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asparaginic相关的网络例句

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与 asparaginic 相关的网络例句 [注:此内容来源于网络,仅供参考]

Several chemical modification agents were applied to investigate amino acid residues in active site of this enzyme and the results demonstrated that histidine, serine, glutamic acid/asparaginic acid are located in the active site.

随后利用不同的化学修饰剂对酶蛋白进行修饰,通过酶催化活力的改变对位于酶活性位的氨基酸残基进行分析,结果表明酶活性位可能含有组氨酸、丝氨酸和谷氨酸。

The composition of amino acid of the enzyme was also analyzed,which indicated that the contents of asparaginic acid, serine, glutamic acid, glycin, alanine are the most, lysine, histidine also have high contents and there are a few praline, tryptophan.

最后,对此酶的氨基酸成分进行了分析,结果表明,此酶分子中天冬氨酸、丝氨酸、谷氨酸、甘氨酸、丙氨酸含量高,两种碱性氨基酸精氨酸、赖氨酸及组氨酸的含量也比较高,而脯氨酸、色氨酸的含量相对较低。

Results: The changes following hydrocortisone intervention of rats could be identified by PCA, and the kidney deficiency syndrome rats entered a state of "hyperfunction", involving a series of changes in metabolic pathway and metabolic network. The response integral area of lactic acid (δ 1.37) increased, indicating the accumulation of metabolites of lactic acid. The amount of dimethylamine (δ 2.72) also increased, indicating that the disorder of kidney function was related to kidney-deficiency syndrome. The response of creatinine (δ 3.87), asparaginic acid (δ 2.83), taurine (δ 3.44, 3.28), and hippurate (δ 7.84, 7.56, 7.64, 3.97) all decreased significantly, indicating that the hydrocortisone might cause adrenal cortex excretion injury. The response integral area of succinate (δ 2.41) and citric acid (δ 2.53, 2.68) decreased, which was usually caused by disorder of the mitochondrial function.

结果:PCA方法处理肾虚证和空白对照大鼠尿液数据显示,两组大鼠的数据可以在得分图实现分类,与对照组比较肾虚组大鼠由于氢化可的松诱导,大鼠机体进入一个过消耗后衰弱的状态,相关代谢发生显著变化,乳酸(δ 1.37)代谢发生堆积;二甲胺(δ 2.72)的含量增加,提示肾虚与肾功能异常是密切相关的;天冬氨酸(δ 2.83),牛磺酸(δ 3.44, 3.25),马尿酸(δ 7.84, 7.56, 7.64, 3.97),肌氨酸(δ 3.87)等的相对积分面积明显下降,这预示糖皮质激素可能引起了肾上腺皮质分泌功能的损害;琥珀酸(δ 2.41)和柠檬酸(δ 2.53, 2.68)是三羧酸循环能量代谢及糖酵解的中间产物,其含量的降低,通常是由于线粒体功能紊乱所引起。

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