查询词典 blood platelet

Besides, HIF-1 can quickly induce a lot of expressions and the adjustment of anoxia-induced genes, so as to enhance the hypoxia-induced genetic transcription and increase the expressions, and engender physiological effects, such as adjusting the transcription of EPO genes to increase their production, thereby the red blood cells quantity is enlarged, which strengthens the ability of oxygen transportation and mitigates anoxia; adjusting VEGF genetic transcription to engender more blood capillaries, consequently the transportation and supply of oxygen and substance of energy source are strengthened; adjusting the glycolytic enzyme to enhance glycolysis ability under anoxemia condition and increase energy supply; functioning on HO-1 genes in vascular smooth muscle cells to produce HO-1, which can engender NO and CO as pneumatic signal molecules to activate the guanylate cyclases, thus CGMP level is improved to make the vascular smooth muscles chalasia, to restrain platelet agglutination, so that theblood flow and vascular permeability are increased, and the anoxic tissues obtain ample oxygen supply, etc..

在低氧环境中，HIF-1可以被很快诱导大量表达并介导低氧诱导基因的调控，使低氧诱导基因转录增强，表达产物增多，产生生理效应。

Including whole blood, RBCs suspension, RBCs concentration, washed RBCs, frozen and deglycerolized RBCs, RhD negative blood. granulocyte, aphaeresis platelet concentration, cryoprecipitate and FFP.

目前，中心为昆明地区的110余家医疗机构提供全血、悬浮红细胞、洗涤红细胞、冰冻解冻去甘油红细胞，各型RH血液，单采血小板，单采粒细胞，新鲜冰冻血浆，冷沉淀等，年供血量达18吨。

Methods Venous blood from every healthy elderly was observed by the platelet nomogram after blood corpuscle analysis.

每例检测对象的静脉血做完血细胞分析后，调出逆方图观察其血小板数量与血小板体积在直角坐标图构成点的位置。

These include blood glucose, electrolytes, complete blood count with platelet count, prothrombin time, activated partial thromboplastin time, international normalized ratio, and renal function studies.

这些措施包括血糖、电解质、全血计数与血小板计数、凝血酶原时间、活化部分凝血活酶时间、国际标准化比值，和肾功能研究。

Both thrombophlebitis and pyemia are typical examples of hypercoagulatory states caused by inflammation.In fact,inflammation had been showed as an important reason that lead to thrombotic events in a lot of studies of atherosclerosis and heart failure.Further investigations have showed a prothrombotic state with blood hypercoagulation,platelet activation,damaged atrial endothelium and atrial blood stasis in AF,while these changes are due to the AF itself,or coexistent cardiovascular risk factors remains a matter of debate.

虽然房颤时高凝状态和血小板功能激活是非常明确的，是造成房颤血栓形成的重要原因之一(其他两个原因包括心房血流淤滞和内皮功能损害)，但是房颤高凝状态和血小板功能激活与炎症水平的关系，尽管对指导采取何种预防血栓形成的治疗策略意义很大，目前已有的研究较少涉及这方面，国内外几乎没有相关的研究报告。

EPA and RFCA can cause the activation of platelet and changes the blood coagulable states by increasing the levels of the blood coagulable substances,lead to thronboembolic events.

EPS和RFCA术可引起血小板活化、纤溶系统激活，血凝状态改变，致血栓形成。

tonifing qi and nourishing yin and promoting blood to restore luo theropy can improve the endothelial function, relief coronary spasm, get rid of OFR and inhibit hyperoxidation of lipid, reduce blood thickness and platelet activity, so reliefathrosclerosis and prevent the formation of throbosis. So unstable angina is treated.

益气养阴活血通络法能缓解冠脉痉挛，改善血管内皮功能，清除氧自由基，抑制脂质过氧化，降低血液粘度和血小板活化，从而起到延缓动脉粥样硬化，防止血栓形成的作用。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后，血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少，Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少，血小板聚集程度及红细胞聚集指数显著上升，病理切片显示胸主动脉内皮细胞破坏明显，脑心肾组织内的血管进一步增厚甚至几乎阻塞，导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR，脑体重比显著升高；心肌内出现明显的缺血梗塞疤痕灶，心体重比显著升高；肾小球明显萎缩、玻璃样变性，肾小管也变性甚至坏死，使肾硬化程度显著增加呈颗粒性固缩肾，肾体重比也显著减少。

The perioperative counts of Hemoglobin, haematocrit and platelet, and also the amount of blood savage blood or the allogenic blood transfusion of the 2 groups were recorded and compared.

进行比较，分别于两组患者术前、术后第1、3、5、14d各取病人静脉血，记录两组患者围手术期血红蛋白、红细胞比容、血小板计数、凝血时间、出血时间检测结果，分别记录术中回输的血量和输入异体血液量。

However, a pancytopenia of unknown cause was verified to peripheral blood. During the course of supportive treatment only with blood cell and platelet transfusion, WBC count of this patient showed a rising trend and the blast cells (8%-15%) started to occur in the peripheral blood.

但患者出现不明原因的外周血全血细胞下降，在单独采用输血和血小板支持治疗的同时动态观察其血常规发现，外周血白细胞开始逐渐上升并出现原始细胞(8%~15%)。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。