查询词典 anoxia

Combining the former investigations on root structure and physiology of these genotypes with the present result, it's concluded that this is a novel waterlogging tolerance gene related to structural adaptation, and can be induced in roots of sesame and wheat under anoxia stress where endogenous ethylene production and formation of aerenchyma have been observed.

结合前期有关根结构和生理研究表明，XET在芝麻和小麦根中受厌氧诱导表达，与根中内源乙烯增生以及根皮层通气组织形成具有同步性，为新的与结构适应相关的耐渍基因。

MSBR system operates through periods of anoxia, anaerobic, aerobiosis and precipitation, micro-organism can metaboly through multiapproaches, making the degradation of organic matters perfect, the energy consumption low and the good denitrification and dephosphorization.

MSBR 系统的运行经历缺氧、厌氧、好氧、沉淀等阶段，微生物可通过多种途径进行代谢，使有机物的降解更完全且能耗少、除磷脱氮效果好的优点。

Besides, HIF-1 can quickly induce a lot of expressions and the adjustment of anoxia-induced genes, so as to enhance the hypoxia-induced genetic transcription and increase the expressions, and engender physiological effects, such as adjusting the transcription of EPO genes to increase their production, thereby the red blood cells quantity is enlarged, which strengthens the ability of oxygen transportation and mitigates anoxia; adjusting VEGF genetic transcription to engender more blood capillaries, consequently the transportation and supply of oxygen and substance of energy source are strengthened; adjusting the glycolytic enzyme to enhance glycolysis ability under anoxemia condition and increase energy supply; functioning on HO-1 genes in vascular smooth muscle cells to produce HO-1, which can engender NO and CO as pneumatic signal molecules to activate the guanylate cyclases, thus CGMP level is improved to make the vascular smooth muscles chalasia, to restrain platelet agglutination, so that theblood flow and vascular permeability are increased, and the anoxic tissues obtain ample oxygen supply, etc..

在低氧环境中，HIF-1可以被很快诱导大量表达并介导低氧诱导基因的调控，使低氧诱导基因转录增强，表达产物增多，产生生理效应。

Methods The model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed. The content of maiondialdehyde,activity of superoxide dismutasein the brain tissues of cerebral ischemia and anoxia mice were measured by TBA,NBT technology respectively.

采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型，观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响；TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

ResultsBVF could remarkably lengthen the survival time of cerebral ischemia and anoxia mice, and the time of catching their breath after mice decollated,could evidently decrease mouse brain capillary vessel permeability and the content of MDA, increase activity of SOD in the brain tissues of cerebral ischemia and anoxia mice.

方法采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型，观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响；TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

ResultsBVF could remarkably lengthen the survival time of cerebral ischemia and anoxia mice, and the time of catching their breath after mice decollated,could evidently decrease mouse brain capillary vessel permeability and the content of MDA, increase activity of SOD in the brain tissues of cerebral ischemia and anoxia mice.

结果苦菜总黄酮能显著延长小鼠存活时间、喘息时间，能显著降低缺血缺氧小鼠脑毛细血管的通透性，能显著降低脑组织MDA含量，能显著提高脑组织SOD活性。

The use of fetal nonstress test combined with fetal umbilical artery doppler velocimetry would improve the diagnosis for fetal anoxia,and give a reliable information to treat fetal anoxia for clinicians.

NST与多普勒脐动脉血流速度测定联合应用可以提高胎儿宫内缺氧状态的诊断，为临床及时纠正宫内缺氧提供了可靠依据。

Outcome:(1) Acute ischemic damage may induce the expression of c-fos protein in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia. And the expression level of c-fos can be up-regulated by putting blood of 12 hand Jing Points. So we think that treated by this therapy can rise the irritability of brain to resist the development of ischemia and enhance the ability to plerosis.(2) Acute ischemic damage may induce the expression of HspVO in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia.

三 结果(1)急性缺血性脑损伤可快速诱导c-fos蛋白在皮层缺血区脑组织的表达，其表达有随缺血时间延长而逐渐增多的趋势，提示脑的缺血性损伤可应激性地增强神经细胞对缺血、缺氧的耐受和适应能力，而应用井穴放血的干预治疗可明显上调缺血区皮层脑组织c-fos的表达水平，进一步增强脑组织对抗缺血性损伤的应激能力，抵抗缺血性脑损害的进一步发展。

The machanism may be that 7-NI inhibits activation of nNOS and decreases the number of overproducing neuronal NO in hippocampal during anoxia,so the ability of hippicampal slices against anoxic injury is increased and neurotoxic role of neuronal NO during anoxia is definite.

作用机理可能是7-NI抑制nNOS活性降低缺氧后海马神经原性NO的过多形成，从而提高海马脑片抗缺氧损伤的能力，更加明确了缺氧早期神经原性NO的神经毒性作用。

In pacemaker cells of SA node superfused with the oxygenated K—H solution, MDP, APA, V〓, VDD, RPF and APD〓 were—45±5mv, 48±4mv, 2. 4±0.9v/s, 89±8 mv/s, 200±11bpm and 206±15ms, respectively. Anoxia (perfused for 20min with a modified K—H solution gassed with 100% N〓 and deprived of glucose) increased MDP, APA and V〓, and decreased VDD and RPF in a timedependent manner. At anoxia for 20min, the above parameters were—58±5mv, 64±5mv, 11. 7±2. 7v/s, 24±7mv/s, 92±11bpm and 179± 14ms, respectively. ADase (10U/L).

环戊腺苷和苯基异丙基腺苷不仅能增加豚鼠窦房结起搏细胞APA、MDP和〓，且能减小VDD、RPF和〓在环戊腺苷20nmol／L浓度时，MDA、APA和〓分别增值5.4±0.7mv、7.6±0.9mv和2.4±0.5v／s，VDD、RPF和〓分别降低32±4mv／s、68±9bpm和31±12ms（p.05或0.01）。0.1μmol／L 8—苯茶碱和10μmol／L格列苯脲能明显地抑制环戊腺苷的电生理效应。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。