMP水平的降低使环核苷酸抑制的阳离子通道去抑制,从而使细胞内Ca2+升高。

Bitter compounds in saliva activate particular T2R/TRB isoforms which activate gustducin heterotimers, activated α-gustducin stimulates PDE to hydrolyze cAMP, the decreased cAMP may disinhibit cyclic nucleotide-inhibited channels to elevate intracellular Ca2+.

唾液中的苦味化合物激活T2R/TRB同型体,这种激活导致味导素异聚体的激活,被激活的α-味导素又激活PDE,PDE水解cAMP,cAMP水平的降低使环核苷酸抑制的阳离子通道去抑制,从而使细胞内Ca2+升高。

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