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aorta相关的网络例句

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与 aorta 相关的网络例句 [注:此内容来源于网络,仅供参考]

The experimental group was given an operation of cutting infundibulum and the contol group was given an exposure of not cutting the infundibulum.Both groups were raised under similar condition.After surviving for 30 days,the animals were killed and specimens from aorta were taken and observed under light and electron microscope.

实验组行手术切断漏斗,对照组行手术暴露但不切断漏斗,手术后2d,两组动物移至同一条件下饲养,存活30d后用1%多聚甲醛+1.25%戊二醛液从心脏灌流固定,取主动脉作光、电镜观察。

AIM: To reconstruct the tissue engineering blood vessel by using acellular matrix of porcine thoracic aorta tissue as the scaffold, and by inoculability of vascular endothelial cells from human umbilical cord vein.

目的: 采用脱细胞处理的猪胸主动脉血管基质作为支架材料,接种人脐带血管内皮细胞,构建组织工程血管。

Sudden deceleration may also result in shearing injury to the intima of the aorta.

突然减速也可能造成主动脉内膜的剪式裂伤。

Results The intima of aorta were smooth with only one layer of endothelial cells in control group.

结果 对照组胸主动脉内膜光滑,内皮细胞排列整齐。

METHODS: The models of abdominal aorta transplantation were made with micro-surgery in rats. The recipients were divided into three groups: allograft control group, atorvastatin-treated group and isograft control group. Vascular intimal thickness in all of the groups was observed by histological examination. The expression of proliferation cell nuclear antigenl and α-smooth muscle actin were determined by immunohistochemistry. The content of nitric oxide was measured by nitrate reductase chromatometry.

建立大鼠腹主动脉移植慢性排斥反应模型,分为3组:异系移植对照组、异系移植治疗组和同系移植组,60d后对移植动脉行病理组织学检测,观察移植动脉内膜增生程度;并行免疫组织化学染色,检测移植动脉增殖细胞核抗原和平滑肌肌动蛋白的表达情况,行硝酸还原酶比色法测定移植动脉组织NO的含量。

Meanwhile,concentrations of KP and ANP in atrium and aorta of the rats were observed as well.

KP不仅具有和ANP一样的利尿、舒张血管、抑制肾素-血管紧张素系统的作用,还具有抑制心肌Na+-K+-ATP酶的作用〔2〕。

In the whole rats, the following tissues or organs were extracted and fixed in the most appropriated fixator (Bouin liquid or Formalin at 10%) then included in paraffin blocks : all the organs showing significant macroscopic lesions, encephalon (brain hemisphere, cerebellum and annular protuberance), spinal cord, sciatic nerve, salivary glands, esophagus, stomach, duodenum, jejunum, ileon, caecum and colon, rectum, liver, spleen, bone marrow, lymph node close to and at distance, thymus, heart, aorta, trachea, lungs, thyroid/parathyroid, surrenals, pancreas, hypophysis, kidney, bladder, gonad, uterus, mammary glands, epididymis, prostate, eyes, lachrymal glands, bones, skin, thigh muscle and implantation sites (2 per animal).

从整体大鼠内,下列组织或器官提取后用最适宜的固化剂( Bouin 液或10%的甲醛溶液)进行固定,然后包入石蜡块内:所有显示有明显肉眼可见损害的器官、脑髓、脊髓、坐骨神经、唾液腺、食道、胃、十二指肠、空肠、回肠、盲肠、大肠、直肠、肝脏、脾、骨髓、近端和远端淋巴结、胸腺、心脏、主动脉、气管、肺、甲状腺/甲状旁腺、肾上腺、胰腺、垂体、肾、膀胱、性腺、子宫、乳腺、附睾、前列腺、眼睛、泪腺、骨骼、皮肤、股肌以及假体植入部位(每只动物2处)。

Result: After treatment of the left ventricular with DD, vs aorta-contricted model group, NO content, cNOS and Na+-K+-ATPase, Ca2+-ATPase activity were significantly increased, the content of AngII in left ventricle and serum and iNOS activity and the ratio of HW/BW, LVI, MD were significantly reduced.

结果:与模型组比较DD能显著提高心肌组织NO含量和结构型NOS,Na+-K+-ATPase,Ca2+-ATPase活性;降低心肌组织诱生型NOS活性;抑制血浆及心肌组织AngⅡ和胶原的产生;减轻心脏质量参数及MD。

Microparticle can carry antisense MCP-1 gene into aorta successfully, which can inhibitate MCP-1 gene expression, microphage infusion and slow down the development of AAA. At the same time, there is no MCP-1 gene expression in other position, suggesting the microparticle si an secure gene transfer carrier.

以微粒子为载体,能成功地将反义MCP-1基因导入动脉组织中,并能明显抑制内源性MCP-1基因的表达和巨噬细胞在动脉壁的浸润程度,减缓腹主动脉瘤的进一步生长,同时外源基因在机体的其他部位没有表达,提示微粒子是一种安全的基因转移载体。

Rat AAA model was made by perfusion PPE elastase through the abdominal aorta, and the specimen was obtained on postoperativeday 3, 7, 14, 28, respectively. Specific elastic fiber staining was used to observe the disruption of elastic fiber in aortic wall. Immunohistochemistry staining of CD68 was applied to observe the microphage infiltration. The in tisu hybridization and western blot of MCP-1 and MMP-2 were used to study the expression of mRNA and protein. Thus we can analyze the facilitated function of MCP-1 gene to the microphage adherence and infiltration on the earlier AAA.

采用经腹主动脉腔内导管灌注的方法制成大鼠AAA模型,分别于术后3天、1周、2周、3周、4周获取各组大鼠腹主动脉标本,行弹力纤维特殊染色观察动脉壁弹力纤维的破坏情况;行CD68免疫组织化学染色观察动脉壁中巨噬细胞的浸润;行MCP-1及MMP-2的原位分子杂交、Western蛋白质印迹观察二者的mRNA表达和蛋白表达;分析MCP-1基因促进巨噬细胞的黏附、浸润在早期AAA发病中的作用。

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