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blood vessels相关的网络例句

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与 blood vessels 相关的网络例句 [注:此内容来源于网络,仅供参考]

The cause of embolism in blood vessels is the gradual gathering of fat,cholesterin and calcium ,resulting in the hardening of interior of blood vessels.

血管栓塞的成因是脂肪、胆固醇和钙质在血管内逐渐集聚并导致血管内部硬化。

Because genetic engineering has the potential to conquer cancer,grow new blood vessels in the heart,block the growth of blood vessels in tumor s,create new organs from stem cells and perhaps even reset the primeval genetic coding that causes cells to age

因为遗传工程有可能征服癌症,在心脏里培植新的血管,阻断肿瘤中血管的生长,从干细胞中选出新的器官,甚至可能重组可以延长细胞寿命的原始遗传编码。

There is an emerging understanding of the importance of the vascular system within stem cell niches. Here, we examine whether neural stem cells in the adult subventricular zone lie close to blood vessels, using three-dimensional whole mounts, confocal microscopy, and automated computer-based image quantification. We found that the SVZ contains a rich plexus of blood vessels that snake along and within neuroblast chains.

在另一篇来自美国和中国台湾科学家的文章中,作者研究了成年SVZ中神经干细胞是否紧密的位于血管周围,利用多种技术,科学家发现SVZ中成神经细胞链内以及周围含有丰富的血管丛。

McGregor (1973) [5] raised by direct skin vascular blood supply of the flap, and the musculocutaneous perforator vessels and blood vessels of the skin that is: axial and random flap flap two concepts.

McGregor(1973)[5]提出了由直接皮肤血管供血的皮瓣和由肌皮血管及穿支血管供血的皮瓣即:轴型皮瓣和随意皮瓣两种概念。

Central aorto-pulmonary shunts were performed with teflon artificial blood vessels in 12 cases and varicosity teflon artificial blood vessels in 16 cases.

使用彩色多普勒超声检测其术后肺动脉直径和左心室容积的变化。

Angiogenesie and vasculogenesis comprise the mechanisms that responsible for the development of new blood vessels. Vasculogenesis is the process of in situ formation of blood vessels from endothelial progenitor cells or angioblast. Angiogenesis involves extension of the already formed primitive vasculature by the sprouting of new capillaries through migration and proliferation of previously differentiated ECs.

新血管形成的机制有血管生成和血管新生,前者是血管内皮前体细胞或成血管母细胞在原位分化为内皮细胞并形成血管的过程,后者则是已经分化的成熟内皮细胞移行并增生使原有血管延伸。

Methods:Lower abdominal flaps were formed by using iliolumbar flaps with the superficial iliac and superficial epigastric blood vessels as the pedicles. They were co-pedicled and bilobate flaps, which were cut thin to form subcutaneous vascular net flaps with axis blood vessels. The flaps were used to repair complete dismantled injuries of two fingers.

在下腹部形成以旋髂浅血管为蒂的髂腰部皮瓣及以腹壁浅血管为蒂的下腹部皮瓣,二皮瓣共蒂形成双叶皮瓣,同时将其修薄,形成有轴心血管的真皮下血管网皮瓣,修复两指完全脱套伤。

Results CT shows 18 cases of mesenteric edema, 2 cases of mesenteric pneumatosis, 7 cases of mesenteric vascular anomalies, 5 cases of mesenteric vascular were filling defect, 2 cases of the trunk of the mesenteric vascular were thickening, 21 cases of segmental mesenteric small blood vessels hyperemia, 7 cases of segmental mesenteric small blood vessels ischemia and 2 cases of the mesenteric venous accumulation of gas.

结果 CT表现:肠系膜水肿18例,肠系膜积气2例,肠系膜血管走行异常7例,肠系膜血管充盈缺损5例,肠系膜主干血管增粗2例,节段性肠系膜小血管充血21例,节段性肠系膜小血管缺血7例,肠系膜静脉积气2例。

While the tissue spaces surrounding a few blood vessels wasAl and Fg positive,no Al or Fg positive cells were observed.In antemortem injurygroup,diffuse subarachnoid hemorrhage,cerebral edema,swelling or pyknotic neu-rons could be observed.The axons showed irregular swelling and disconnection at1~3h,marked swelling and disconnection at 6h,and retraction ball at 15h whichwas more remarkable at 24h after injury.The space between myelin sheaths andaxons was increased at 3~6h after injury.Tortuous and wavelike myelin sheathswhich adhered on axons incompletely,or even peeled off could be found from 15hto 24h after injury.Perinuclear lysis of Nissl bodies began at 24h after injury.Thenumber of GFAP positive cells in cerebrum and brain-stem increased significantlyfollowed by decrease,and then increased again,but the time courses of the changesin different areas of brain were not same.Al and Fg positive neural cells,mainlysurrounded blood vessels,with diffuse or peripherally distributed positive matter incytoplasm could be observed at 0.5h after injury.The number of Al or Fg positivecells and the intensity of immunoreaction increased with the time of injury.The areaof SYN positivity in medulla oblongata and pons decreased notably 3~6h afterinjury,then return to normal levels and continued to 24h after injury.

生前损伤组,可见广泛蛛网膜下腔出血,脑组织水肿,神经细胞肿胀,晚期神经元固缩;伤后1~3h见部分神经轴突不规则增粗、断裂,伤后6h断端膨大,伤后15h可见收缩球,至伤后24h更为明显;伤后3~6h可见部分神经髓鞘与轴突之间的间隙增宽,伤后15h髓鞘明显曲折,不完全附着在轴突两侧,甚至剥脱,持续到伤后24h;核周尼氏体减少在伤后24h才开始出现;同一部位的GFAP阳性细胞数目随损伤时间发生改变,先增多(最早在伤后0.5h),达到高峰后减少,其后又有增多趋势,但不同部位的GFAP阳性细胞数目增减的时间过程不尽相同,同时,大脑中的GFAP阳性细胞数目也有改变;伤后0.5h,可在脑干组织中见到Al和Fg阳性神经细胞,主要位于血管周围,阳性物在胞浆中呈弥散性分布,但部分细胞的阳性物仅分布于靠近胞膜的胞浆中而呈环状,随损伤时间延长,阳性细胞数目增多,反应强度增加;伤后3~6h,延髓及桥脑中的SYN阳性物面积减少,其后恢复到正常水平,并持续到伤后24h。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

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