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breast-fed相关的网络例句

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与 breast-fed 相关的网络例句 [注:此内容来源于网络,仅供参考]

The risk factors of breast carcinoma includes family history (The likelihood of developing breast cancer for a patient, if one of whose mother or sisters does, will increase by 1.5 to 3 times.), age, BMI, hereditary factors (The risk of developing breast cancer for a female whose BRCA1 gene has been mutated reaches 50-85% in her life.), female, contact history of the endogenous and exogenous estrogen, past history of chest being exposed to the radiation, dietary pattern, benign mastopathy and environmental factors.

发生乳腺肿瘤的危险因素包括:家族史(假如患者母亲或姐妹当中有一人发生乳腺癌,该患者乳腺癌发生的可能性增加1.5至3倍),年龄,肥胖,遗传性素因(BRCA1基因突变的女性一生中发生乳腺癌的可能性为50-85%),女性性别,内源性和外源性雌性激素接触,既往有过胸部放射性暴露史,膳食结构,良性乳腺疾病和环境因素。

The dietary intake was recorded for 3 consecutive days during the 4th and 6th month of the infants,and the infants were divided into 4 groups with di fferent feeding patterns,namely exclusive breast feeding,breast feeding as the only milk source with other supple- mentary food,mixed feeding(breast and formula feeding plus other supplementary food),and bottle feeding.

记录4、6月龄连续3d的食物摄入情况,按4月龄摄入食物种类分为完全母乳喂养(除母乳、水、鱼肝油外不吃其他含热能食物)、不完全母乳喂养(母乳为唯一乳类,同时摄入非乳类辅食)、混合喂养(母乳、配方乳及其他辅食均有摄入)和人工喂养(以婴儿配方乳为唯一乳品,同时也摄入其他辅食)4种喂养方式。

Objective To study the expression of epithelial-cadherin in breast cancer cells and the microvessel density in breast cancer mesenchyma, and to analyze the correlation between two factors and metastasis in breast cancer.

目的:研究乳腺癌细胞上表皮细胞黏附分子的表达和癌间质的微血管密度,并分析E-cd和MVD与乳腺癌转移的关系。

Major pathological changes are duct ectasia and obvious periductal inflammatory changes.The clinical manifestations of PCM are breast mass and inflammatory changes,which should to be distinguished with advanced breast cancer and inflammatory breast cancer.

结果:乳腺导管扩张症临床表现为乳头溢液和乳腺肿物,主要与乳腺肿物和早期乳腺癌鉴别诊断,手术以局部切除多见,病理表现为导管扩张及导管周围明显炎性改变。

Methods:The changes of serum CYFRA21-1 level were detected by RIA method in 34 patients with breast benign diseases,47 patients with protopathic breast cancer before and after their operations and 56 patients with breast cancer only after operations.

采用放免法测定 34例乳腺良性疾病患者术前、47例乳腺癌患者术前和术后 4周及 56例乳腺癌术后随访患者的血清 CYFRA21-1 水平,进行对照分析。

Methods:The changes of serum CYFRA21-1 level were detected by RIA method in 34 patients with breast benign diseasesˇ47 patients with protopathic breast cancer before and after their operations and 56 patients with breast cancer only after operations.

方法ˇ采用放免法测定 34例乳ˇ良性疾病患者术前、47例乳ˇ癌患者术前和术后 4周及 56例乳ˇ癌术后随访患者的血清 CYFRA21-1 水平ˇ进行对照分析。

All the LOH on 〓 and 〓 was observed in invasive ductal carcino- ma, carcinoma simplex, medulary carcinoma and scirrhous carcino- ma, no deletions at these sites were observed in any invasive lobular carcinoma and others. These results imply an etiological difference.P53 gene is a hot point gene in the occurrence and development of breast, cancer. PCR-SSCP analysis was performed to detect P53 gene point mutation in the region between exon 5 and 8, 5 of 12 (41. 6%) stage I breast cancer patients contain mutation of P53, 3 of 5 patients were accompanied by 〓 deletion. These results suggested that point mutation and allelic loss of P53 gene are two vi- tal genetic events in earlier stage of breast tumorigenesis.

我们还发现,〓和〓位点LOH均分布在乳腺浸润性导管癌、单纯癌、髓样癌及硬癌中,在浸润性小叶癌和某些特殊类型乳腺癌中全部为LOH阴性,上述位点LOH可能与某些组织学类型乳腺癌的发生有关。P53基因是乳腺癌形成过程中的一个热点基因,本研究对12例Ⅰ期乳腺癌组织标本中P53基因热点区域第5、6、7、8外显子点突变进行了测定,发现41.6%(5/12)的病人有P53基因一个或多个点突变,其中3例同时伴有〓位点LOH,表明P53基因点突变和等位基因缺失是发生在乳腺癌形成早期的一个重要遗传学事件。

CHICAGO - Automated breast ultrasound can detect new breast lesions as well as confirm findings preiously found on mammography, according to the phase II results of a screening trial at the Elizabeth Wende Breast Clinic in Rochester, NY.

芝加哥——根据纽约Rochester,Elizabeth Wende乳腺门诊的筛查性研究的二期临床测试结果,全自动乳腺超声可以发现乳腺的新发病变,证实既往乳腺摄影发现的陈旧病变。

RESULTS:Our results indicate that the chemokine CCL5 can induce migration in humanbreast carcinoma cell line MCF-7 in vitro and suggest a potential role for Ezrin in theprocesses of breast cancer cell migration,invasion and possibly metastasis responsed toCCL5.We demonstrate that Ezrin siRNA specifically inhibits Ezrin expression of MCF-7cells and deregulates several functions such as cell motility and cell invasion,indicatingthat Ezrin plays a key role in the development of human breast cancer cells.It\'s suggestedthat Ezrin may act as a new anti-invasion therapeutic target for human breast cancer.

趋化因子CCL5促进人乳腺癌细胞MCF-7的趋化与侵袭,Ezrin蛋白的激活在此过程中起着重要的作用;成功构建Ezrin特异性siRNA表达载体,可用于探讨乳腺癌的侵袭转移机制,并为有效地抑制肿瘤的转移提供新的生物治疗靶点。

Methods A hundred and thirty - three patients with surgically or pathologically proved breast diseases which comprised 46 breast carcinomas and 87 breast benign lesions(58 fibromas, 7 inflammatory masses and 22 proliferous lesions)underwent 2D uhraaonographies and measurements of PSV by color Doppler flow imaging .

133例经手术或病理证实的乳腺疾病患者,其中,乳腺癌46例,乳腺良性病变87例(包括纤维瘤58例,炎性包块7例及增生性病灶22例),均经二维超声检查,彩色多普勒血流显像测量PSV,对所有病人的超声表现进行回顾性分析,并重点地比较了乳腺癌与乳腺良性病变之间的PSV差异。

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