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bleeding相关的网络例句

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与 bleeding 相关的网络例句 [注:此内容来源于网络,仅供参考]

Of the follow-up studies on 10 mixed bleeding cases,2 was normal,1 was dead,and the others were cerebromalacia,hydrocephalus,cerebral atrophy,porencephalia and calcium.

在10例混合性出血中,随访复查发现2例恢复正常,1例死亡,7例继发脑软化、脑积水、脑萎缩、脑穿通畸形等后遗改变。

Of the follow?up studies on 30 mixed bleeding cases,17 was normal,2 was dead,and the others were cerebromalacia,cerebral atrophy,porencephaty and calcium.

在30例合并颅内出血中,随访复查发现17例恢复正常;其他13例中,2例死亡,11例均有脑软化、脑萎缩、脑穿通畸形、脑钙化灶等不可逆性后遗症的表现。

Of the followup studies on 30 mixed bleeding cases,17 was normal,2 was dead,and the others were cerebromalacia,cerebral atrophy,porencephaty and calcium.

在30例合并颅内出血中,随访复查发现17例恢复正常;其他13例中,2例死亡,11例均有脑软化、脑萎缩、脑穿通畸形、脑钙化灶等不可逆性后遗症的表现。

The middle hepatic vein intruded in the cholecystic bed should be protected during cholecystertomy to avoid unnecessary bleeding.

胆囊切除时,应注意保护肝中静脉及属支,以防止出血。

Bleeding during operation is 4 cases and post operation is 1 case.conclusion it must be perform the surgery indications strictly, reasonable operation skill, standard manipulation, familiar with the anatomic variation of cholecystic vessels and biliary ducts and abdominal operation.in time all methods can effectively decrease the complication of lc.

结果 胆管损伤2例,术中出血4例,术后胆漏5例,术后出血1例。结论严格掌握手术指征,合理的手术技巧,规范的操作,熟悉胆囊管和血管的解剖及变异,及时果断的中转开腹等,可以有效的减少lc并发症的发生。

Results: Among 80 cases of ACST, 76 cases were successfully implemented LC, gallbladder puncture and decompression choledocholithotomy T-tube drainage were performed in 11 cases, choledocholithotomy T-tube drainage in 15 cases, common bile duct gall bladder fistula Toishi T-tube drainage in 14 cases, gallbladder removal choledocholithotomy T-tube drainage in 36 cases, operative time 15-120 min, bleeding 10-80 ml; 4 cases transitted laparotomy, 1 case of gallbladder and surrounding tissue adhesion serious, ampulla signs disappear, and the hepatoduodenal ligament ill-defined, 2 cases of gallbladder contraction deep within the liver, gallbladder triangle scar adhesions, and 1 case of duct stone impaction and severe adhesions; postoperative hospital stay was 8-11 d, all were well recovered; pathological diagnosis, acute cholecystitis in 41 cases (51.2%), acute suppurative cholecystitis in 24 cases (30.0%), acute gangrenous cholecystitis in 15 cases (18.8%).

结果:重症急性胆囊炎80例,76例成功实施LC,其中,胆囊穿刺减压胆总管切开取石T管引流11例,胆总管切开取石T管引流15例,胆囊造瘘胆总管切开取石T管引流14例,胆囊切除胆总管切开取石T管引流36例,手术时间15~120 min,术中出血10~85 ml;4例中转开腹,其中,1例胆囊与周围组织粘连严重,壶腹部标志消失,与肝十二指肠韧带界限不清,2例胆囊萎缩深陷肝脏内,胆囊三角区瘢痕粘连,1例胆囊管结石嵌顿且严重粘连,术后住院时间8~11 d,术后恢复均良好,治愈出院;80例术后病理诊断,急性单纯性胆囊炎41例(51.2%),急性化脓性胆囊炎24例(30.0%),急性坏疽性胆囊炎15例(18.8%)。

Results: All these patients under went the second laparotomy, the residual gallbladder and cholelith were excised successfully in 55 cases. 1 case was unsuccessful because of severe adhesion and bleeding. After successful operation, the symptoms disappeared and the results were satisfactory.

结果:除1例因粘连严重并发门脉高压症,分离时引起大出血被迫终止手术外,55例再次手术成功,切除残留胆囊及结石后,症状均消失,疗效满意。

And because the fast light performance, not the ink pigment caused by bleeding and chromotropic, not because of aging and yellowing change colour.

又因其具有耐光性能,不造成油墨颜料的渗色和变色,不因老化而返黄变色。

objective:To study the role of non-steroidal anti-inflammatory drugs in esophageal variceal bleeding in liver cirrhotic patients.

目的:探讨非甾体类消炎药在肝硬化食道曲张静脉破裂出血中的作用。

Vascular hyperpermeability is one of the important causein hypovole-mia and organs disfunction after various kinds of shock,and its treatment is still one of the problems in the medical field. For example,the operative methods of preventing hyperpermeability and plasm bleeding is still unreliable after severity burn,and traditional treatment just passively transfuses according to the burn clysis formula instead of inhibiting permeability by injecting drugs. The principal reason is that pathogenesis of vascular endothelial cell permeabilty has not been entirely revealed at the level of molecular biology.

血管通透性增高是休克时有效血容量减少和器官功能损害的重要原因之一,它的防治一直是医学界的一个难题,如在大面积烧伤时,至今未能找到一个简单有效的防治血管通透性升高和血浆渗出丢失的方法,传统的方法只是根据烧伤补液公式测算进行被动的补液来纠正,而不能依靠注射化学制剂来终止其渗出,主要原因是未能从分子生物学水平揭示血管内皮细胞渗出的机制。

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