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bronchiolar相关的网络例句

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与 bronchiolar 相关的网络例句 [注:此内容来源于网络,仅供参考]

Dyspnea in the presence of bronchiolar edema is associated with wheezing due to airflow obstruction; frothy and sometimes blood-tinged sputum is expectorated.

细支气管水肿性呼吸困难与气流梗阻引起的喘息关,可咳出泡沫痰,有时则咳带血痰。

PS is mostly synthesized and secreted by alveolar typeⅡ cells and also by Clara cells in the peripheral airways to a lesser extent, and approximately 7% of alveolar surfactant reaches the bronchiolar tree via the mucociliary escalar.

PS主要由肺泡Ⅱ型上皮细胞产生,少部分由外周气道的Clara细胞产生,约7%的肺泡表面活性物质经纤毛运送到达细支气管。

Examination of the lungs of young smokers has revealed an accumulation of macrophages in the bronchiolar region and bronchiolitis.1 2 Phagocytosis of cigarette smoke-derived particles is obviously an important defense mechanism with which to neutralize and clear the toxic particulate fraction.

对年轻吸烟患者进行体检发现巨噬细胞在细支气管区积聚并产生细支气管炎症。对吸烟产生的微粒的进行吞噬作用显然是一项重要的防御机制,使有毒颗粒部分失效后并清除出去。

Results: we estalished ideal copd models in stort time. there existed the expression of nos in both the normal lung tissue and the lung tissue of copd,mainly orienrared in the cytoplasm of cell.the immunohistochemical result indicated that inos increased significantly ,in comparison with the control group, in bronchiolar mycoderma epithelial and enos remakably decreased in pulmonary blood vessel.

结果:联合应用烟熏和ppe在较短的时间内建立了较理想的copd模型;一氧化氮合酶在正常肺组织与copd的肺组织均有表达,主要定位在细胞浆内;免疫组化结果表明模型组inos在支气管黏膜上皮呈强阳性表达,肺血管enos表达明显减弱,与健康对照组比较有显著性(p.01)。

Results The number of cellular apoptosis was obviously increased in multiple organs from the six patients died from SARS. The cellular apoptosis occurred predominantly in cytokeratin-positive pneumoncytes, terminal bronchiolar epithelium, CD3 + and CD8 + lymphocytes, as well as a part of CD20 + lymphocytes and CD68 + macrophages.

结果 与正常组织比较,SARS患者全身多器官组织细胞凋亡数目明显增多,尤以肺脏和免疫器官为甚,主要的凋亡细胞类型为CK阳性肺泡上皮细胞、终末支气管黏膜上皮细胞、CD3+细胞、CD8+细胞,以及少部分CD2 0 +细胞和CD6 8+巨噬细胞。

In lungs, AQP1 was abundant in peribronchiolar capillaries, postcapillary venules, bronchiolar basal membrane and alveolar epithelial cells.

结果表明,托吡酯在给药剂量范围内(0.068μg-6.8μg/鸡胚)可抑制鸡胚尿囊膜的血管生成,剂量为0.68μg/鸡胚和6.8μg/鸡胚时抑制效果明显,最高抑制率达33.5%。

Dy ea in the presence of bronchiolar edema is a ociated with wheezing due to airflow o tructio frothy and sometimes blood-tinged utum is expectorated.

细支气管水肿性呼吸困难与气流梗阻引起的喘息关,可咳出泡沫痰,有时则咳带血痰。

Jq Ez6_{0An outbreak of highly pathogenic avian influenza caused by H5N1 virus occurred in Vietnam, Japan and Korea, commencing in December, 2003. By February, 2004, it has been distributed widely in Asia and brought heavy losses. H5N1 are ball-like RNA viruses, birds can shed them via respiratory system, ocular secretions and feces, sometimes transferred to human and cause lethal infection. H5N1 patients have prominent features of reactive hemophagocytic syndrome,bronchiolar inflammation, necrosis of bronchial epithelium and organizing,diffuse alveolar damage with interstitial fibrosis.

2003年12月,H5N1型禽流感病毒在越南、日本和韩国爆发,到2004年2月,该病毒已在亚洲广泛蔓延并带来了巨大损失。H5N1是球型RNA病毒,禽类间可通过呼吸系统、眼分泌物和粪便传播,也可传染人类甚至导致死亡。H5N1感染者的特征是反应性噬血细胞综合征、细支气管炎症、支气管上皮坏死和机化、弥漫性肺泡病变和间质纤维化。

Pleural calcification was missed by chest film but found in 4 by CT. Pleural effusion was noted in 3 by chest film and 4 by CT. HRCT is superior to conventional chest film in demonstrating the changes in the seconday pulmonary lobule such as focal emphysema, interlobular septa thickness and bronchiolar wall thickness.

胸部x光显示肋膜增厚者10例,CT18例,肋膜钙化者无,CT4例,肋膜积水者3例,CT4例,高解像力电脑断层检查更可显示次级肺小叶內的变化如局部气肿,小叶问隔增厚,小支气管壁增厚,这些均优於传统胸部x光片。

At 4th week, the percentages of Clara cells over bronchiolar epithelial cells were similar between groups. At 24th week, the percentages of Clara cells were reduced in mice exposed to TCDD, NNK, CSC, or TCDD+CSC exposure, but not in mice exposed to TCDD+1 mg NNK. Thus, these findings suggested that TCDD, NNK, CSC, or TCDD+CSC exposure injuried Clara cells to lose CCSP protein in mice lung.

在A/J mice 4周实验组中,TCDD或NNK各实验组Clara cell的数目(26.6%-34.3%)与控制组(34.2%)比较,并没有明显的差异;反观A/J mice 24周实验组中,除了TCDD+NNK (1 mg)实验组外,其他各实验组Clara cell的数目与控制组(55.9%)比较明显下降11.7%-41.4%,显示暴露NNK、TCDD、CSC或TCDD+CSC皆会伤害Clara cell,暗示Clara cell对这些环境毒物比其他呼吸道上皮细胞敏感。

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这两个团体间的分歧难以掩饰。

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