查询词典 bilateral

Methods Arterial bypass with prosthesis - graft was carried out in 15 cases: ascending aorta- bilateral axillar arteries and unilateral ICA bypass in 6 cases, ascending aorta - bilateral axillar arteries bypass in 3 cases, ascending aorta - unilateral axillar arteries and unilateral ICA bypass in 5 cases, and in one case of complicated TA combined with abdominal aorta, ascending aorta- bilateral axillar arteries and unilateral ICA bypass were performed, followed by unilateral axilla - femeral bypass.

方法15例重症头臂型大动脉炎患者经胸行人工血管旁路术，升主动脉-双腋动脉、单颈内动脉架桥6例，升主动脉-双腋动脉架桥3例，升主动脉一单腋动脉、单ICA架桥5例，1例合并腹主动脉闭塞的复合型，一期行升主动脉-双腋动脉、单ICA架桥，二期行单侧腋股搭桥术。

1、Neural network correlated with LIFG and RIFG exists in normal subjects in resting-state,which might be the neural foundation in retaining the normal language function;2、The left fusiform gyrus,bilateral medial frontal gyrus and right anterior cingulum in aphasia patients after stroke showed low degree correlation,that might be one of the mechanisms of aphasia;3、The brain regions showed positive correlation with RIFG in aphasia patients only exist in right hemisphere in resting-state as well as which exist in bilateral hemisphere in normal subjects,suggested that RIFG is out of correlation with the left hemisphere in aphasia patients;4、The left thalamus showed positive correlation with RIFG only in normal subjects, inferred that the left thalamus might be an important mesomerism structure in the correlation of bilateral hemisphere;5、Left insula showed stronger positive correlation with RIFG in normal subjects than that in aphasia patients,suggested that dominant hemisphere insula is important in retaining in normal language function.

1、正常人在静息状态下即存在与LIFG和RIFG具有相关性的神经网络，该网络可能是维持正常语言功能的神经基础；2、脑梗死后运动性失语患者左侧梭状回、双侧额叶内侧回、右侧前扣带与LIFG的连接程度的减低可能是运动性失语的发生机制之一；3、与RIFG正相关的脑区在对照组为双侧半球分布，而在患者组仅出现在右侧半球，说明患者组RIFG与左侧半球的连接中断；4、RIFG与左侧丘脑的正相关性仅出现在正常对照组中，推测左侧丘脑可能是联系两侧半球功能区的重要中介结构；5、对照组与RIFG正相关性明显高于患者组的脑区位于左侧岛叶，说明优势半球岛叶对维持正常语言功能具有重要作用；第二部分脑梗死运动性失语患者语言恢复机制的静息态fMRI初步探讨目的：运用fMRI技术，采用种子点方法在静息状态下分析脑梗死运动性失语患者不同的恢复阶段语言功能连接方式。

Results: In the 13 cases, the CT diagnosis show that five cases of low-density lesions of Bilateral hepatolenticular, five cases of globus pallidus, one case of Bilateral hepatolenticular with corpus callosum, one cases of External capsule putamen, one case of Putamen with the caudate nucleus, three cases of low-density of Bilateral lobi frontalis and lobi parietalis, one case of unilateral putamen hemorrhage, three cases of cerebral atrophy.

结果 13例中，CT示双侧豆状核低密度5例、苍白球5例、双侧豆状核伴胼胝体1例、壳核伴外囊1例、壳核伴尾状核1例；双额顶叶低密度3例、单侧壳核出血1例、大脑萎缩3例。

Chapter 4 is the central part of this dissertation, where the bayes bilateral integrated moment is established on the base of Bayes theory, downside risk and upside potential. According to the variation coefficient which is based on the M-V approach, we substitute lower partial moment for the numerator of variation coefficient, and use higher partial moment as the substitute of the denominator, then we got bilateral integrated moment. Subsequently, through Bayes method we can absorb new information, revise the return distribution from transcendental distribution to posterior distribution, so we got bayes bilateral integrated moment.

第四章是本文的核心部分，在简要介绍Bayes理论、下方风险和上行潜能的基础上，仿照来源于Markowitz的均值——方差理论的变异系数方法，以下偏矩取代其分子部分，以上行潜能取代其分母部分，设计了双侧综合矩方法，接着再引入Bayes方法，吸纳新信息，采用后验概率，设计了Bayes双侧综合矩方法，并在理论上说明了新方法的优越性。

Her 9 years old daughter has bilateral blue irides, bilateral total deafness, dystopia canthorum and bilateral fundus pigmentary changes.

第三例为第一例九岁之小女儿，双侧虹彩全蓝，双侧全聋，内眥移位和双侧眼底色素变化。

Via generalizing the Cauchy method we obtain a new method,called the modified Cauchy method.By means of this method we establish two bilateral _3ψ_3 and _4ψ_4 series summation formulae,two four-term summation and transformation formulae for unilateral _3φ_2-series and bilateral _3ψ_3-series,and two five-term summation and transformation formulae for unilateral _3φ_2-series and bilateral _3φ_3-series,which contain many known results as their special cases,such as non-terminating q-Saalschütz summation formula,Bilateral _6ψ_6 series summation formula of Bailey,non-terminating Watson transformation formula and some transformations of _3φ_2-series etc.

通过对Cauchy方法的推广，我们得到修正的Cauchy方法，采用这个方法分别得到两个双边的_3ψ_3和_4ψ_4基本超几何级数的求和公式、单边_3φ_~(2-)级数和双边_3ψ_(3~-)级数的两个四项求和变换公式和两个五项求和变换公式，它们包括许多已有的结果为特例，如非终止的q-Saalschütz求和公式、Bailey的very-well-poised双边级数_6ψ_6求和公式、非终止的Watson变换公式和一些关于单边_3φ_(2~-)级数的变换公式等。

After 60-day treatment with clozapine, ACI group was treated with ACI alternatively on Baihui (GV20), bilateral Xinshu (BL15) and bilateral Quchi (LI11), or on Dazhui (GV14), bilateral Pishu (BL20), bilateral Zusanli (ST36), one time every 5 days, 12 times in total.

服用氯氮平60 d后埋线组选取两组穴位：①百会、心俞、曲池；②大椎、脾俞、足三里，交替埋线，每5 d治疗1次，共12次。

One-sample t test was used in profoundly congenital hearing loss infants and control group,the activated brain areas were:bilateral transverse temporal gyri, superior temporal gyrus,middle temporal gyrus,bilateral insular lobe,bilateral precentral gyrus,postcentral gyrus,bilateral cingulate gyrus,bilateral inferior parietal lobule,bilateral superior parietal lobule,bilateral superior frontal gyrus.

在对极重度感音神经性耳聋患儿和对照组进行组间比较发现，病人组数据减去对照组数据时可见激活的脑区主要有：双侧颞横回、双侧颞上回、双侧颞中回、双侧岛叶、双侧中央前、后回、双侧扣带回、双侧顶下小叶、双侧顶上小叶、双侧额上回等，提示极重度感音神经性耳聋患儿在接受刺激后动用了更多的脑区而且激活强度明显增加。

Objective: To analyze the clinical characteristics and pathogensis of Bilateral Ventromedial Thalamic Syndrome and to improve our understanding of the disease.

目的：分析少见的双侧腹内侧丘脑综合征(Bilateral Ventromedial Thalamic Syndrome， BVTS)，就其临床特点及发病机制进行探讨，提高对该病的认识。

The effects and mechanism of GABAergic neurons, NOergic neurons, opioid peptide and cyclic adenosine monophosphate in the nucleus reticularis thalami on sleep-wakefulness cycle of rats and the effects and mechanism of the 5-HTergic nerve fibers project from the nucleus raphes dorsalis to RT on sleep-wakefulness cycle of rats were investigated with the methods of brain stereotaxic, nucleus spile, microinjection and polysomngraphy.1. The effects of GABAergic neurons in RT on sleep-wakefulness cycle of rats1.1 Microinjection of 3-mercaptopropionic acid (3-MP, a kind of glutamate decarboxylase inhibitor) into RT. On the day of microinjection, sleep only decreased a litter. On the second day, sleep marked decreased and wakefulness marked increased. On the third and fourth day, sleep and wakefulness stages resumed to normal.1.2 Microinjection of gamma-amino butyric acid (GABA 1.0μg) into RT enhanced sleep and reduced wakefulness compared with control; while microinjection of L-glutamate (L-Glu, 0.2μg) decreased sleep and increased wakefulness; microinjection of bicuculline (BIC, 1.0μg), a GABAA receptor antagonist, enhanced wakefulness and reduced sleep; microinjection of baclofen (BAC, 1.0μg), GABAB receptor agonist, had the same effects as GABA.2. The effects of NOergic neurons in RT on sleep-wakefulness cycle of rats2.1 Microinjection of L-arginine (L-Arg, 0.5μg) into RT decreased sleep compared with control, but there were on statistaical difference between L-Arg group and control; while microinjection of sodium nitroprusside (SNP, 0.2μg), a NO donor into RT, sleep marked decreased and wakefulness marked increased. Microinjection of nitric oxide synthase inhibitor, N-nitro-L-arginine (L-NNA, 2.0μg) into RT enhanced sleep and reduced wakefulness.2.2 After simultaneous microinjection of L-NNA (2.0μg) and SNP (0.2μg) into RT, SNP abolished the sleep-promoting effect of L-NNA compared with L-NNA group; after simultaneous microinjection of L-NNA (2.0μg) and L-Arg(0.5μg) into RT, we found that L-NNA could not blocked the wakefulness-promoting effect of L-Arg.3. The effects of opioid peptide in RT on sleep-wakefulness cycle of rats3.1 Microinjection of morphine sulfate (MOR, 1.0μg) into RT increased wakefulness and decreased sleep compared with control; while microinjection of naloxone hydrochloride (NAL, 1.0μg), the antagonist of opiate receptors, into RT, enhanced sleep and reduced wakefulness.3.2 After simultaneous microinjection of MOR (1.0μg) and NAL (1.0μg) into RT, the wakefulness-promoting effect of MOR and the sleep-promoting effect of NAL were not observed compared with control.4. The effects of cAMP in RT on sleep-wakefulness cycle of rats Microinjection of cAMP (1.0μg) into RT increased sleep and decreased wakefulness compared with control; microinjection of methylene blue (MB,1.0μg) into RT enhanced sleep and reduced wakefulness compared with control.5. The effects of the 5-HTergic nerve fibers project from DRN to RT on sleep-wakefulness cycle of rats5.1 When L-Glu (0.2μg) was microinjected into DRN and normal sodium (NS,1.0μg) was microinjected into bilateral RT. We found that sleep was decreased and wakefulness was increased compared with control; when L-Glu (0.2μg) was microinjected into DRN and methysergide (MS,1.0μg), a non-selective 5-HT antagonist, was microinjected into bilateral RT, We found that sleep was enhanced and wakefulness was reduced compared with L-Glu group.5.2 When p-chlorophenylalanine (PCPA, 10μg) was microinjected into DRN and NS (1.0μg) was microinjected into bilateral RT, We found that sleep was increased and wakefulness was decreased compared with control; microinjection of 5-hydroxytryptaphan (5-HTP, 1.0μg), which can convert to 5-HT by the enzyme tryptophane hydroxylase and enhance 5-HT into bilateral RT, could block the effect of microinjection of PCPA into DRN on sleep-wakefulness cycle.

本研究采用脑立体定位、核团插管、微量注射、多导睡眠描记等方法，研究丘脑网状核(nucleus reticularis thalami，RT)中γ-氨基丁酸(gamma-amino butyric acid ，GABA)能神经元、一氧化氮(nitrogen monoxidum，NO)能神经元、阿片肽类神经递质、环一磷酸腺苷(cyclic adenosine monophosphate，cAMP)及中缝背核(nucleus raphes dorsalis，DRN)至RT的5-羟色胺(5-hydroxytryptamine，5-HT)能神经纤维投射对大鼠睡眠-觉醒周期的影响及其作用机制。1 RT内GABA能神经元对大鼠睡眠-觉醒周期的影响1.1大鼠RT内微量注射GABA合成关键酶抑制剂3-巯基丙酸(3-MP，5μg)，注射当天睡眠时间略有减少，第二日睡眠时间显著减少，觉醒时间明显增多，第三、四日睡眠和觉醒时间逐渐恢复至正常。1.2大鼠RT内微量注射GABA受体激动剂GABA( 1.0μg)后，与生理盐水组比较，睡眠时间增加，觉醒时间减少；而RT内微量注射L-谷氨酸(glutamic acid， L-Glu， 0.2μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAA受体阻断剂荷包牡丹碱(bicuculline，BIC，1.0μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAB受体激动剂氯苯氨丁酸(baclofen，BAC，1.0μg)后，产生了与GABA相似的促睡眠效果。2 RT内NO能神经元对大鼠睡眠-觉醒周期的影响2.1大鼠RT内微量注射NO的前体L-精氨酸(L-Arg，0.5μg)后，与生理盐水组对比，睡眠时间略有减少，但无显著性意义；而RT内微量注射NO的供体硝普钠(Sodium Nitroprusside，SNP，0.2μg)后可明显增加觉醒时间，缩短睡眠时间；微量注射一氧化氮合酶抑制剂L-硝基精氨酸(L-arginine，L-NNA，2.0μg)后，引起睡眠时间增多，觉醒时间减少。2.2大鼠RT内同时微量注射L-NNA(2.0μg)和SNP(0.2μg)后与L-NNA组比较发现SNP逆转了L-NNA的促睡眠作用；RT内同时微量注射L-NNA(2.0μg)和L-Arg(0.5μg)后，与L-NNA(2.0μg)组比较发现L-Arg可以增加觉醒而缩短睡眠，其促觉醒作用未能被NOS的抑制剂L-NNA所逆转。3 RT内阿片肽对大鼠睡眠-觉醒周期的影响3.1大鼠RT内微量注射硫酸吗啡(morphine sulfate，MOR，1.0μg)后与生理盐水组对比，睡眠时间减少而觉醒时间增加； RT内微量注射阿片肽受体拮抗剂盐酸纳洛酮(naloxone hydrochloride，NAL，1.0μg)后与生理盐水组比较，睡眠时间增加而觉醒时间减少。3.2大鼠RT内同时微量注射MOR(1.0μg)和NAL(1.0μg)后，与生理盐水组对比，原有的MOR促觉醒效果和NAL的促睡眠效果都没有表现。4 RT内环一磷酸腺苷信使对大鼠睡眠-觉醒周期的影响大鼠RT内微量注射cAMP(1.0μg)后与NS(1.0μg)组比较，睡眠时间增多而觉醒时间减少；RT内微量注射亚甲蓝(methylene blue，MB，1.0μg)后，与NS组比较，睡眠时间增多而觉醒时间减少。5中缝背核投射到丘脑网状核的5-羟色胺能神经纤维对大鼠睡眠-觉醒周期的影响5.1大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 0.2μg)比较，睡眠时间减少，觉醒时间增多；大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射二甲基麦角新碱(methysergide， MS， 1.0μg )后，与对照组(DRN注射L-Glu 0.2μg，双侧RT注射NS 1.0μg)比较，睡眠时间增多，觉醒时间减少。5.2大鼠DRN内微量注射对氯苯丙氨酸(p-chlorophenylalanine，PCPA，10μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 1.0μg)比较，睡眠时间增多，觉醒时间减少；大鼠DRN内微量注射PCPA(10μg)，产生睡眠增多效应后，在双侧RT内微量注射5-羟色胺酸(5-hydroxytryptaphan ， 5-HTP， 1.0μg )后，与对照组(DRN注射PCPA 10μg，双侧RT注射NS 1.0μg)比较，睡眠时间减少，觉醒时间增多。

The basic concept of FOP can be summarized as to further optimize effective prescription according to the standard of curative effects and with the aid of modern science and technology and theories of traditional Chinese medicine.

其基本内涵可概括为：以确有疗效的中药复方为研究对象，以现代科学技术和传统中医药理论为技术支持，以该复方所治病证的药效响应为评价标准，以优化重组疗效更优的新复方为研究目的。

Ever since our world has been a world, native forests have been indiscriminately exploited by man.

自从我们的世界一直是世界原生森林被任意剥削人。