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bone-black相关的网络例句

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During the process of osteophyte formation mesenchymal cells covering the periosteal bone surface proliferate and differentiate into chondroblasts and osteoblasts, which build up cartilage and bone matrix.

在骨赘形成过程中,覆盖骨膜骨的间充质细胞增生、分化为软骨母细胞和成骨细胞,后两者构建了软骨和骨基质。

Results Soft tissue swelling: 19 cases showed on CT (degree Ⅰ 6 cases; degree Ⅱ: 5 cases; degree Ⅲ: 8 cases), and 12 cases showed on MRI (degree Ⅰ 4 cases; degree Ⅱ 3 cases; degree Ⅲ 5 cases); Arthroedema seven on CT, and Ⅱ on MRI; Synovium granulation hyperblastosis 13 on CT, and 10 on MRI; Changes of joint space 13 on CT, and 17 on MRI. Destruction of bone (6 cases), osteosclerosis (15), osteoporosis (4), bone pneumatosis (6) and abscess-like cyst (1) were seen on CT.

结果 CT显示软组织肿胀19例(Ⅰ度6例,Ⅱ度5例,Ⅲ度8例),MRI显示12例(Ⅰ度4例,Ⅱ度3例,Ⅲ度5例);CT显示关节腔积液7例,MRI显示Ⅱ例;CT显示滑膜肉芽组织增生13例,MRI显示10例;CT显示关节间隙改变13例,MRI显示7例;CT显示髋关节骨质破坏6例,骨质硬化15例,骨质疏松4例,骨内积气6例,脓肿样囊腔1例。

Objective:Osteoporosis is syndrome shown on the bone absorptive rate is higher than formative rate during the metabolism of their bones. As the result, the progression of calcium deficiency and the degeneration of bone tissue are the characters in this period. Clinical manifestations appear on these elder persons, which are osteosclerosis and increasing the possibility of fracture.

目的: 骨质疏松症(Osteoporosis,简称OP)是指在骨代谢过程中,骨吸收大于骨形成,出现以低骨量和骨组织的显微结构的退行性变为其特征,临床表现为骨脆性和骨折易感性增加的一种代谢性疾病。

Objective:Postmenopausal Osteoporosis is syndrome shown on women caused by the functional descent on both ovaries and the level of estrogen. During the metabolism of their bones, the bone absorptive rate is higher than formative rate. As the result, the progression of calcium deficiency and the degeneration of bone tissue are the characters in this period. Clinical manifestations appear on these elder women, which are osteosclerosis and increasing the possibility of fracture.

目的: 绝经后骨质疏松症(Postmenopausal Osteoporosis,PMO)是指绝经后妇女由于卵巢功能衰退,雌激素水平下降,从而在骨代谢过程中,骨吸收大于骨形成,出现以低骨量和骨组织的显微结构的退行性变为其特征,临床表现为骨脆性和骨折易感性增加的一种代谢性疾病。

Objective: Menopausal osteoporosis is syndrome shown on women caused by thefunctional descent on both ovaries and the level of estrogen. During the metabolism oftheir bones, the bone absorptive rate is higher than formative rate. As the result, theprogression of calcium deficiency and the degeneration of bone tissue are thecharacters in this period. Clinical manifestations appear on these elder women, whichare osteosclerosis and increasing the possibility of fracture.

目的: 围绝经期骨质疏松症是指围绝经期女性由于卵巢功能衰退,雌激素水平下降,从而在骨代谢过程中,骨吸收大于骨形成,出现以低骨量和骨组织的显微结构退行性变为特征,临床表现为骨脆性和骨折易感性增加的一种代谢性疾病。

Results Sacroiliac joints in 62 cases were abnormal:in early stage,articular face was affected and the bone cortex incrassated and osteosclerosis partly,joint space was normal;in late stage,vermiform destruction of bone was seen in articular face,the density of cortex was heighten irregular,joint space widen or narrow,and at last the joint space vanished,the joint ankylosed.

结果 62例患者骶髂关节表现异常,包括:早期,骨关节面受侵蚀、骨皮质局限增厚、硬化,关节间隙正常;后期,关节面呈虫蚀状改变,关节面皮质密度不规则增高,关节间隙增宽或变窄,最终关节间隙消失,关节强直。

Osteotome technique and Sinus floor elevation change the density of bone and increase the height of support bone,it is offer a good condition for success of implant.

骨挤压法提升上颌窦底的过程中,改变了骨密度,增加了支持骨的高度,为种植体的成功提供了良好的条件。

All the patients with 8-11 mm height of residual bone in the posterior maxilla underwent sinus floor elevation and implant placement using osteotome without any bone grafts simultaneously.

患者均因上颌后牙种植区牙槽嵴顶至上颌窦底之间的剩余骨高度在8~11mm,无法植入足够长度的种植体,因而采用上颌窦底冲压法局部提升上颌窦底骨板及黏骨膜,提升幅度为2~5mm。

An osteoporosis model was established by ovariotomy. The rats were given drugs after 12 weeks. Bone mineral density, bone biomechanics and biochemical indexes were measured after 24 weeks.

用手术切除卵巢方法制造骨质疏松模型,12周后按组别给药,24周后处死,分别测定股骨骨密度、骨生物力学水平以及血清生化指标并作统计学分析。

TNFR1/RANK chimera cloning of cDNA, with its plat E-retroviral package by retroviral infection such transfer to TNFR1/R2 gene knock-out mice (TNFR1-/R2-) Bone marrow macrophages in; TNFɑ used to stimulate, respectively, to observe the OC and differentiation of bone re-absorption capacity; confirmed by Western Blot method TNFR1/RANK through NF-?

克隆TNFR1/RANK嵌合体的cDNA,用plat E将其包装成逆转录病毒,通过感染将这种逆转录病毒转染到敲除TNFR1/R2基因的小鼠(TNFR1-/R2-)骨髓巨噬细胞中;用TNFɑ刺激,分别观察OC的分化及骨的重吸收能力;用Western Blot方法证实TNFR1/RANK是通过NF-?

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