查询词典 brain cell

The histopathological changes of brain tissue include neuronal degeneration, edema of nerve cell and gliacyte, the obfuscation of chondriosome structure, dilation of endoplasmic reticulum and brain edema of the cerebral cortex, which can gradually recover.

结扎后主要病理改变：神经元变性，神经细胞与胶质细胞水肿，线粒体结构模糊，内质网扩张及窦旁皮质组织水肿；均能逐渐恢复。

The brain capillary and star glue quality cell which enwrap around it under thenormal condition form the permeation extremely low blood-brain barrier, onlypermit some little molecule to pass; When cerebral isc.

正常状态下的脑毛细血管及包裹在其周围的星形胶质细胞构成了通透性极低的血脑屏障，只允许一些小分子物质通过；当缺血性中风发生时，微血管通透性发生显著改变，这种病理变化与脑内皮细胞损伤导致的功能变化有直接的关联。

Meanwhile, activatedNF-κB also appeared in vascular endothelial cell in brain for more than 1 year. Long-term activation of NF-κB in neuroglia cells induced atrophy of involved brain.

同时，激活的NF-κB在创伤1小时后出现于脑血管内皮细胞内，持续时间也在1年以上，神经胶质细胞内NF-κB的长期激活可引起该部位的脑萎缩。

RESULTS: Fluorescein concentrated cell presented extensively around micro-vessels after injection of both FA and FD. The speed of diffusion of FD in the brain was faster than that of FA. After FA injection, fluorescein concentrated image presented also around vessels, which developed to the distal along vessels, those in the cortex developed toward cerebral surface and those in the white substance toward ventral basicranial vessels of brain. The speed and distance were all bigger than the simple diffusion.

结果：注入FA，FD之后均可见微血管周围广泛分布荧光浓集细胞；FD在脑内扩散速度大于FA；FA注入后还出现血管周围荧光浓集影像，并沿血管通向远处，在皮质者通向脑表面，在白质者通向脑腹侧颅底血管，速度与距离均大于简单扩散。

Therefore, this study would calculate and survey the following indexes as the quantitative indexes for evaluating the degrees of cell senescence in hepatic and brain tissues, including numbers of neurons and dendrites in brain tissues; numbers of normal hepatocytes, dikaryotic hepatocytes, swollen hepatocytes as well as flake necrosis.

为此，本研究将计测脑组织神经元与树突数量；正常肝细胞，双核肝细胞、肿胀肝细胞数目、肝组织小片坏死个数作为评价脑神经及肝组织细胞衰老程度的量化指标。

The cell from hippocampus, striatum, ependyma, thalamus opticus, olfactory bulb, cortex, brain stem, cerebellum, and spinal cord in human fetal brain were studied using HE staining and immunocytochemistry.

应用HE染色和免疫细胞化学方法观察人胎脑室管膜下区、海马、纹状体、丘脑、嗅球、皮层、脑干、小脑、脊髓的Nestin阳性细胞。

Multiple factors participated in the process of secondary brain edema and nerve injuries,such as the hydrostatic pressure during formation of the hematoma and clot retraction, activation of blood coagulation cascade, generation of thrombin,erythrocytolysis and the toxicity of hemoglobin, the activation of the complement system, space-occupying effects, rupture of the blood-brain barrier,secondary ischemia around the hematoma, inflammatory reaction of the neurons around hematoma and cell apoptosis.

脑出血后继发水肿及神经损害有多种因素参与，血肿形成过程中的流体静力压和血凝块的回缩、凝血瀑布的激活、凝血酶的产生、红细胞溶解和血红蛋白的毒性、补体的激活、占位效应、血脑屏障的破坏、血肿周围继发缺血、血肿周围组织神经细胞炎症反应及细胞凋亡等。

These results suggested that in RHR, ATPase activity of brain and kidney were decreased. Pra-C preserves ATPase activity in cell membrane of brain and kidney, which may attribute to its effect as a hypotensor.

提示：肾血管性高血压时伴有脑、肾细胞膜ATP酶活力的下降，Pra－C的有效降压作用与维护脑、肾细胞膜ATP酶活力的正常有关。

In the past twelve years there have been hundreds of studies showing these nonthermal effects--- such as DNA damage and nonrepair, opening of the blood-brain barrier (allowing toxins and pathogens to reach the brain), lowered immunity, decreased melatonin levels, effects on stress proteins, formation of micronuclei (aberrations in cell nuclei which are often markers for cancer), changes in calcium metabolism affecting communication between cells, changes in brainwave patterns as seen on EEG's, plus effects observed on many different systems of the body.

在过去十二个月里，已有数以百计的研究表明这些非热效应---如DNA损伤和nonrepair ，开放血脑屏障（允许毒素和病原体到达大脑），降低免疫力，降低褪黑激素水平，对应激蛋白，形成的微（畸变细胞核往往是癌症的标志），改变钙代谢的影响细胞之间的沟通，改变脑电波模式脑电图上看到的，加上效果观察许多不同的系统的机构。

These results suggest that the activate spleen-energy method can achieve appetitive purpose by not only affecting the gastrointestinal tract function in periphery, but also regulating the process of ingestion in brain. The basic research about ingestion indicate: one hand, after food enters the gastro-intestinal tract, the endocrine cell of the intestinal tract is activated and secrete many kinds of brains intestines peptide, the level of the brain intestines peptide in blood as the periphery signal spreads to the central nervous system regions that control digest and feeding behavior. On the other hand, it has been shown that dorsal parabranchial neurons, containing CCK-8S, extend fibers to the VMH and are involved in the inhibition of feeding .

关于摄食控制的基础研究表明，一方面食物进入胃肠道后，激活肠道的内分泌细胞，分泌多种脑肠肽如CCK-8S，这些脑肠肽在血中的水平作为外周信号可通过&肠—脑轴&传入中枢神经系统介导消化和摄食行为的部位，影响摄食中枢神经元的活动调节动物的摄食行为；另一方面中枢神经元可合成释放内源性的神经肽直接作用于摄食中枢神经元调节摄食，如中枢鳃旁体神经元的神经纤维就可延伸到达VMH，并且合成释放内源性的CCK-8S。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。