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articular dropsy相关的网络例句

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与 articular dropsy 相关的网络例句 [注:此内容来源于网络,仅供参考]

Three months later, the surface of the articular was smoother than the sham-operated side. The histological score of cartilage was significantly higher than the sham-operated side in patellofemoral joint group p<0.05=, in the tibiofemoral group the difference was not obvious.

处理后3月,发现髌股关节组双极射频治疗侧髌骨软骨表面仍光滑,软骨组织评分高于对照侧(p<0.05);胫股关节组双极射频治疗侧股骨内髁软骨面的形态比对照侧平整,软骨组织学评分没有显著差异。

Smad3 was shown to be expressed in articular cartilage and trabecular bone surface.

我们测定了Smad3在骨骼发育的表达并发现Smad3表达在关节软骨细胞以及骨小梁表面。

RESULTS: During 10 - 12 weeks, in cuntrol group: The defect area was repaired by white and soft tissue that had no resistance to press. The repaired tissue was still lower than the surrounding articular surface with clear boundary. By histological observation, it was found that the defect was repaired by the mechanism similar to inflammatory reaction and the defect is ultimately filled by the hyperplasia of hyaline degenerative fibrous tissues. In filling group: the defect was repaired by semi-transparent, smooth, textured tissues with polish that had resistance to press as well as elasticity. The repaired tissue was almost similar to the shape of the surrounding cartilage,difficult to be distinguished. After histological observation, it was found that there was no inflammatory reaction, but active hyperplasia of inner bonetissue and cartilage tissues; a lot of osteoid tissues and trabeculation were found. Newlborn cartilage was fused with surrounding cartilage tissue and connected with surrounding tissues.

结果:10~12周,对照组:缺损区由白色、质软、按压无阻抗的组织修复,修复组织仍低于周围关节面,边界仍清晰可辨,组织学以类似炎症反应的机制修复缺损,最终以透明变性的纤维组织的增生来填补缺损部位;填充组:缺损区由半透明状、质韧光滑有光泽,按压有阻抗并有弹性的组织修复,修复组织与周围软骨外形上已基本相似,不易区分,组织学未见有炎症反应的过程,内骨组织和软骨组织增生活跃,并可见大量类骨组织和骨小梁形成,新生软骨和周围软骨组织融合,并与周围组织连接。

La identidad es la trama invisible que permite articular a los integrantes de una comunidad y vincular armoniosamente el pasado con el futuro.

同一性是一条无形的线,把集体的各个部分联系在一起,并协调城市的过去和未来。

Inhomogeneous 4 parameters triphasic model can describe the depthdependent material attribute of articular cartilage more exactly.

该模型可以更准确地描述软骨组织随深度变化的材料属性。

Two defects of articular cartilage were made in the trochlear groove of each new zealand rabbit.

二、动物体内实验部分:将48只双侧膝关节软骨缺损的3月龄新西兰兔动物模型随机分为四组。

Methods Thirty adult New Zealand rabbits were divided into three groups, group A,B,C, with ten rabbits in each group. One full-thickness defects of articular cartilage, 4 mm×5 mm×2.5 mm in size, was made in the trochlear groove of the left femur of each rabbit. The defects of group A and B were filled with BMP/FG and FG respectively. The defects of group C were left empty.

30只新西兰种成年兔随机分为A,B,C三组,每只兔子左膝股骨髁间凹做一大小为4 mm×5 mm×2.5 mm的全层关节软骨缺损。A、B组缺损内分别填充BMP/FG及FG,C组为空白。

INTERVENTIONS: A full-thickness defect in articular cartilage was made on the femoral trochlear surface by a drill of 5 mm in diameter and 3 mm in depth. Rabbits were allocated into filling group(type Ⅱ collagen sponge was grafted into left keen joint defect) and control group(right knee joint defect site was set as control) according to random number table.

干预:在股骨滑车面钻孔为直径5 mm、深3 mm的全层关节软骨缺损,按随机数分为填充组(左膝关节缺损部位植入Ⅱ型胶原海绵)和对照组(右膝关节缺损部位作为空白对照)。

Results: The finding showed that experimental group have different promotional action in repairing articular cartilage defect, and the group A have a more ideal effect, there are unvisible results in the control group.

结果:实验组的复合材料对软骨缺损的修复都有不同程度的促进作用,其中A组的效果最佳,对照组的软骨缺损的修复不明显,实验组总评分与对照组相比差异有显著性意义(P.05)。

At 4,8,12 weeks after operation,the articular cartilages of tibial plateau were taken and the contents of uronic acidand hydroxyprolineand the water ratioin the cartilage were determined.

术后 4、 8、 1 2周取胫骨平台软骨做糖醛酸、羟脯氨酸含量及含水率的测定。

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