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caliciform cells相关的网络例句

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The hematopoietic system was reconstituted and the chimaeras were formed.The results showed that hematopoietic stem cells coming from ES-D3 cells could reconstitute the mice following high dose chemotherapy in vivo. This research offers theories for HSCs induced from embryonic stem cells supporting the high dose chemotherapy of cancer.

将鉴定的造血干细胞移植入大剂量化疗的C57BL/6鼠,观察造血重建功能并证明有嵌合体形成,从体内实验方面证实了ES-D3细胞来源的造血干/祖细胞移植能够重建接受大剂量化疗小鼠的造血功能,为胚胎干细胞来源的造血干细胞支持大剂量化疗提供了实验依据。

The Piwi clade is found in all animals examined so far, and its presence is tightly correlated with the emergence of specialized germ cells. Most animals separate germline and somatic cells early in development and restrict Piwi expression specifically to germ cells.

Piwi蛋白迄今为止被发现存在于所有动物中,它的存在与专门的生殖细胞紧密相关,大多数动物在发育的早期开始了不同组织和体细胞的分离和分化,并且对于生殖细胞的Piwi蛋白的表达进行特别的限制。

Methods:We cultured PBMC and CD4+ T cells with or without FBM-MSC,and we named the coculture of PBMC or CD4+ T cells with FBM-MSC as coculture cells.

我们在存在或不存在FBM-MSC的情况下培养PBMC和CD4+T细胞,PBMC或CD4+T细胞与FBM-MSC的共培养被称为共培养细胞。

Results Only DCs cocultured with apoptotic cells can induce special CTLs, while the group of U937 cells and culture cells can not.

结果 与凋亡胶质瘤细胞共培养之DCs可以有效提呈胶质瘤细胞抗原,有强烈的免疫应答,刺激的细胞毒T淋巴细胞特异性杀伤胶质瘤细胞。

It is further confirmed by the experiment of muti-inducement that MSMPs can differentiate into osteocyte sand fibrocartilage cytes and it is osteo-chondro progenitor cells with the potential ability to evolve muti-differentiated. This added evidence to the view that condylar cartilage differentiated from osteo-chondro progenitor cells in the mesenchymal cells of mancibular periosteal.

体外分离培养鼠胚下颌体骨膜间质细胞并研究其生物学特性,通过多向诱导实验进一步证实:下颌骨膜间质细胞可能为骨—软骨的前体细胞,可分化为成骨细胞及纤维软骨细胞,具有多向分化潜能,此结果进一步支持髁突软骨是由下颌骨膜间质中骨—软骨前体细胞发育而来。

Results A middle age woman had a history of leiomyoma of uterus incision and CT scan shows several nodes in lung. Two gray-whitish nodes were found in the removal specimen, 6 cm、12 cm in respectively diameter, and well demarked from the around tissue. The neoplasia is plenty of tumor cells, some lumenoid structure lined with epithelium in the tumor tissue. Tumor cells is well differentiated and look like smooth muscle cells without the necrosis and karyo-kinesis. The pattern of tumor is similar to the original tumor.

结果 中年女性患者,有子宫平滑肌瘤手术史,胸部CT示右肺多发性结节切除标本见2个灰白色结节,直径分别为6 cm、12 cm,边界清楚,镜下见肿瘤细胞较丰富,并见上皮衬覆的管腔样结构,未见坏死及核分裂象,与原发子宫平滑肌瘤形态相似免疫组化证实为平滑肌源性,且表达雌激素受体。

Desmosomal junctions were seen between cells. Conclusion The culture conditions used in the experiment are suitable for the primary culture of human laryngeal epithelial cells. The cultured cells will facilitate further research on etiology and pathogenesis of larynx carcinoma because of their proliferative and differentiative potency.

本实验采用的培养条件适宜人喉上皮细胞的生长,所获细胞纯度高,培养过程简便;从生物学行为来看,体外培养的人喉上皮细胞同样具有增殖能力和分化潜能,是进行癌变研究的良好对象。

The digestions should be less than 6 times, and every digestion should last for less than 20 min. Two-hour differential adherence could improve the purity of myocardial cells. By this protocol, 94.6% of cultured cells were live myocardial cells.

分离细胞时消化次数最好控制在不多于6次,每次消化时间不超过20min.2h差速贴壁可以提高心肌细胞纯度,经肌动蛋白鉴定,心肌细胞达94.6%。

RESULTS The results indicated that PCF could significantly increase the viability of thymocytes and enhance the activities of SOD, GSH-Px, CAT in cells. PCF decreased the amounts of ROS and the rate of apoptosis in cells. PCF inhibited the morpholog alteration of the thymocytes irradiated by UVB and maintained the ultrastructure of cells in normal. PCF attenuated UVB caused DNA fragmentation in thymocytes. While combined ERKs inhibitor yielded typical DNA fragmentation, co-treatment of thymocytes with PCF and JNKs and p38 kinase inhibitors completely blocked UVB-induced DNA fragmentation.

结果 PCF能显著提高UVB辐射后小鼠胸腺淋巴细胞的活性;提高胞浆中SOD、GSH-px、CAT的活性,降低ROS含量及细胞凋亡率;抑制UVB辐射所致的胸腺淋巴细胞凋亡的形态学改变,维持细胞的正常超微结构;抑制UVB所致的胸腺淋巴细胞DNA片段的出现;预先应用ERK抑制剂使DNA片段增加,给予JNK和P38抑制剂后协同PCF使DNA片段带减少。

Results By incubating cardiomyocytes with bFGF, ERKs was activated and the cellular viability 〔(70.0±4.6)%in bFGF 10 ng group vs.(53.0±4.5)%in H/R group, P<0.01) and the ATP content 〔(23.1±2.3) nmol/106 cells in bFGF 10 ng group vs.(12.3±2.1) nmol/106 cells in H/R group, P<0.01〕 were increased, also the LDH leakage 〔(257.3±51.0) U/L in bFGF 10 ng group vs.(372.5±69.2) U/L in H/R group, P<0.05〕, was decreased, then attenuation of H/R injury to cardiomyocytes was observed. PD098059, an inhibitor of upstream kinase of ERKs completely abolished the protective effects 〔viability (57.0±5.8)%,ATP content (15.1±2.6) nmol/106 cells,LDH activity in medium (325.5±59.0)U/L in PD098059 group vs. bFGF 10 ng group, P<0.01, respectively〕.

结果 bFGF激活ERKs,并呈剂量依赖性减轻H/R所致心肌细胞损伤,表现为细胞存活率〔bFGF 10 ng组(70.0±4.6)%,H/R组(53.0±4.5)%,P<0.01〕和细胞内ATP含量〔bFGF 10 ng组(23.1±2.3)nmol/106细胞,H/R组(12.3±2.1)nmol/106细胞,P<0.01〕升高,细胞浆酶LDH漏出〔bFGF 10 ng组(257.3±51.0)U/L,H/R组(372.5±69.2)U/L,P<0.05〕减少;ERKs上游激酶抑制剂PD098059完全消除上述保护作用〔PD098059组细胞存活率(57.0±5.8)%,ATP含量(15.1±2.6)nmol/106细胞,培养液中LDH活性(325.5±59.0)U/L,与bFGF 10 ng组比较均为P<0.01〕。

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